Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives - Wikidata - awgldk - TriplyDB

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

http://www.wikidata.org/entity/Q27307777

about

Neurovascular control of blood pressure is influenced by aging, sex, and sex hormones.Evidence of alterations in the expression of orphan receptors GPR26 and GPR39 due to the etiology of the metabolic syndrome.Influence of sex, menstrual cycle, and oral contraceptives on cerebrovascular resistance and cardiorespiratory function during Valsalva or standing.Plasma Homoarginine Concentrations According to Use of Hormonal Contraception

P2860

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P2860

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

http://www.wikidata.org/entity/Q27307777

description

2016 nî lūn-bûn

@nan

2016 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2016 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

name

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

@ast

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

@en

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

@nl

type

label

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

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Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

@en

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

@nl

prefLabel

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

@ast

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

@en

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

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FPHYS.2016.00215

P1433

Frontiers in Physiology

P1476

Greater Beta-Adrenergic Receptor Mediated Vasodilation in Women Using Oral Contraceptives

@en

P2093

Benjamin J Walker

http://www.w3.org/2001/XMLSchema#string

Garrett L Peltonen

http://www.w3.org/2001/XMLSchema#string

Jacqueline K Limberg

http://www.w3.org/2001/XMLSchema#string

John W Harrell

http://www.w3.org/2001/XMLSchema#string

Joshua J Sebranek

http://www.w3.org/2001/XMLSchema#string

Marlowe W Eldridge

http://www.w3.org/2001/XMLSchema#string

Rebecca E Johansson

http://www.w3.org/2001/XMLSchema#string

William G Schrage

http://www.w3.org/2001/XMLSchema#string

P2860

Regulation of increased blood flow (hyperemia) to muscles during exercise: a hierarchy of competing physiological needs

Preserved Microvascular Endothelial Function in Young, Obese Adults with Functional Loss of Nitric Oxide Signaling.

A real-time device for converting Doppler ultrasound audio signals into fluid flow velocity

Effect of oral contraceptives on the renin angiotensin system and renal function.

A combined oral contraceptive containing 30 mcg ethinyl estradiol and 3.0 mg drospirenone does not impair endothelium-dependent vasodilation.

Hormonal contraception and cardiovascular system.

Forearm vasodilator responses to a β-adrenergic receptor agonist in premenopausal and postmenopausal women.

New progestagens for contraceptive use.

Effect of vitamin C on hyperoxia-induced vasoconstriction in exercising skeletal muscle.

Prostaglandins do not contribute to the nitric oxide-mediated compensatory vasodilation in hypoperfused exercising muscle.

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215

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scholarly article

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954980

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10.3389/FPHYS.2016.00215

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P478

7

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J Mikhail Kellawan

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2016-06-08T00:00:00Z

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27375493

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4896959

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