about
Specification and function of hemogenic endothelium during embryogenesisThe functional sites of miRNAs and lncRNAs in gastric carcinogenesisIdentification of a gene expression driven progression pathway in myxoid liposarcoma.Assessment of the Potential of CDK2 Inhibitor NU6140 to Influence the Expression of Pluripotency Markers NANOG, OCT4, and SOX2 in 2102Ep and H9 Cells.Comparative computational analysis of pluripotency in human and mouse stem cells.Novel insights into embryonic stem cell self-renewal revealed through comparative human and mouse systems biology networksTex10 Coordinates Epigenetic Control of Super-Enhancer Activity in Pluripotency and ReprogrammingThe Notch-mediated hyperplasia circuitry in Drosophila reveals a Src-JNK signaling axis.Overexpression of cyclin D1 induces the reprogramming of differentiated epidermal cells into stem cell-like cells.Hemogenic endothelial cell specification requires c-Kit, Notch signaling, and p27-mediated cell-cycle control.STAT3 Target Genes Relevant to Human Cancers.The knockdown of H19lncRNA reveals its regulatory role in pluripotency and tumorigenesis of human embryonic carcinoma cells.Diabetes mellitus and cellular replacement therapy: Expected clinical potential and perspectives.Importance of Myc-related microRNAs in induced pluripotency.Effects of cell cycle phases on the induction of dental pulp stem cells toward dopaminergic-like cells.Transcriptomic profiling of human embryonic stem cells upon cell cycle manipulation during pluripotent state dissolution.Atypical heterochromatin organization and replication are rapidly acquired by somatic cells following fusion-mediated reprogramming by mouse ESCs.ZD7288, a blocker of the HCN channel family, increases doubling time of mouse embryonic stem cells and modulates differentiation outcomes in a context-dependent manner.HiHiMap: single-cell quantitation of histones and histone posttranslational modifications across the cell cycle by high-throughput imaging.Proliferation and survival of human amniotic epithelial cells during their hepatic differentiation.Repression of the Aryl Hydrocarbon Receptor Is Required to Maintain Mitotic Progression and Prevent Loss of Pluripotency of Embryonic Stem Cells.Cell Cycle Regulation of Stem Cells by MicroRNAs.Cycling to Meet Fate: Connecting Pluripotency to the Cell Cycle.
P2860
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P2860
description
2013 nî lūn-bûn
@nan
2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
The cell cycle and pluripotency
@ast
The cell cycle and pluripotency
@en
The cell cycle and pluripotency
@nl
type
label
The cell cycle and pluripotency
@ast
The cell cycle and pluripotency
@en
The cell cycle and pluripotency
@nl
prefLabel
The cell cycle and pluripotency
@ast
The cell cycle and pluripotency
@en
The cell cycle and pluripotency
@nl
P2860
P356
P1433
P1476
The cell cycle and pluripotency
@en
P2093
Anna Philpott
Christopher Hindley
P2860
P304
P356
10.1042/BJ20121627
P407
P577
2013-04-15T00:00:00Z