Cerebral biochemical pathways in experimental autoimmune encephalomyelitis and adjuvant arthritis: a comparative metabolomic study

Item

http://www.wikidata.org/entity/Q27315145

description

2013 nî lūn-bûn

@nan

2013 թուականին հրատարակուած գիտական յօդուած

@hyw

2013 թվականին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

Cerebral biochemical pathways ...... comparative metabolomic study

@ast

Cerebral biochemical pathways ...... comparative metabolomic study

@en

Cerebral biochemical pathways ...... comparative metabolomic study

@nl

type

Item

label

Cerebral biochemical pathways ...... comparative metabolomic study

@ast

Cerebral biochemical pathways ...... comparative metabolomic study

@en

Cerebral biochemical pathways ...... comparative metabolomic study

@nl

prefLabel

Cerebral biochemical pathways ...... comparative metabolomic study

@ast

Cerebral biochemical pathways ...... comparative metabolomic study

@en

Cerebral biochemical pathways ...... comparative metabolomic study

@nl

P1476

Cerebral biochemical pathways ...... comparative metabolomic study

@en

P2093

Carla Fernandez

http://www.w3.org/2001/XMLSchema#string

Evelyne Béraud

http://www.w3.org/2001/XMLSchema#string

Jean-François Pellissier

http://www.w3.org/2001/XMLSchema#string

Norbert W Lutz

http://www.w3.org/2001/XMLSchema#string

Patrick J Cozzone

http://www.w3.org/2001/XMLSchema#string

P2860

Evidence supporting a role for N-acetyl-L-aspartate as a molecular water pump in myelinated neurons in the central nervous system. An analytical review

Inflammatory multiple-sclerosis plaques generate characteristic metabolic profiles in cerebrospinal fluid

Metabolic fingerprints of altered brain growth, osmoregulation and neurotransmission in a Rett syndrome model

Evidence supporting a role for N-acetyl-l-aspartate as a molecular water pump in myelinated neurons in the central nervous system

Non-invasive measurement of brain damage in a primate model of multiple sclerosis.

Hypoperfusion of the cerebral white matter in multiple sclerosis: possible mechanisms and pathophysiological significance.

Inflammatory pain alters blood-brain barrier permeability and tight junctional protein expression.

Measurement of whole-brain atrophy in multiple sclerosis.

Multiparametric optimization of (31)P NMR spectroscopic analysis of phospholipids in crude tissue extracts. 1. Chemical shift and signal separation.

The c-Jun N-terminal kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain condition

P304

e56101

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1371/JOURNAL.PONE.0056101

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2013-01-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

235778435

http://www.w3.org/2001/XMLSchema#string

P698

23457507

http://www.w3.org/2001/XMLSchema#string

P921

experimental autoimmune encephalomyelitis

Bayokimischri

P932

3573043

http://www.w3.org/2001/XMLSchema#string