The LTB4-BLT1 axis mediates neutrophil infiltration and secondary injury in experimental spinal cord injury. - Wikidata - awgldk - TriplyDB

The LTB4-BLT1 axis mediates neutrophil infiltration and secondary injury in experimental spinal cord injury.

The LTB4-BLT1 axis mediates neutrophil infiltration and secondary injury in experimental spinal cord injury.

http://www.wikidata.org/entity/Q27863415

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New paradigms in the establishment and maintenance of gradients during directed cell migration Liposomal clodronate selectively eliminates microglia from primary astrocyte cultures Spinal Cord Injury Neutrophil contribution to spinal cord injury and repair.The tumor microenvironment shapes lineage, transcriptional, and functional diversity of infiltrating myeloid cells.Clostridium difficile-induced colitis in mice is independent of leukotrienes The prevalence and phenotype of activated microglia/macrophages within the spinal cord of the hyperostotic mouse (twy/twy) changes in response to chronic progressive spinal cord compression: implications for human cervical compressive myelopathy.Deficient CX3CR1 signaling promotes recovery after mouse spinal cord injury by limiting the recruitment and activation of Ly6Clo/iNOS+ macrophages.The absence of the leukotriene B4 receptor BLT1 attenuates peripheral inflammation and spinal nociceptive processing following intraplantar formalin injury.Matrix metalloproteinase-9 and stromal cell-derived factor-1 act synergistically to support migration of blood-borne monocytes into the injured spinal cord Systemic leukotriene B4 receptor antagonism lowers arterial blood pressure and improves autonomic function in the spontaneously hypertensive rat.Neutrophils cascading their way to inflammation Non-viral gene therapy for spinal cord regeneration Licofelone modulates neuroinflammation and attenuates mechanical hypersensitivity in the chronic phase of spinal cord injury Human spinal cord injury causes specific increases in surface expression of β integrins on leukocytes.The feasibility of in vivo imaging of infiltrating blood cells for predicting the functional prognosis after spinal cord injury.BLT1 signalling protects the liver against acetaminophen hepatotoxicity by preventing excessive accumulation of hepatic neutrophils.Emerging concepts in myeloid cell biology after spinal cord injury.Spatial and temporal activation of spinal glial cells: role of gliopathy in central neuropathic pain following spinal cord injury in rats.Lipid-cytokine-chemokine cascades orchestrate leukocyte recruitment in inflammation Role of phospholipase A2s and lipid mediators in secondary damage after spinal cord injury.n-3 Polyunsaturated fatty acids and mast cell activation.Naja sputatrix Venom Preconditioning Attenuates Neuroinflammation in a Rat Model of Surgical Brain Injury via PLA2/5-LOX/LTB4 Cascade Activation.Pleiotropic regulations of neutrophil receptors response to sepsis.Neutrophils and viral-induced neurologic disease.Macrophage Transcriptional Profile Identifies Lipid Catabolic Pathways That Can Be Therapeutically Targeted after Spinal Cord Injury.Critical Role of LTB4/BLT1 in IL-23-Induced Synovial Inflammation and Osteoclastogenesis via NF-κB.CCR5 blockade promotes M2 macrophage activation and improves locomotor recovery after spinal cord injury in mice.Involvement of the choroid plexus in the inflammatory response after acute spinal cord injury in dogs: an immunohistochemical study.Transplantation of mesenchymal stem cells promotes an alternative pathway of macrophage activation and functional recovery after spinal cord injury Blockade of interleukin-6 signaling inhibits the classic pathway and promotes an alternative pathway of macrophage activation after spinal cord injury in mice Valproic acid attenuates blood-spinal cord barrier disruption by inhibiting matrix metalloprotease-9 activity and improves functional recovery after spinal cord injury.Ly6C+ Ly6G- Myeloid-derived suppressor cells play a critical role in the resolution of acute inflammation and the subsequent tissue repair process after spinal cord injury.The pathophysiological role of acute inflammation after spinal cord injury.Multiple mechanisms underlying neuroprotection by secretory phospholipase A2 preconditioning in a surgically induced brain injury rat model.MicroRNA-155 Deficiency Suppresses Th17 Cell Differentiation and Improves Locomotor Recovery after Spinal Cord Injury.Clonidine, an alpha-2 adrenoceptor agonist relieves mechanical allodynia in oxaliplatin-induced neuropathic mice; potentiation by spinal p38 MAPK inhibition without motor dysfunction and hypotension.Too much of a good thing: How modulating LTB4 actions restore host defense in homeostasis or disease.Early transplantation of mesenchymal stem cells after spinal cord injury relieves pain hypersensitivity through suppression of pain-related signaling cascades and reduced inflammatory cell recruitment.Dioxin-induced increase in leukotriene B4 biosynthesis through the aryl hydrocarbon receptor and its relevance to hepatotoxicity owing to neutrophil infiltration.

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P2860

The LTB4-BLT1 axis mediates neutrophil infiltration and secondary injury in experimental spinal cord injury.

http://www.wikidata.org/entity/Q27863415

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

The LTB4-BLT1 axis mediates ne ...... perimental spinal cord injury.

@ast

The LTB4-BLT1 axis mediates ne ...... perimental spinal cord injury.

@en

The LTB4-BLT1 axis mediates ne ...... perimental spinal cord injury.

@nl

type

label

The LTB4-BLT1 axis mediates ne ...... perimental spinal cord injury.

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The LTB4-BLT1 axis mediates ne ...... perimental spinal cord injury.

@en

The LTB4-BLT1 axis mediates ne ...... perimental spinal cord injury.

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The LTB4-BLT1 axis mediates ne ...... xperimental spinal cord injury

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prefLabel

The LTB4-BLT1 axis mediates ne ...... perimental spinal cord injury.

@ast

The LTB4-BLT1 axis mediates ne ...... perimental spinal cord injury.

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The LTB4-BLT1 axis mediates ne ...... perimental spinal cord injury.

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AJPATH.2010.090839

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The American Journal of Pathology

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The LTB4-BLT1 axis mediates ne ...... perimental spinal cord injury.

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Akihito Harada

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Hirokazu Saiwai

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Hiromi Kumamaru

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Hisakata Yamada

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Yukihide Iwamoto

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P2860

A G-protein-coupled receptor for leukotriene B4 that mediates chemotaxis

Leukotrienes promote plasma leakage and leukocyte adhesion in postcapillary venules: in vivo effects with relevance to the acute inflammatory response

TNF-alpha blockage in a mouse model of SCI: evidence for improved outcome.

Targeted disruption of the leukotriene B(4) receptor in mice reveals its role in inflammation and platelet-activating factor-induced anaphylaxis

BLTR mediates leukotriene B(4)-induced chemotaxis and adhesion and plays a dominant role in eosinophil accumulation in a murine model of peritonitis

Characterization of a mouse second leukotriene B4 receptor, mBLT2: BLT2-dependent ERK activation and cell migration of primary mouse keratinocytes

NKT cell activation mediates neutrophil IFN-gamma production and renal ischemia-reperfusion injury.

Characterization and modeling of monocyte-derived macrophages after spinal cord injury.

MMP-9-positive neutrophil infiltration is associated to blood-brain barrier breakdown and basal lamina type IV collagen degradation during hemorrhagic transformation after human ischemic stroke.

Review of current evidence for apoptosis after spinal cord injury.

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Takehiko Yokomizo

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2010-05-01T00:00:00Z

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42371923

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