Naturally Occurring Hepatitis C Virus Subgenomic Deletion Mutants Replicate Efficiently in Huh-7 Cells and Are trans-Packaged In Vitro To Generate Infectious Defective Particles
about
Metabolism of phosphatidylinositol 4-kinase IIIα-dependent PI4P Is subverted by HCV and is targeted by a 4-anilino quinazoline with antiviral activityGenetic characterization of hepatitis C virus in long-term RNA replication using Li23 cell culture systemsHepatitis C virus RNA replication depends on specific cis- and trans-acting activities of viral nonstructural proteinsProduction of infectious hepatitis C virus by using RNA polymerase I-mediated transcription.Defective interfering viral particles in acute dengue infections.Productive homologous and non-homologous recombination of hepatitis C virus in cell culture.Production of hepatitis C virus lacking the envelope-encoding genes for single-cycle infection by providing homologous envelope proteins or vesicular stomatitis virus glycoproteins in trans.Permissivity of primary human hepatocytes and different hepatoma cell lines to cell culture adapted hepatitis C virus.Transient replication of Hepatitis C Virus sub-genomic RNA in murine cell lines is enabled by miR-122 and varies with cell passageDistribution of fitness in populations of dengue virusesA conserved NS3 surface patch orchestrates NS2 protease stimulation, NS5A hyperphosphorylation and HCV genome replication.Hepatitis C Virus Deletion Mutants Are Found in Individuals Chronically Infected with Genotype 1 Hepatitis C Virus in Association with Age, High Viral Load and Liver Inflammatory Activity.Dynamics of defective hepatitis C virus clones in reinfected liver grafts in liver transplant recipients: ultradeep sequencing analysis.Small heterodimer partner 1 directly interacts with NS5A viral protein and has a key role in HCV related liver cell transformation.Are trans-complementation systems suitable for hepatitis C virus life cycle studies?Incorporation of hepatitis C virus E1 and E2 glycoproteins: the keystones on a peculiar virion.Identification of novel RNA secondary structures within the hepatitis C virus genome reveals a cooperative involvement in genome packagingThe acidic domain of hepatitis C virus NS4A contributes to RNA replication and virus particle assembly.Genetic Analysis of Serum-derived HCV Defective Genomes Revealed Novel Viral Cis-Elements for Virus Replication and Assembly.
P2860
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P2860
Naturally Occurring Hepatitis C Virus Subgenomic Deletion Mutants Replicate Efficiently in Huh-7 Cells and Are trans-Packaged In Vitro To Generate Infectious Defective Particles
description
2009 nî lūn-bûn
@nan
2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Naturally Occurring Hepatitis ...... Infectious Defective Particles
@ast
Naturally Occurring Hepatitis ...... Infectious Defective Particles
@en
Naturally Occurring Hepatitis ...... Infectious Defective Particles
@nl
type
label
Naturally Occurring Hepatitis ...... Infectious Defective Particles
@ast
Naturally Occurring Hepatitis ...... Infectious Defective Particles
@en
Naturally Occurring Hepatitis ...... Infectious Defective Particles
@nl
prefLabel
Naturally Occurring Hepatitis ...... Infectious Defective Particles
@ast
Naturally Occurring Hepatitis ...... Infectious Defective Particles
@en
Naturally Occurring Hepatitis ...... Infectious Defective Particles
@nl
P2093
P2860
P356
P1433
P1476
Naturally Occurring Hepatitis ...... Infectious Defective Particles
@en
P2093
Giacomo Paonessa
Laura Pacini
Linda Bartholomew
Raffaele De Francesco
Rita Graziani
P2860
P304
P356
10.1128/JVI.00308-09
P577
2009-09-01T00:00:00Z