Genetic Variation in OAS1 Is a Risk Factor for Initial Infection with West Nile Virus in Man - Wikidata - awgldk - TriplyDB

Genetic Variation in OAS1 Is a Risk Factor for Initial Infection with West Nile Virus in Man

Genetic Variation in OAS1 Is a Risk Factor for Initial Infection with West Nile Virus in Man

http://www.wikidata.org/entity/Q27487937

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Activation of 2' 5'-oligoadenylate synthetase by stem loops at the 5'-end of the West Nile virus genome Structural basis for cytosolic double-stranded RNA surveillance by human oligoadenylate synthetase 1 RNA sequencing reveals the role of splicing polymorphisms in regulating human gene expression The Interferon-Stimulated Gene Ifi27l2a Restricts West Nile Virus Infection and Pathogenesis in a Cell-Type- and Region-Specific Manner West Nile virus: review of the literature Novel approaches and challenges to treatment of central nervous system viral infections West Nile Virus: biology, transmission, and human infection Systems analysis of West Nile virus infection Impact of allele-specific peptides in proteome quantification Controlling complexity: the clinical relevance of mouse complex genetics Virus and Host Determinants of West Nile Virus Pathogenesis Progress on the development of therapeutics against West Nile virus Variation and genetic control of protein abundance in humans Genetics of gene expression in immunity to infection Immunogenetic factors associated with severe respiratory illness caused by zoonotic H1N1 and H5N1 influenza viruses.Genome-wide mRNA expression correlates of viral control in CD4+ T-cells from HIV-1-infected individuals OAS1 polymorphisms are associated with susceptibility to West Nile encephalitis in horses Signatures of Archaic Adaptive Introgression in Present-Day Human Populations.2'-5'-Oligoadenylate synthetase single-nucleotide polymorphisms and haplotypes are associated with variations in immune responses to rubella vaccine.Identification of a Sjögren's syndrome susceptibility locus at OAS1 that influences isoform switching, protein expression, and responsiveness to type I interferons.Host genetic risk factors for West Nile virus infection and disease progression.West Nile virus and its emergence in the United States of America.Identification of genes critical for resistance to infection by West Nile virus using RNA-Seq analysis.The interferon-induced gene Ifi27l2a is active in lung macrophages and lymphocytes after influenza A infection but deletion of Ifi27l2a in mice does not increase susceptibility to infection.Viral pathogen-associated molecular patterns regulate blood-brain barrier integrity via competing innate cytokine signals.Protection against West Nile virus infection in mice after inoculation with type I interferon-inducing RNA transcripts.A functional Toll-like receptor 3 gene (TLR3) may be a risk factor for tick-borne encephalitis virus (TBEV) infection A novel RNA molecular signature for activation of 2'-5' oligoadenylate synthetase-1 2'-5' oligoadenylate synthetase 1 polymorphism is associated with prostate cancer Mitochondrial localization of the OAS1 p46 isoform associated with a common single nucleotide polymorphism.Cervico-vaginal tissue ex vivo as a model to study early events in HIV-1 infection.Genetic variants and susceptibility to neurological complications following West Nile virus infection.Poorly neutralizing cross-reactive antibodies against the fusion loop of West Nile virus envelope protein protect in vivo via Fcgamma receptor and complement-dependent effector mechanisms.Long-term balancing selection at the antiviral gene OAS1 in Central African chimpanzees.The contribution of rodent models to the pathological assessment of flaviviral infections of the central nervous system Chemokine receptor Ccr2 is critical for monocyte accumulation and survival in West Nile virus encephalitis Cell-intrinsic innate immune control of West Nile virus infection.Association between high expression macrophage migration inhibitory factor (MIF) alleles and West Nile virus encephalitis.Activation of RNase L is dependent on OAS3 expression during infection with diverse human viruses Contrasted patterns of variation and evolutionary convergence at the antiviral OAS1 gene in old world primates.

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Genetic Variation in OAS1 Is a Risk Factor for Initial Infection with West Nile Virus in Man

http://www.wikidata.org/entity/Q27487937

description

2009 nî lūn-bûn

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2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի փետրվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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Andrea Lisco

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Epidemic West Nile encephalitis, New York, 1999: results of a household-based seroepidemiological survey

Inherited resistance to HIV-1 conferred by an inactivating mutation in CC chemokine receptor 5: studies in populations with contrasting clinical phenotypes, defined racial background, and quantified risk

CCR5 deficiency increases risk of symptomatic West Nile virus infection

Inhibition of Alpha/Beta Interferon Signaling by the NS4B Protein of Flaviviruses

Positional cloning of the murine flavivirus resistance gene

A nonsense mutation in the gene encoding 2'-5'-oligoadenylate synthetase/L1 isoform is associated with West Nile virus susceptibility in laboratory mice

Alpha/beta interferon protects against lethal West Nile virus infection by restricting cellular tropism and enhancing neuronal survival

RNase L plays a role in the antiviral response to West Nile virus.

PKR and RNase L Contribute to Protection against Lethal West Nile Virus Infection by Controlling Early Viral Spread in the Periphery and Replication in Neurons

Early Production of Type I Interferon during West Nile Virus Infection: Role for Lymphoid Tissues in IRF3-Independent Interferon Production

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David H. McDermott

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24044645

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West Nile virus

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