Tnni3k modifies disease progression in murine models of cardiomyopathy - Wikidata - awgldk - TriplyDB

Tnni3k modifies disease progression in murine models of cardiomyopathy

Tnni3k modifies disease progression in murine models of cardiomyopathy

http://www.wikidata.org/entity/Q27349094

about

TNNI3K mutation in familial syndrome of conduction system disease, atrial tachyarrhythmia and dilated cardiomyopathy Dissection of a quantitative trait locus for PR interval duration identifies Tnni3k as a novel modulator of cardiac conduction Troponin I-interacting protein kinase: a novel cardiac-specific kinase, emerging as a molecular target for the treatment of cardiac disease Characterisation of a C1qtnf5 Ser163Arg knock-in mouse model of late-onset retinal macular degeneration Strain-specific modifier genes of Cecr2-associated exencephaly in mice: genetic analysis and identification of differentially expressed candidate genes Additional sex combs-like family genes are required for normal cardiovascular development Transcription of a protein-coding gene on B chromosomes of the Siberian roe deer (Capreolus pygargus)Genetic variations strongly influence phenotypic outcome in the mouse retina.Identification of a cardiac disease modifier gene using forward genetics in the mouse Adenovirus-mediated overexpression of cardiac troponin I-interacting kinase promotes cardiomyocyte hypertrophy.Modifier genes and the plasticity of genetic networks in mice.Overexpression of TNNI3K, a cardiac-specific MAPKKK, promotes cardiac dysfunction.Defining the pathways underlying the prolonged PR interval in atrioventricular conduction disease.Distinct genetic regions modify specific muscle groups in muscular dystrophy.TNNI3K, a cardiac-specific kinase, promotes physiological cardiac hypertrophy in transgenic mice.Inhibition of the cardiomyocyte-specific kinase TNNI3K limits oxidative stress, injury, and adverse remodeling in the ischemic heart A founder mutation in the Ashkenazi Jewish population affecting messenger RNA splicing of the CCM2 gene causes cerebral cavernous malformations Mapping genetic contributions to cardiac pathology induced by Beta-adrenergic stimulation in mice Genetics of common forms of heart failure: challenges and potential solutions.Rare Variants Association Analysis in Large-Scale Sequencing Studies at the Single Locus Level Genetic mutations and mechanisms in dilated cardiomyopathy A modifier screen identifies DNAJB6 as a cardiomyopathy susceptibility gene.The CRISPR/Cas9 System and the Possibility of Genomic Edition for Cardiology Systems Genetics Approach Identifies Gene Pathways and Adamts2 as Drivers of Isoproterenol-Induced Cardiac Hypertrophy and Cardiomyopathy in Mice The genetic landscape of cardiomyopathy and its role in heart failure.Frequency of mononuclear diploid cardiomyocytes underlies natural variation in heart regeneration.Factors other than genotype account largely for the phenotypic variation of the pulmonary valve in Syrian hamsters.Genetically alike Syrian hamsters display both bifoliate and trifoliate aortic valves.Comparative phenotypic assessment of cardiac pathology, physiology, and gene expression in C3H/HeJ, C57BL/6J, and B6C3F1/J mice.Whole exome sequencing identifies the TNNI3K gene as a cause of familial conduction system disease and congenital junctional ectopic tachycardia.

P2860

Q24299150-460D1E26-9B83-4A67-9661-D4E03B5FBC61 Q24305930-8CE93BC1-13C6-4FE7-8395-F68CC1CDBB3E Q26864088-FBC3FAFF-EF7E-4150-9648-B713EA207E1E Q28478012-0FE2E516-6D1E-4CCF-84D9-11AB7B63282D Q28510741-16589B75-1B86-4BF9-907C-B1F0F284F064 Q28585968-26782CDD-53FC-4B74-BF8E-2E8790DCAF11 Q28681575-3A94ABEB-2EEB-4171-A4FD-A0E6E45C0D76 Q30474767-662A6379-4C01-4A9E-ABA4-048D939A8738 Q33504526-1942C571-DFEF-49B6-B2FB-D9C1E8829AA9 Q34164033-F8C7D619-BD0E-4BA4-AD64-DBBF81BB45B8 Q34236664-BC12BA76-A5EE-459C-A7F4-ECA61D4E9CB6 Q34307352-F11EE4FA-BF03-4E89-BE81-D5A1A201E6D9 Q34510215-6BC2D0AC-AD1D-4280-99D7-21F2CCBEB24B Q34518008-A9DD7FA5-0DA1-4331-A476-6591120C16D3 Q34615034-104B3AF3-7102-4E72-B893-A85306A98C83 Q35019445-89563482-829B-47D0-8753-C6B44CF06B67 Q35093781-30DF7AC3-6789-4E5C-A45A-CAB98A790222 Q35102230-4450950C-1FC3-4224-9641-F0E4A614642C Q35525031-DE647ED6-0CCA-4AAA-AF1E-B91E493C8F5B Q36063830-DA4D44BE-4A06-4FEA-8A1D-D40315C5160E Q36497170-F7C3C31D-2795-4AD9-85E1-EE7808F0DE1D Q37255814-54D0A8B2-253A-4648-B0E8-9EC32E6383ED Q37594531-AB47EE2B-F714-4398-AC3E-1CBD55F3A741 Q37682494-92B3AFAC-1299-41F3-874F-E3B346F1A833 Q38343935-C9070D1B-C52F-4E1D-B37F-6285C08EC166 Q46345451-8F440FB3-4B3F-4927-B266-8631CD51CB41 Q47756812-F4B5098B-4685-45E8-A735-15684E20C083 Q51764243-E26A1800-1750-4F43-A314-81DE22FE1E60 Q53452720-CF9A32E4-821F-4797-A95D-EC3916597619 Q54982915-7A0A14C0-79DF-4EB7-8F9D-129D4496DDB9

P2860

Tnni3k modifies disease progression in murine models of cardiomyopathy

http://www.wikidata.org/entity/Q27349094

description

2009 nî lūn-bûn

@nan

2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Tnni3k modifies disease progression in murine models of cardiomyopathy

@ast

Tnni3k modifies disease progression in murine models of cardiomyopathy

@en

Tnni3k modifies disease progression in murine models of cardiomyopathy

@nl

type

label

Tnni3k modifies disease progression in murine models of cardiomyopathy

@ast

Tnni3k modifies disease progression in murine models of cardiomyopathy

@en

Tnni3k modifies disease progression in murine models of cardiomyopathy

@nl

prefLabel

Tnni3k modifies disease progression in murine models of cardiomyopathy

@ast

Tnni3k modifies disease progression in murine models of cardiomyopathy

@en

Tnni3k modifies disease progression in murine models of cardiomyopathy

@nl

P1433

Q27349094-BED93939-F574-436F-9499-1B2C09DB4413

P1476

Q27349094-82701C56-0268-46AB-A692-D097D9AC0422

P2093

Q27349094-3544709F-D967-4DB2-8AC3-AF97C11F86A8

Q27349094-3BB1634F-B6D7-4A52-B77C-0EEA84073958

Q27349094-7708057B-EE2D-4F3F-8307-C2D8CA7128B5

Q27349094-A7A964D1-FE97-430D-923E-836F651FC3C9

Q27349094-C4D1BB48-9846-402F-B795-C3BE8BD8C5D0

Q27349094-D633632C-130E-41AC-9E2E-E5E6EADC5806

Q27349094-DF4D5A6B-2FBD-4BC0-996C-A0BC59B2DE6B

Q27349094-FAD4ED69-669B-4A64-825B-B31786C5472F

P2860

Q27349094-14D5B049-DE24-464C-8B61-A4F003D6DE89

Q27349094-47D17E91-6F04-4F5E-9E52-15CA517CA495

Q27349094-621EC91D-10F4-4F5F-9930-AA6DD72E3802

Q27349094-69059F8A-A3EC-4B89-845F-4F11F8DCE9AF

Q27349094-793C230C-0325-4314-9437-09E47C1EB23E

Q27349094-811FF385-B184-426C-A41C-A49F1D9EC15E

Q27349094-876D12C2-E725-422F-96C0-F2F0246C3292

Q27349094-B6A59520-4D9A-4DE5-BF91-9DEB7E6CF287

Q27349094-B806C600-EC67-45A2-B692-D678CD06BC18

Q27349094-BCCE868D-B766-43BE-A98F-93BEBA269837

P304

Q27349094-34836C37-6267-4D93-B3A3-1B70A7112655

P31

Q27349094-D41873D2-D79C-4D3D-84EB-F488B542C61F

P3181

Q27349094-8401300E-ACD4-47FD-B517-D02F28B9E9A2

P356

Q27349094-2020265F-0EC7-4133-81AF-89DD0E619C01

P407

Q27349094-911DB2E1-B38D-4890-A72B-A5EBBEAA50C3

P433

Q27349094-2176B3DC-7059-454F-84E7-02326B85E9DB

P478

Q27349094-0C727396-B1C0-4F0B-8D3F-75DF972AFEAA

P577

Q27349094-97D241E7-2425-4350-B545-5F5A67696593

P5875

Q27349094-BF659D18-A13C-483B-8705-AB56240DEDD7

P698

Q27349094-C1B472C4-2917-49C6-BF97-7FD5D08D394F

P932

Q27349094-26C106AC-4C71-4F78-9101-3B9D6B51FB74

P3181

P356

JOURNAL.PGEN.1000647

P1433

P1476

Tnni3k modifies disease progression in murine models of cardiomyopathy

@en

P2093

Douglas A Marchuk

http://www.w3.org/2001/XMLSchema#string

Ferrin C Wheeler

http://www.w3.org/2001/XMLSchema#string

Hao Tang

http://www.w3.org/2001/XMLSchema#string

Howard A Rockman

http://www.w3.org/2001/XMLSchema#string

Lan Mao

http://www.w3.org/2001/XMLSchema#string

Odessa A Marks

http://www.w3.org/2001/XMLSchema#string

Pei-Lun Chu

http://www.w3.org/2001/XMLSchema#string

Tracy N Hadnott

http://www.w3.org/2001/XMLSchema#string

P2860

Cloning and characterization of a novel cardiac-specific kinase that interacts specifically with cardiac troponin I

Regulation of Ca2+ signaling in transgenic mouse cardiac myocytes overexpressing calsequestrin

HL-1 cells: a cardiac muscle cell line that contracts and retains phenotypic characteristics of the adult cardiomyocyte

P19 embryonal carcinoma cells.

Overexpression of TNNI3K, a cardiac-specific MAP kinase, promotes P19CL6-derived cardiac myogenesis and prevents myocardial infarction-induced injury.

Impact of genetic polymorphisms on heart failure prognosis.

Integrin-linked kinase at the heart of cardiac contractility, repair, and disease.

Segregation of atrial-specific and inducible expression of an atrial natriuretic factor transgene in an in vivo murine model of cardiac hypertrophy.

Analysis of canonical and non-canonical splice sites in mammalian genomes

Computer methods to locate signals in nucleic acid sequences.

P304

e1000647

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P3181

412157

http://www.w3.org/2001/XMLSchema#string

P356

10.1371/JOURNAL.PGEN.1000647

http://www.w3.org/2001/XMLSchema#string

P407

P433

9

http://www.w3.org/2001/XMLSchema#string

P478

5

http://www.w3.org/2001/XMLSchema#string

P577

2009-09-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

26820737

http://www.w3.org/2001/XMLSchema#string

P698

19763165

http://www.w3.org/2001/XMLSchema#string

P932

2731170

http://www.w3.org/2001/XMLSchema#string