Positional cloning of zinc finger domain transcription factor Zfp69, a candidate gene for obesity-associated diabetes contributed by mouse locus Nidd/SJL - Wikidata - awgldk - TriplyDB

Positional cloning of zinc finger domain transcription factor Zfp69, a candidate gene for obesity-associated diabetes contributed by mouse locus Nidd/SJL

Positional cloning of zinc finger domain transcription factor Zfp69, a candidate gene for obesity-associated diabetes contributed by mouse locus Nidd/SJL

http://www.wikidata.org/entity/Q27312155

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Insights into obesity and diabetes at the intersection of mouse and human genetics Genetic and epigenetic control of metabolic health Identification of Four Mouse Diabetes Candidate Genes Altering β-Cell Proliferation Positional cloning of a type 2 diabetes quantitative trait locus; tomosyn-2, a negative regulator of insulin secretion Is there a role for endogenous retroviruses to mediate long-term adaptive phenotypic response upon environmental inputs?Dissociation of lipotoxicity and glucotoxicity in a mouse model of obesity associated diabetes: role of forkhead box O1 (FOXO1) in glucose-induced beta cell failure.Mouse obesity network reconstruction with a variational Bayes algorithm to employ aggressive false positive control The ancient mammalian KRAB zinc finger gene cluster on human chromosome 8q24.3 illustrates principles of C2H2 zinc finger evolution associated with unique expression profiles in human tissues.Genetic and genomic analysis of hyperlipidemia, obesity and diabetes using (C57BL/6J × TALLYHO/JngJ) F2 mice.The genetic basis of obesity-associated type 2 diabetes (diabesity) in polygenic mouse models Congenic strains provide evidence that a mapped locus on chromosome 15 influences excitotoxic cell death Subcongenic analysis of tabw2 obesity QTL on mouse chromosome 6.An interval of the obesity QTL Nob3.38 within a QTL hotspot on chromosome 1 modulates behavioral phenotypes QTL analysis of dietary obesity in C57BL/6byj X 129P3/J F2 mice: diet- and sex-dependent effects.Altered pancreatic growth and insulin secretion in WSB/EiJ mice.Lipase maturation factor 1: a lipase chaperone involved in lipid metabolism.Systems genetics of susceptibility to obesity-induced diabetes in mice The diabetes gene Zfp69 modulates hepatic insulin sensitivity in mice.The Transcriptional Repressive Activity of KRAB Zinc Finger Proteins Does Not Correlate with Their Ability to Recruit TRIM28.Skeletal muscle mitochondrial uncoupling prevents diabetes but not obesity in NZO mice, a model for polygenic diabesity.Single diabetic QTL derived from OLETF rat is a sufficient agent for severe diabetic phenotype in combination with leptin-signaling deficiency.Genetic background (DDD/Sgn versus C57BL/6J) strongly influences postnatal growth of male mice carrying the A(y) allele at the agouti locus: identification of quantitative trait loci associated with diabetes and body weight loss.The KRAB zinc finger protein RSL1 modulates sex-biased gene expression in liver and adipose tissue to maintain metabolic homeostasis.Mouse genetic approaches applied to the normal tissue radiation response.How mice are indispensable for understanding obesity and diabetes genetics.Genome-wide association study of body mass index in subjects with alcohol dependence.Loss of function of Ifi202b by a microdeletion on chromosome 1 of C57BL/6J mice suppresses 11β-hydroxysteroid dehydrogenase type 1 expression and development of obesity.DNA-Methylation and Body Composition in Preschool Children: Epigenome-Wide-Analysis in the European Childhood Obesity Project (CHOP)-Study.Moo1 obesity quantitative trait locus in BTBR T+ Itpr3tf/J mice increases food intake.Genetics of metabolic syndrome: potential clues from wild-derived inbred mouse strains.Congenic mapping identifies a novel Idd9 subregion regulating type 1 diabetes in NOD mice.Altered Mitochondria, Protein Synthesis Machinery, and Purine Metabolism Are Molecular Contributors to the Pathogenesis of Creutzfeldt-Jakob Disease.A computational biology approach of a genome-wide screen connected miRNAs to obesity and type 2 diabetes.

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Positional cloning of zinc finger domain transcription factor Zfp69, a candidate gene for obesity-associated diabetes contributed by mouse locus Nidd/SJL

http://www.wikidata.org/entity/Q27312155

description

2009 nî lūn-bûn

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2009 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2009 թվականի հուլիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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Positional cloning of zinc fin ...... ibuted by mouse locus Nidd/SJL

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Positional cloning of zinc fin ...... ibuted by mouse locus Nidd/SJL

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Positional cloning of zinc fin ...... ibuted by mouse locus Nidd/SJL

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Positional cloning of zinc fin ...... ibuted by mouse locus Nidd/SJL

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Positional cloning of zinc fin ...... ibuted by mouse locus Nidd/SJL

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Positional cloning of zinc fin ...... ibuted by mouse locus Nidd/SJL

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Positional cloning of zinc fin ...... ibuted by mouse locus Nidd/SJL

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Annette Schürmann

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Hadi Al-Hasani

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Heike Vogel

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Marcel-Dominique Block

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Marko Teichert

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Reinhart Kluge

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P2860

Predicting deleterious amino acid substitutions

The evolutionarily conserved Krüppel-associated box domain defines a subfamily of eukaryotic multifingered proteins

The structurally disordered KRAB repression domain is incorporated into a protease resistant core upon binding to KAP-1-RBCC domain

SMAP2, a novel ARF GTPase-activating protein, interacts with clathrin and clathrin assembly protein and functions on the AP-1-positive early endosome/trans-Golgi network

KRAB-containing zinc-finger repressor proteins

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

A mutation in the gene encoding the alpha 2 chain of the fibril-associated collagen IX, COL9A2, causes multiple epiphyseal dysplasia (EDM2)

A genome-wide association study identifies novel risk loci for type 2 diabetes

Skeletal abnormalities in mice lacking extracellular matrix proteins, thrombospondin-1, thrombospondin-3, thrombospondin-5, and type IX collagen

Positional cloning of "Lisch-Like", a candidate modifier of susceptibility to type 2 diabetes in mice

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