Do plaques rapidly progress prior to myocardial infarction? The interplay between plaque vulnerability and progression
about
Imaging Atherosclerosis.Morphological features of coronary plaques in WHHLMI rabbits (Oryctolagus cuniculus), an animal model for familial hypercholesterolemiaNoninvasive Molecular Imaging of Disease Activity in Atherosclerosis.Cohort profile of BIOMArCS: the BIOMarker study to identify the Acute risk of a Coronary Syndrome-a prospective multicentre biomarker study conducted in the Netherlands.The Updated NICE Guidelines: Cardiac CT as the First-Line Test for Coronary Artery Disease.Invasive or non-invasive imaging for detecting high-risk coronary lesions?Pathophysiology of ST-segment elevation myocardial infarction: novel mechanisms and treatments.Thrombus aspiration in acute myocardial infarction.C1q/TNF-related protein 9 inhibits the cholesterol-induced Vascular smooth muscle cell phenotype switch and cell dysfunction by activating AMP-dependent kinase.High wall shear stress and high-risk plaque: an emerging concept.Presence of Severe Stenosis in Most Culprit Lesions of Patients with ST-segment Elevation Myocardial InfarctionPrognostic Significance of Nonobstructive Left Main Coronary Artery Disease in Women Versus Men: Long-Term Outcomes From the CONFIRM (Coronary CT Angiography Evaluation For Clinical Outcomes: An International Multicenter) Registry.Comparison of circulating dendritic cell and monocyte subsets at different stages of atherosclerosis: insights from optical coherence tomography.Multimodality Imaging of Angiogenesis in a Rabbit Atherosclerotic Model by GEBP11 Peptide Targeted Nanoparticles.Carotid Intraplaque Hemorrhage Imaging with Quantitative Vessel Wall T1 Mapping: Technical Development and Initial Experience.Multifunctional Role of Chymase in Acute and Chronic Tissue Injury and Remodeling.Relationship between monocyte-to-lymphocyte ratio and coronary plaque vulnerability in patients with stable angina.Contribution of TLR4 signaling in intermittent hypoxia-mediated atherosclerosis progression.Endoplasmic reticulum stress in perivascular adipose tissue promotes destabilization of atherosclerotic plaque by regulating GM-CSF paracrine.Subclinical hypothyroidism is associated with lipid-rich plaques in patients with coronary artery disease as assessed by optical coherence tomography
P2860
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P2860
Do plaques rapidly progress prior to myocardial infarction? The interplay between plaque vulnerability and progression
description
2015 nî lūn-bûn
@nan
2015 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Do plaques rapidly progress pr ...... vulnerability and progression
@ast
Do plaques rapidly progress pr ...... vulnerability and progression
@en
Do plaques rapidly progress pr ...... vulnerability and progression
@nl
type
label
Do plaques rapidly progress pr ...... vulnerability and progression
@ast
Do plaques rapidly progress pr ...... vulnerability and progression
@en
Do plaques rapidly progress pr ...... vulnerability and progression
@nl
prefLabel
Do plaques rapidly progress pr ...... vulnerability and progression
@ast
Do plaques rapidly progress pr ...... vulnerability and progression
@en
Do plaques rapidly progress pr ...... vulnerability and progression
@nl
P2093
P1433
P1476
Do plaques rapidly progress pr ...... vulnerability and progression
@en
P2093
G. W. Stone
J. Leipsic
R. Blankstein
P304
P356
10.1161/CIRCRESAHA.117.305637
P407
P577
2015-06-19T00:00:00Z