Carbon monoxide, hydrogen sulfide, and nitric oxide as signaling molecules in the gastrointestinal tract
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Neuromodulatory effects and targets of the SCFAs and gasotransmitters produced by the human symbiotic microbiotaEmerging Roles of Hydrogen Sulfide in Inflammatory and Neoplastic Colonic DiseasesEpigenetic Regulation of Enteric Neurotransmission by Gut BacteriaMolecular Regulation of Toll-like Receptors in Asthma and COPDGut Bacteria and Hydrogen Sulfide: The New Old Players in Circulatory System HomeostasisPathophysiological Mechanisms in Gaseous Therapies for Severe MalariaExtracellular hemoglobin: the case of a friend turned foeHydrogen Sulfide in Physiology and Diseases of the Digestive TractThe elements of life and medicinesH2S Donor NaHS Changes the Production of Endogenous H2S and NO in D-Galactose-Induced Accelerated Ageing.Different Types of Dietary Fibers Trigger Specific Alterations in Composition and Predicted Functions of Colonic Bacterial Communities in BALB/c MiceImportance of NO and its related compounds in enteric nervous system regulation of gut homeostasis and disease susceptibilityHydrogen sulfide regulates the colonic motility by inhibiting both L-type calcium channels and BKCa channels in smooth muscle cells of rat colon.Regulation of gap junction channels and hemichannels by phosphorylation and redox changes: a revision.Nitric Oxide and Hydrogen Sulfide Interact When Modulating Gastric Physiological Functions in Rodents.Nitric oxide, afferent sensory nerves, and antioxidative enzymes in the mechanism of protection mediated by tricarbonyldichlororuthenium(II) dimer and sodium hydrosulfide against aspirin-induced gastric damage.Carbon monoxide contributes to the constipating effects of granisetron in rat colon.Changes in nitrergic and tachykininergic pathways in rat proximal colon in response to chronic treatment with otilonium bromide.Carbon monoxide: A new player in the redox regulation of connexin hemichannels.An emerging role for gasotransmitters in the control of breathing and ionic regulation in fish.A novel pH-controlled hydrogen sulfide donor protects gastric mucosa from aspirin-induced injury.Gaseous Mediators in Gastrointestinal Mucosal Defense and Injury.Mucosal acidification increases hydrogen sulfide release through up-regulating gene and protein expressions of cystathionine gamma-lyase in the rat gastric mucosa.The Protective Role of Carbon Monoxide (CO) Produced by Heme Oxygenases and Derived from the CO-Releasing Molecule CORM-2 in the Pathogenesis of Stress-Induced Gastric Lesions: Evidence for Non-Involvement of Nitric Oxide (NO).Hydrogen Sulfide and Carbon Monoxide Protect Gastric Mucosa Compromised by Mild Stress Against Alendronate InjuryInhibition of RhoA/Rho kinase pathway and smooth muscle contraction by hydrogen sulfide.Carbon Monoxide Modulates Connexin Function through a Lipid Peroxidation-Dependent Process: A Hypothesis.A novel orally administered trimebutine compound (GIC-1001) is anti-nociceptive and features peripheral opioid agonistic activity and Hydrogen Sulphide-releasing capacity in mice.Sulfur Cycling and the Intestinal Microbiome.Effect of chemical structure of S-nitrosothiols on nitric oxide release mediated by the copper sites of a metal organic framework based environment.Reply to letter to the editor by Assoc. Prof. Yusuf Ozgur Cakmak, MD, PhD.International Union of Basic and Clinical Pharmacology. CII: Pharmacological Modulation of H2S Levels: H2S Donors and H2S Biosynthesis Inhibitors.Carbon monoxide released from its pharmacological donor, tricarbonyldichlororuthenium (II) dimer, accelerates the healing of pre-existing gastric ulcers.Diligustilide releases H2S and stabilizes S-nitrosothiols in ethanol-induced lesions on rat gastric mucosa.H2 S-induced gastric fundus smooth muscle tension potentiation is mediated by the phosphoinositide 3-kinase/Akt/endothelial nitric oxide synthase pathway.Ironing out the Details: Exploring the Role of Iron and Heme in Blood-Sucking Arthropods.Hydrogen sulfide is expressed in the human and the rat cultured nucleus pulposus cells and suppresses apoptosis induced by hypoxia.Excited by fasting? NO.Partial conversion of thioamide into nitrile in a copper(II) complex of 2,6-diacetylpyridine bis(thiosemicarbazone), a drug prototype for Alzheimer's disease.Angiogenic, Antiangiogenic Molecules, and Bioactive Lipids in Preeclampsia.
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P2860
Carbon monoxide, hydrogen sulfide, and nitric oxide as signaling molecules in the gastrointestinal tract
description
2014 nî lūn-bûn
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2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2014年の論文
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2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
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name
Carbon monoxide, hydrogen sulf ...... in the gastrointestinal tract
@ast
Carbon monoxide, hydrogen sulf ...... in the gastrointestinal tract
@en
Carbon monoxide, hydrogen sulf ...... in the gastrointestinal tract
@nl
type
label
Carbon monoxide, hydrogen sulf ...... in the gastrointestinal tract
@ast
Carbon monoxide, hydrogen sulf ...... in the gastrointestinal tract
@en
Carbon monoxide, hydrogen sulf ...... in the gastrointestinal tract
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prefLabel
Carbon monoxide, hydrogen sulf ...... in the gastrointestinal tract
@ast
Carbon monoxide, hydrogen sulf ...... in the gastrointestinal tract
@en
Carbon monoxide, hydrogen sulf ...... in the gastrointestinal tract
@nl
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Carbon monoxide, hydrogen sulf ...... in the gastrointestinal tract
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Gianrico Farrugia
Joseph H. Szurszewski
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10.1053/J.GASTRO.2014.04.041
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2014-08-01T00:00:00Z