The adherens junctions control susceptibility to Staphylococcus aureus α-toxin - Wikidata - awgldk - TriplyDB

The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

http://www.wikidata.org/entity/Q27321037

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Making Bunyaviruses Talk: Interrogation Tactics to Identify Host Factors Required for Infection Genome-wide CRISPR screen reveals novel host factors required for Staphylococcus aureus α-hemolysin-mediated toxicity.A CRISPR toolbox to study virus-host interactions Staphylococcus aureus Pore-Forming Toxins.PLEKHA7 Recruits PDZD11 to Adherens Junctions to Stabilize Nectins The ever-emerging complexity of α-toxin's interaction with host cells Creating a RAW264.7 CRISPR-Cas9 Genome Wide Library.Bacteria and endothelial cells: a toxic relationship.Staphylococcus aureus pore-forming toxins: The interface of pathogen and host complexity.Staphylococcus aureus-Associated Skin and Soft Tissue Infections: Anatomical Localization, Epidemiology, Therapy and Potential Prophylaxis.Scissor sisters: regulation of ADAM10 by the TspanC8 tetraspanins.α-Toxin Regulates Local Granulocyte Expansion from Hematopoietic Stem and Progenitor Cells in Staphylococcus aureus-Infected Wounds.Haploid Mammalian Genetic Screen Identifies UBXD8 as a Key Determinant of HMGCR Degradation and Cholesterol Biosynthesis.Neutralizing α-toxin accelerates healing of Staphylococcus aureus-infected wounds in normal and diabetic mice.Cell-specific diversity in the expression and organization of cytoplasmic plaque proteins of apical junctions.Virulence gene profiles: alpha-hemolysin and clonal diversity in Staphylococcus aureus isolates from bovine clinical mastitis in China.Deciphering the Pathological Role of Staphylococcal α-Toxin and Panton-Valentine Leukocidin Using a Novel Ex Vivo Human Skin Model.More Than Skin Deep: Autophagy Is Vital for Skin Barrier Function.Surviving Deadly Lung Infections: Innate Host Tolerance Mechanisms in the Pulmonary System.

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The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

http://www.wikidata.org/entity/Q27321037

description

2015 nî lūn-bûn

@nan

2015 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

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The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

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The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

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The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

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The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

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The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

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The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

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The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

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The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

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PNAS.1510265112

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Proceedings of the National Academy of Sciences of the United States of America

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The adherens junctions control susceptibility to Staphylococcus aureus α-toxin

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Caleb D Marceau

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Donna M Bouley

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Fabio Bagnoli

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Hiroshi Kiyonari

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Jennifer H Lumb

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Jimit Shah

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Lauren M Popov

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Rachel L Cooper

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Sandra Citi

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PLEKHA7 is an adherens junction protein with a tissue distribution and subcellular localization distinct from ZO-1 and E-cadherin

Genome engineering using the CRISPR-Cas9 system

The TspanC8 subgroup of tetraspanins interacts with A disintegrin and metalloprotease 10 (ADAM10) and regulates its maturation and cell surface expression

Anchorage of microtubule minus ends to adherens junctions regulates epithelial cell-cell contacts

Haploid genetic screens in human cells identify host factors used by pathogens

Staphylococcus aureus α-toxin: nearly a century of intrigue

Listeria monocytogenes internalin B activates junctional endocytosis to accelerate intestinal invasion

Helicobacter pylori usurps cell polarity to turn the cell surface into a replicative niche

Structure of staphylococcal alpha-hemolysin, a heptameric transmembrane pore

Staphylococcus aureus infections

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46

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Masatoshi Takeichi

Christina Merakou

Stephen J Galli

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2015-10-21T00:00:00Z

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283207942

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