Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Genome-wide analysis of structural variants reveals genetic differences in Chinese pigs.Autosomal Dominant Polycystic Kidney Patients May Be Predisposed to Various Cardiomyopathies.Polycystin 2-dependent cardio-protective mechanisms revealed by cardiac stress.

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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Barbara E Ehrlich

http://www.w3.org/2001/XMLSchema#string

Ivana Y Kuo

http://www.w3.org/2001/XMLSchema#string

Lily Nguyen

http://www.w3.org/2001/XMLSchema#string

Sophie L Duong

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P2860

Polycystin-2 is an intracellular calcium release channel

Polycystin 2 interacts with type I inositol 1,4,5-trisphosphate receptor to modulate intracellular Ca2+ signaling

Cardiovascular abnormalities in autosomal-dominant polycystic kidney disease

Polycystin-2 traffics to cilia independently of polycystin-1 by using an N-terminal RVxP motif.

Regulation of ryanodine receptor-dependent calcium signaling by polycystin-2

Polycystin-2 mutations lead to impaired calcium cycling in the heart and predispose to dilated cardiomyopathy

The HALT polycystic kidney disease trials: design and implementation.

Dynamic gene expression patterns in animal models of early and late heart failure reveal biphasic-bidirectional transcriptional activation of signaling pathways.

HEART DISEASE. Titin mutations in iPS cells define sarcomere insufficiency as a cause of dilated cardiomyopathy.

Decreased polycystin 2 expression alters calcium-contraction coupling and changes β-adrenergic signaling pathways.

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scholarly article

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2291884

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10.1371/JOURNAL.PONE.0153632

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English

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P478

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2016-01-01T00:00:00Z

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27081851

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P932

4833351

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Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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P2860

P2860

Decreased Polycystin 2 Levels Result in Non-Renal Cardiac Dysfunction with Aging

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P1433

P1476

P2093

P2860

P304

P31

P3181

P356

P407

P433

P478

P577

P698

P932

P3181

P356

P1433

P1476

P2093

P2860

P304

P31

P3181

P356

P407

P433

P478

P577

P698

P932