ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
about
Alpha 2-chimerin, an SH2-containing GTPase-activating protein for the ras-related protein p21rac derived by alternate splicing of the human n-chimerin gene, is selectively expressed in brain regions and testesA Ras-GTPase-activating protein SH3-domain-binding proteinRas activation in platelets after stimulation of the thrombin receptor, thromboxane A2 receptor or protein kinase CMuscarinic acetylcholine receptor subtypes as agonist-dependent oncogenesPurification and N-terminal sequence of the p21rho GTPase-activating protein, rho GAPAn interaction between p21ras and heat shock protein hsp60, a chaperoninsar1, a gene from Schizosaccharomyces pombe encoding a protein that regulates ras1p190 RhoGAP, the major RasGAP-associated protein, binds GTP directlyThe GTPase-activating protein n-chimaerin cooperates with Rac1 and Cdc42Hs to induce the formation of lamellipodia and filopodiaSmall G proteins in the cardiovascular system: physiological and pathological aspectsDevelopmental regulation and neuronal expression of the mRNA of rat n-chimaerin, a p21rac GAP:cDNA sequencePurification of a plasma membrane-associated GTPase-activating protein specific for rap1/Krev-1 from HL60 cellsDiversity of G proteins in signal transductionp120RasGAP Protein Mediates Netrin-1 Protein-induced Cortical Axon Outgrowth and Guidance.The Ras-GTPase-activating protein SH3 domain is required for Cdc2 activation and mos induction by oncogenic Ras in Xenopus oocytes independently of mitogen-activated protein kinase activation.Muscarinic receptors transform NIH 3T3 cells through a Ras-dependent signalling pathway inhibited by the Ras-GTPase-activating protein SH3 domain.GTPase-activating protein SH2-SH3 domains induce gene expression in a Ras-dependent fashion.Guanine nucleotide exchange factor-like factor (Rlf) induces gene expression and potentiates alpha 1-adrenergic receptor-induced transcriptional responses in neonatal rat ventricular myocytes.Consistent linkage of the long-QT syndrome to the Harvey ras-1 locus on chromosome 11Molecular genetic aspects of the Romano-Ward long QT syndrome.A Ras-dependent pathway regulates RNA polymerase II phosphorylation in cardiac myocytes: implications for cardiac hypertrophy.Pathogenesis and therapy of the idiopathic long QT syndrome.The nature and origin of spontaneous noise in G protein-gated ion channels.Involvement of the protein tyrosine phosphatase SHP-1 in Ras-mediated activation of the mitogen-activated protein kinase pathway.The Saccharomyces cerevisiae SDC25 C-domain gene product overcomes the dominant inhibitory activity of Ha-Ras Asn-17.A ras effector domain mutant which is temperature sensitive for cellular transformation: interactions with GTPase-activating protein and NF-1The posttranslational processing of ras p21 is critical for its stimulation of yeast adenylate cyclase.Specific changes of Ras GTPase-activating protein (GAP) and a GAP-associated p62 protein during calcium-induced keratinocyte differentiationras effector loop mutations that dissociate p120GAP and neurofibromin interactions.Biological characterization of Drosophila Rapgap1, a GTPase activating protein for Rap1Effector domain mutations dissociate p21ras effector function and GTPase-activating protein interaction.Ion channel regulation by Ras, Rho, and Rab small GTPases.Ca2+ channel activation by platelet-derived growth factor-induced tyrosine phosphorylation and Ras guanine triphosphate-binding proteins in rat glomerular mesangial cells.The biochemistry of ras p21.Heterologously expressed serotonin 1A receptors couple to muscarinic K+ channels in heartA novel GTPase-activating protein for R-Ras.The novel Rho GTPase-activating protein family protein, Rga8, provides a potential link between Cdc42/p21-activated kinase and Rho signaling pathways in the fission yeast, Schizosaccharomyces pombe.The role of Gln61 and Glu63 of Ras GTPases in their activation by NF1 and Ras GAP.Genetics and molecular biology of the inherited long QT syndrome.Mutational and kinetic analyses of the GTPase-activating protein (GAP)-p21 interaction: the C-terminal domain of GAP is not sufficient for full activity.
P2860
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P2860
ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
description
1990 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի հունիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1990
@ast
im Juni 1990 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 1990/06/01)
@sk
vědecký článek publikovaný v roce 1990
@cs
wetenschappelijk artikel (gepubliceerd op 1990/06/01)
@nl
наукова стаття, опублікована в червні 1990
@uk
مقالة علمية (نشرت في يونيو 1990)
@ar
name
ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
@ast
ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
@en
ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
@nl
type
label
ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
@ast
ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
@en
ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
@nl
prefLabel
ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
@ast
ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
@en
ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
@nl
P2093
P1433
P1476
ras p21 and GAP inhibit coupling of muscarinic receptors to atrial K+ channels
@en
P2093
A. M. Brown
F. McCormick
P. Polakis
R. Halenbeck
P304
P356
10.1016/0092-8674(90)90187-J
P407
P577
1990-06-01T00:00:00Z