Complement C5-deficient mice are protected from seizures in experimental cerebral malaria - Wikidata - awgldk - TriplyDB

Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

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Experimental Cerebral Malaria Spreads along the Rostral Migratory Stream A Murine Model to Study Epilepsy and SUDEP Induced by Malaria Infection Infections, inflammation and epilepsy 2014 Epilepsy Benchmarks Area III: Improve Treatment Options for Controlling Seizures and Epilepsy-Related Conditions Without Side Effects Target deletion of complement component 9 attenuates antibody-mediated hemolysis and lipopolysaccharide (LPS)-induced acute shock in mice.Increased susceptibility to pentylenetetrazol following survival of cerebral malaria in mice.

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Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

http://www.wikidata.org/entity/Q27312562

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2014 nî lūn-bûn

@nan

2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

@ast

Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

@en

Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

@nl

type

label

Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

@ast

Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

@en

Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

@nl

prefLabel

Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

@ast

Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

@en

Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

@nl

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Complement C5-deficient mice are protected from seizures in experimental cerebral malaria

@en

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Scott R Barnum

http://www.w3.org/2001/XMLSchema#string

Susan C Buckingham

http://www.w3.org/2001/XMLSchema#string

Theresa N Ramos

http://www.w3.org/2001/XMLSchema#string

P2860

Diagnosis and management of the neurological complications of falciparum malaria

The complement inhibitor eculizumab in paroxysmal nocturnal hemoglobinuria.

Cutting edge: the membrane attack complex of complement is required for the development of murine experimental cerebral malaria.

The C5 convertase is not required for activation of the terminal complement pathway in murine experimental cerebral malaria.

C5 deficiency and C5a or C5aR blockade protects against cerebral malaria

Functional roles for C5a and C5aR but not C5L2 in the pathogenesis of human and experimental cerebral malaria.

Inflammatory pathways of seizure disorders.

Deletion of carboxypeptidase N delays onset of experimental cerebral malaria.

Intracerebroventricular injection of the terminal complement complex causes inflammatory reaction in the rat brain.

Formation of complement membrane attack complex in mammalian cerebral cortex evokes seizures and neurodegeneration.

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e139-42

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scholarly article

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4446781

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10.1111/EPI.12858

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12

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55

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2014-11-10T00:00:00Z

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268156387

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25385326

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cerebral malaria

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4285666

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