MiR-410 is overexpressed in liver and colorectal tumors and enhances tumor cell growth by silencing FHL1 via a direct/indirect mechanism
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MicroRNAs in the Myocyte Enhancer Factor 2 (MEF2)-regulated Gtl2-Dio3 Noncoding RNA Locus Promote Cardiomyocyte Proliferation by Targeting the Transcriptional Coactivator Cited2.Functional elucidation of miR-494 in the tumorigenesis of nasopharyngeal carcinoma.MicroRNA-410 acts as oncogene in NSCLC through downregulating SLC34A2 via activating Wnt/β-catenin pathwayMiR-410 induces stemness by inhibiting Gsk3β but upregulating β-catenin in non-small cells lung cancer.MicroRNA-410 regulates autophagy-related gene ATG16L1 expression and enhances chemosensitivity via autophagy inhibition in osteosarcomaIdentification of microRNA signature and potential pathway targets in prostate cancer.MiR-495 functions as an adjuvant to radiation therapy by reducing the radiation-induced bystander effect.MicroRNA-410 promotes cell proliferation by targeting BRD7 in non-small cell lung cancer.Differentially expressed MicroRNAs provide mechanistic insight into fibrosis-associated liver carcinogenesis in mice.LncRNA OIP5-AS1 loss-induced microRNA-410 accumulation regulates cell proliferation and apoptosis by targeting KLF10 via activating PTEN/PI3K/AKT pathway in multiple myeloma.MiRNA-target network analysis identifies potential biomarkers for Traditional Chinese Medicine (TCM) syndrome development evaluation in hepatitis B caused liver cirrhosis.MicroRNA Responses to the Genotoxic Carcinogens Aflatoxin B1 and Benzo[a]pyrene in Human HepaRG Cells.Profound changes in miRNA expression during cancer initiation by aflatoxin B1 and their abrogation by the chemopreventive triterpenoid CDDO-Im.
P2860
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P2860
MiR-410 is overexpressed in liver and colorectal tumors and enhances tumor cell growth by silencing FHL1 via a direct/indirect mechanism
description
2014 nî lūn-bûn
@nan
2014 թուականին հրատարակուած գիտական յօդուած
@hyw
2014 թվականին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
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2014年论文
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name
MiR-410 is overexpressed in li ...... ia a direct/indirect mechanism
@ast
MiR-410 is overexpressed in li ...... ia a direct/indirect mechanism
@en
MiR-410 is overexpressed in li ...... ia a direct/indirect mechanism
@nl
type
label
MiR-410 is overexpressed in li ...... ia a direct/indirect mechanism
@ast
MiR-410 is overexpressed in li ...... ia a direct/indirect mechanism
@en
MiR-410 is overexpressed in li ...... ia a direct/indirect mechanism
@nl
prefLabel
MiR-410 is overexpressed in li ...... ia a direct/indirect mechanism
@ast
MiR-410 is overexpressed in li ...... ia a direct/indirect mechanism
@en
MiR-410 is overexpressed in li ...... ia a direct/indirect mechanism
@nl
P2093
P2860
P1433
P1476
MiR-410 is overexpressed in li ...... ia a direct/indirect mechanism
@en
P2093
Haifeng Song
Jing Zhang
Long Cheng
Mengmeng Jiang
Xiaoai Zhang
Xiaojie Xu
Zhongyi Fan
P2860
P304
P356
10.1371/JOURNAL.PONE.0108708
P407
P577
2014-01-01T00:00:00Z