The severity of murray valley encephalitis in mice is linked to neutrophil infiltration and inducible nitric oxide synthase activity in the central nervous system
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Dual Function of Ccr5 during Langat Virus Encephalitis: Reduction in Neutrophil-Mediated Central Nervous System Inflammation and Increase in T Cell-Mediated Viral ClearanceImmunobiology of congenital cytomegalovirus infection of the central nervous system—the murine cytomegalovirus modelNeuroadapted yellow fever virus 17D: genetic and biological characterization of a highly mouse-neurovirulent virus and its infectious molecular cloneLack of both Fas ligand and perforin protects from flavivirus-mediated encephalitis in mice.Nitric oxide and virus infectionMorphological features of Murray Valley encephalitis virus infection in the central nervous system of swiss miceApoptosis in animal models of virus-induced diseaseWest Nile virus and its emergence in the United States of America.Excess neutrophil infiltration during cytomegalovirus brain infection of interleukin-10-deficient miceIL-22 signaling contributes to West Nile encephalitis pathogenesis.Neuroprotection and progenitor cell renewal in the injured adult murine retina requires healing monocyte-derived macrophages.Both spike and background genes contribute to murine coronavirus neurovirulenceAntiviral macrophage responses in flavivirus encephalitisInduction of virus-specific effector immune cell response limits virus replication and severe disease in mice infected with non-lethal West Nile virus Eg101 strain.The contribution of rodent models to the pathological assessment of flaviviral infections of the central nervous systemCD8 T cell-initiated blood-brain barrier disruption is independent of neutrophil supportTargeted blockade in lethal West Nile virus encephalitis indicates a crucial role for very late antigen (VLA)-4-dependent recruitment of nitric oxide-producing macrophages.The immune response to herpes simplex virus type 1 infection in susceptible mice is a major cause of central nervous system pathology resulting in fatal encephalitis.Encephalitic Arboviruses: Emergence, Clinical Presentation, and Neuropathogenesis.Duck Tembusu virus exhibits neurovirulence in BALB/c mice.Apoptosis of hippocampal pyramidal neurons is virus independent in a mouse model of acute neurovirulent picornavirus infectionChemokine receptors as important regulators of pathogenesis during arboviral encephalitisThe intrathecal expression and pathogenetic role of Th17 cytokines and CXCR2-binding chemokines in tick-borne encephalitis.
P2860
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P2860
The severity of murray valley encephalitis in mice is linked to neutrophil infiltration and inducible nitric oxide synthase activity in the central nervous system
description
1999 nî lūn-bûn
@nan
1999 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
The severity of murray valley ...... in the central nervous system
@ast
The severity of murray valley ...... in the central nervous system
@en
The severity of murray valley ...... in the central nervous system
@nl
type
label
The severity of murray valley ...... in the central nervous system
@ast
The severity of murray valley ...... in the central nervous system
@en
The severity of murray valley ...... in the central nervous system
@nl
prefLabel
The severity of murray valley ...... in the central nervous system
@ast
The severity of murray valley ...... in the central nervous system
@en
The severity of murray valley ...... in the central nervous system
@nl
P2093
P2860
P1433
P1476
The severity of murray valley ...... in the central nervous system
@en
P2093
A C Carrello
D M Andrews
L M Sammels
P C McMinn
V B Matthews
P2860
P304
P577
1999-10-01T00:00:00Z