Defective Hepatic Response to Interferon and Activation of Suppressor of Cytokine Signaling 3 in Chronic Hepatitis C - Wikidata - awgldk - TriplyDB

Defective Hepatic Response to Interferon and Activation of Suppressor of Cytokine Signaling 3 in Chronic Hepatitis C

Defective Hepatic Response to Interferon and Activation of Suppressor of Cytokine Signaling 3 in Chronic Hepatitis C

http://www.wikidata.org/entity/Q27478298

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Vitamin D supplementation improves sustained virologic response in chronic hepatitis C (genotype 1)-naïve patients Host restriction factors for hepatitis C virus SOCS, inflammation, and cancer Hepatitis C virus and ethanol alter antigen presentation in liver cells Gene expression and hepatitis C virus infection Using high-throughput genomics to study hepatitis C: What determines the outcome of infection?Inhibition of Intrahepatic Gamma Interferon Production by Hepatitis C Virus Nonstructural Protein 5A in Transgenic Mice Mechanisms of action of interferon and ribavirin in chronic hepatitis C: Summary of a workshop.Interleukin-6 haplotypes and the response to therapy of chronic hepatitis C virus infection Influenza A virus inhibits type I IFN signaling via NF-kappaB-dependent induction of SOCS-3 expression.Hepatic gene expression during treatment with peginterferon and ribavirin: Identifying molecular pathways for treatment response.Expression of selected genes in liver biopsy specimens in relation to early virological response in patients with chronic hepatitis C with HCV mono- and HIV/HCV co-infection.Suppressor of cytokine signaling 3 suppresses hepatitis C virus replication in an mTOR-dependent manner.SOCS1, a Negative Regulator of Cytokine Signals and TLR Responses, in Human Liver Diseases.Differential transcriptional responses to interferon-alpha and interferon-gamma in primary human hepatocytes.Predicting the probable outcome of treatment in HCV patients.S-adenosyl methionine improves early viral responses and interferon-stimulated gene induction in hepatitis C nonresponders.Hepatic SOCS3 expression is strongly associated with non-response to therapy and race in HCV and HCV/HIV infection CD4 T-cell transcriptome analysis reveals aberrant regulation of STAT3 and Wnt signaling pathways in rheumatoid arthritis: evidence from a case-control study.Hepatitis C Virus Driven AXL Expression Suppresses the Hepatic Type I Interferon Response Vitamin D improves viral response in hepatitis C genotype 2-3 naïve patients.The Mechanism of Interferon Refractoriness During Hepatitis C Virus Infection and Its Reversal with a Peroxisome Proliferator-Activated Receptor α Agonist The impact of host factors on management of hepatitis C virus.Insulin resistance and metabolic syndrome in chronic liver diseases: old entities with new implications.Treatment failure in hepatitis C: mechanisms of non-response.Will vitamin d supplementation have a role in the treatment of patients with chronic hepatitis C?Antiviral therapy: why does it fail in HCV-related chronic hepatitis?Use of HOMA-IR in hepatitis C.Steatosis and insulin resistance in response to treatment of chronic hepatitis C.Hepatitis C virus induces a prediabetic state by directly impairing hepatic glucose metabolism in mice.SOCS3 revisited: a broad regulator of disease, now ready for therapeutic use?Regulation of suppressors of cytokine signaling as a therapeutic approach in autoimmune diseases, with an emphasis on multiple sclerosis.Specific and nonhepatotoxic degradation of nuclear hepatitis B virus cccDNA.miR-19a: an effective regulator of SOCS3 and enhancer of JAK-STAT signalling.Pegylated interferon-α induced hypoferremia is associated with the immediate response to treatment in hepatitis C.Geranylgeranylacetone has anti-hepatitis C virus activity via activation of mTOR in human hepatoma cells.SOCS3 and IRS-1 gene expression differs between genotype 1 and genotype 2 hepatitis C virus-infected HepG2 cells.Noninvasive monitoring of hepatic damage from hepatitis C virus infection.Characterisation of the cytokine milieu associated with the up-regulation of IL-6 and suppressor of cytokine 3 in chronic hepatitis C treatment non-responders.Are metabolic factors still important in the era of direct antiviral agents in patients with chronic hepatitis C?

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P2860

Defective Hepatic Response to Interferon and Activation of Suppressor of Cytokine Signaling 3 in Chronic Hepatitis C

http://www.wikidata.org/entity/Q27478298

description

2007 nî lūn-bûn

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2007 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2007 թվականի փետրվարին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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Defective Hepatic Response to ...... aling 3 in Chronic Hepatitis C

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J.GASTRO.2006.11.045

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Gastroenterology

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Jiing-Huey Lin

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Jordan J Feld

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Krishna Murthy

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Lawrence M Blatt

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Michael W Fried

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Santosh Nanda

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T Jake Liang

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Ying Huang

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P2860

Analysis of ISG expression in chronic hepatitis C identifies viperin as a potential antiviral effector

Effect of alpha interferon on the hepatitis C virus replicon

Nonlinear partial differential equations and applications: Genomic analysis of the host response to hepatitis C virus infection

Relationships between Hepatitis C Virus Replication and CXCL-8 Production In Vitro

Hepatitis C Virus Down-Regulates Insulin Receptor Substrates 1 and 2 through Up-Regulation of Suppressor of Cytokine Signaling 3

Activation of STAT3 by the Hepatitis C Virus Core Protein Leads to Cellular Transformation

Negative regulation of cytokine signaling pathways

Mechanisms of type-I- and type-II-interferon-mediated signalling

Identification of genes differentially regulated by interferon alpha, beta, or gamma using oligonucleotide arrays

Evasion of intracellular host defence by hepatitis C virus

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733-44

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6544855

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