Axonal regeneration. Systemic administration of epothilone B promotes axon regeneration after spinal cord injury - Wikidata - awgldk - TriplyDB

Axonal regeneration. Systemic administration of epothilone B promotes axon regeneration after spinal cord injury

Axonal regeneration. Systemic administration of epothilone B promotes axon regeneration after spinal cord injury

http://www.wikidata.org/entity/Q27331607

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Regulation of Microtubule Dynamics in Axon Regeneration: Insights from C. elegans Traumatic Axonal Injury: Mechanisms and Translational Opportunities Microtubule plus-end tracking proteins in neuronal development Biomaterial Approaches to Enhancing Neurorestoration after Spinal Cord Injury: Strategies for Overcoming Inherent Biological Obstacles The axonal cytoskeleton: from organization to function Intravenous multipotent adult progenitor cell treatment decreases inflammation leading to functional recovery following spinal cord injury.Tau Biology and Tau-Directed Therapies for Alzheimer's Disease Neuronal responses to stress and injury in C. elegans Growth control mechanisms in neuronal regeneration Aβ-mediated spine changes in the hippocampus are microtubule-dependent and can be reversed by a subnanomolar concentration of the microtubule-stabilizing agent epothilone D Autophagy induction stabilizes microtubules and promotes axon regeneration after spinal cord injury.Neurite outgrowth is driven by actin polymerization even in the presence of actin polymerization inhibitors.Extrinsic Repair of Injured Dendrites as a Paradigm for Regeneration by Fusion in Caenorhabditis elegans.Epothilone D prevents binge methamphetamine-mediated loss of striatal dopaminergic markers.Mechanisms responsible for the inhibitory effects of epothilone B on scar formation after spinal cord injury Cortex-dependent recovery of unassisted hindlimb locomotion after complete spinal cord injury in adult rats.Astrocyte scar formation aids central nervous system axon regeneration.Protein deacetylases and axonal regeneration.Unraveling the mechanisms of synapse formation and axon regeneration: the awesome power of C. elegans genetics.Administration of low dose estrogen attenuates persistent inflammation, promotes angiogenesis, and improves locomotor function following chronic spinal cord injury in rats.The fusogen AFF-1 can rejuvenate the regenerative potential of adult dendritic trees by self-fusion.Signaling Over Distances.High-throughput proteomics reveal alarmins as amplifiers of tissue pathology and inflammation after spinal cord injury Context Specificity of Stress-activated Mitogen-activated Protein (MAP) Kinase Signaling: The Story as Told by Caenorhabditis elegans.Characterizing the Epothilone Binding Site on β-Tubulin by Photoaffinity Labeling: Identification of β-Tubulin Peptides TARGSQQY and TSRGSQQY as Targets of an Epothilone Photoprobe for Polymerized Tubulin.NB-3 signaling mediates the cross-talk between post-traumatic spinal axons and scar-forming cells.Extracellular vimentin is a novel axonal growth facilitator for functional recovery in spinal cord-injured mice Enhancement of Peripheral Nerve Regrowth by the Purine Nucleoside Analog and Cell Cycle Inhibitor, Roscovitine.Reconnecting Eye to Brain.Effects of Microtubule Stabilization by Epothilone B Depend on the Type and Age of Neurons.Branch-Specific Microtubule Destabilization Mediates Axon Branch Loss during Neuromuscular Synapse Elimination.Zonisamide-loaded triblock copolymer nanomicelles as a novel drug delivery system for the treatment of acute spinal cord injury Stability properties of neuronal microtubules.Epothilone B induces apoptosis and enhances apoptotic effects of ABT-737 on human cancer cells via PI3K/AKT/mTOR pathway.CNS repair and axon regeneration: Using genetic variation to determine mechanisms.CRMPs Function in Neurons and Glial Cells: Potential Therapeutic Targets for Neurodegenerative Diseases and CNS Injury.Effective improvement of the neuroprotective activity after spinal cord injury by synergistic effect of glucocorticoid with biodegradable amphipathic nanomicelles.Microtubule Destabilization Paves the Way to Parkinson's Disease.Tunable tissue scaffolds fabricated by in situ crosslink in phase separation system.Natural products from myxobacteria: novel metabolites and bioactivities.

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Axonal regeneration. Systemic administration of epothilone B promotes axon regeneration after spinal cord injury

http://www.wikidata.org/entity/Q27331607

description

2015 nî lūn-bûn

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2015 թուականի Մարտին հրատարակուած գիտական յօդուած

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2015 թվականի մարտին հրատարակված գիտական հոդված

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Axonal regeneration. Systemic ...... ation after spinal cord injury

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Axonal regeneration. Systemic ...... ation after spinal cord injury

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Christopher Sliwinski

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Kristina Dobrindt

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Margaret Bates

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P2860

Amyloid-β oligomers induce synaptic damage via Tau-dependent microtubule severing by TTLL6 and spastin

PTEN deletion enhances the regenerative ability of adult corticospinal neurons

The 3Ms of central spindle assembly: microtubules, motors and MAPs

Microtubule-binding agents: a dynamic field of cancer therapeutics.

Nogo-66 receptor antagonist peptide promotes axonal regeneration

Reactive astrocytes protect tissue and preserve function after spinal cord injury

Nogo-66 receptor prevents raphespinal and rubrospinal axon regeneration and limits functional recovery from spinal cord injury

Functional regeneration of respiratory pathways after spinal cord injury.

Chondroitinase ABC promotes functional recovery after spinal cord injury

Microtubule stabilization reduces scarring and causes axon regeneration after spinal cord injury.

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