RUNX1 mutations in acute myeloid leukemia: results from a comprehensive genetic and clinical analysis from the AML study group. - Wikidata - awgldk - TriplyDB

RUNX1 mutations in acute myeloid leukemia: results from a comprehensive genetic and clinical analysis from the AML study group.

RUNX1 mutations in acute myeloid leukemia: results from a comprehensive genetic and clinical analysis from the AML study group.

http://www.wikidata.org/entity/Q27851623

about

The genetic basis of early T-cell precursor acute lymphoblastic leukaemia.Risk factors for relapse after allogeneic transplantation in acute myeloid leukemia Molecular landscape of acute myeloid leukemia in younger adults and its clinical relevance Genomics-based Approach and Prognostic Stratification Significance of Gene Mutations in Intermediate-risk Acute Myeloid Leukemia Molecular Genetic Markers in Acute Myeloid Leukemia Post-remission therapy for acute myeloid leukemia Molecular prognostic factors in cytogenetically normal acute myeloid leukemia CIViC database RUNX1 mutations are associated with poor outcome in younger and older patients with cytogenetically normal acute myeloid leukemia and with distinct gene and MicroRNA expression signatures.The molecular profile of adult T-cell acute lymphoblastic leukemia: mutations in RUNX1 and DNMT3A are associated with poor prognosis in T-ALL.RUNX1 mutations in acute myeloid leukemia are associated with distinct clinico-pathologic and genetic features.Level of RUNX1 activity is critical for leukemic predisposition but not for thrombocytopenia.Two novel RUNX1 mutations in a patient with congenital thrombocytopenia that evolved into a high grade myelodysplastic syndrome Spectrum of clinical and genetic features of patients with inherited platelet disorder with suspected predisposition to hematological malignancies: a nationwide survey in Japan.Bone marrow morphology and disease progression in congenital thrombocytopenia: a detailed clinicopathologic and genetic study of eight cases.Molecular therapy for acute myeloid leukaemia.Submicroscopic deletion of 5q involving tumor suppressor genes (CTNNA1, HSPA9) and copy neutral loss of heterozygosity associated with TET2 and EZH2 mutations in a case of MDS with normal chromosome and FISH results.The Runx-PU.1 pathway preserves normal and AML/ETO9a leukemic stem cells.Two decades of leukemia oncoprotein epistasis: the MLL1 paradigm for epigenetic deregulation in leukemia.ASXL1 mutations in younger adult patients with acute myeloid leukemia: a study by the German-Austrian Acute Myeloid Leukemia Study Group.HES1 is an independent prognostic factor for acute myeloid leukemia.Integrated genomic profiling, therapy response, and survival in adult acute myelogenous leukemia Accurate computation of survival statistics in genome-wide studies Runx1 loss minimally impacts long-term hematopoietic stem cells Prognostic relevance of RUNX1 mutations in T-cell acute lymphoblastic leukemia.Similarities and differences between therapy-related and elderly acute myeloid leukemia.RUNX1 mutations in clonal myeloid disorders: from conventional cytogenetics to next generation sequencing, a story 40 years in the making Residual Disease in a Novel Xenograft Model of RUNX1-Mutated, Cytogenetically Normal Acute Myeloid Leukemia Therapy-related myeloid neoplasms following treatment with radioiodine.Identification of a dynamic core transcriptional network in t(8;21) AML that regulates differentiation block and self-renewal.Heritable polymorphism predisposes to high BAALC expression in acute myeloid leukemia Deregulation of RAD21 and RUNX1 expression in endometrial cancer.High frequency of additional gene mutations in acute myeloid leukemia with MLL partial tandem duplication: DNMT3A mutation is associated with poor prognosis.RUNX1 mutations in cytogenetically normal acute myeloid leukemia are associated with a poor prognosis and up-regulation of lymphoid genes.Splicing factor mutations predict poor prognosis in patients with de novo acute myeloid leukemia Clinical role of microRNAs in cytogenetically normal acute myeloid leukemia: miR-155 upregulation independently identifies high-risk patients Genetic predispositions to childhood leukemia.CXXC5 (retinoid-inducible nuclear factor, RINF) is a potential therapeutic target in high-risk human acute myeloid leukemia The new provisional WHO entity 'RUNX1 mutated AML' shows specific genetics but no prognostic influence of dysplasia.Human BDH2, an anti-apoptosis factor, is a novel poor prognostic factor for de novo cytogenetically normal acute myeloid leukemia.

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RUNX1 mutations in acute myeloid leukemia: results from a comprehensive genetic and clinical analysis from the AML study group.

http://www.wikidata.org/entity/Q27851623

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2011 nî lūn-bûn

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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2011 թվականի ապրիլին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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RUNX1 mutations in acute myelo ...... ysis from the AML study group.

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RUNX1 mutations in acute myelo ...... ysis from the AML study group.

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RUNX1 mutations in acute myelo ...... ysis from the AML study group.

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RUNX1 mutations in acute myelo ...... ysis from the AML study group.

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RUNX1 mutations in acute myelo ...... ysis from the AML study group.

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RUNX1 mutations in acute myelo ...... ysis from the AML study group.

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RUNX1 mutations in acute myelo ...... ysis from the AML study group.

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RUNX1 mutations in acute myelo ...... ysis from the AML study group.

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RUNX1 mutations in acute myelo ...... ysis from the AML study group.

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JCO.2010.30.7926

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Journal of Clinical Oncology

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RUNX1 mutations in acute myelo ...... ysis from the AML study group.

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Andrea Corbacioglu

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Andrea Kündgen

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Andreas S Zimmermann

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Arnold Ganser

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Daniela Späth

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David Nachbaur

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Heinz A Horst

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Ingo G H Schmidt-Wolf

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Jürgen Krauter

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Jürgen Röck

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10.1200/JCO.2010.30.7926

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29

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Michael Pfreundschuh

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Brigitte Schlegelberger

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2011-04-01T00:00:00Z

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21343560

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