A quantitative analysis of oligodendrocytes in multiple sclerosis lesions. A study of 113 cases
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The origins of concentric demyelination: self-organization in the human brainOligodendrocyte, Astrocyte, and Microglia Crosstalk in Myelin Development, Damage, and RepairRegenerative Capacity of Macrophages for RemyelinationPromoting remyelination in multiple sclerosis-recent advancesThe immunopathology of multiple sclerosis: an overviewGalanin transgenic mice with elevated circulating galanin levels alleviate demyelination in a cuprizone-induced MS mouse modelOligodendrocytes: biology and pathologyNudging oligodendrocyte intrinsic signaling to remyelinate and repair: Estrogen receptor ligand effects.CXCR2-positive neutrophils are essential for cuprizone-induced demyelination: relevance to multiple sclerosisRostrocaudal analysis of corpus callosum demyelination and axon damage across disease stages refines diffusion tensor imaging correlations with pathological features.Issues and practices in multiple sclerosis.Time-series modeling of multiple sclerosis disease activity: a promising window on disease progression and repair potential?CXCR2 signaling protects oligodendrocytes and restricts demyelination in a mouse model of viral-induced demyelination.Importance of oligodendrocyte protection, BBB breakdown and inflammation for remyelination.Oligodendroglia in cortical multiple sclerosis lesions decrease with disease progression, but regenerate after repeated experimental demyelinationSurprising pleiotropy of nerve growth factor in the treatment of experimental autoimmune encephalomyelitis.Drug treatment of multiple sclerosisPathogenic implications of iron accumulation in multiple sclerosis.Pathophysiology of translational regulation by microRNAs in multiple sclerosis.Periventricular demyelination and axonal pathology is associated with subependymal virus spread in a murine model for multiple sclerosis.Glial expression of tumor necrosis factor in transgenic animals: how do these models reflect the "normal situation"?The role of immune cells, glia and neurons in white and gray matter pathology in multiple sclerosis.Promoting remyelination: utilizing a viral model of demyelination to assess cell-based therapiesExtensive cortical remyelination in patients with chronic multiple sclerosis.The pathology of primary progressive multiple sclerosis.Association of chronic progressive neurological disease and ubiquitous viral agents: lessons from human herpesvirus 6 and multiple sclerosis.Reduced axonopathy and enhanced remyelination after chronic demyelination in fibroblast growth factor 2 (Fgf2)-null mice: differential detection with diffusion tensor imaging.Extracellular acidic pH inhibits oligodendrocyte precursor viability, migration, and differentiationIncreased expression of cystine/glutamate antiporter in multiple sclerosisOligodendrocytes and progenitors become progressively depleted within chronically demyelinated lesionsEndogenous cell repair of chronic demyelination.Clinical course, pathological correlations, and outcome of biopsy proved inflammatory demyelinating diseaseDiagnosis and disease modifying treatments in multiple sclerosis.Fibroblast growth factor 1 (FGFR1) modulation regulates repair capacity of oligodendrocyte progenitor cells following chronic demyelination.Cell therapy for multiple sclerosis.Quantitative ultrastructural analysis of a single spinal cord demyelinated lesion predicts total lesion load, axonal loss, and neurological dysfunction in a murine model of multiple sclerosisThe pathogenesis of multiple sclerosis: is it really a primary inflammatory process?Stem cell therapy for central nervous system demyelinating disease.IL-17-induced Act1-mediated signaling is critical for cuprizone-induced demyelinationDemyelination patterns in a mathematical model of multiple sclerosis.
P2860
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P2860
A quantitative analysis of oligodendrocytes in multiple sclerosis lesions. A study of 113 cases
description
1999 nî lūn-bûn
@nan
1999 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
A quantitative analysis of oli ...... lesions. A study of 113 cases
@ast
A quantitative analysis of oli ...... lesions. A study of 113 cases
@en
A quantitative analysis of oli ...... lesions. A study of 113 cases
@nl
type
label
A quantitative analysis of oli ...... lesions. A study of 113 cases
@ast
A quantitative analysis of oli ...... lesions. A study of 113 cases
@en
A quantitative analysis of oli ...... lesions. A study of 113 cases
@nl
altLabel
A quantitative analysis of oli ...... lesions: A study of 113 cases
@ast
A quantitative analysis of oli ...... lesions: A study of 113 cases
@en
prefLabel
A quantitative analysis of oli ...... lesions. A study of 113 cases
@ast
A quantitative analysis of oli ...... lesions. A study of 113 cases
@en
A quantitative analysis of oli ...... lesions. A study of 113 cases
@nl
P2093
P2860
P3181
P356
P1433
P1476
A quantitative analysis of oli ...... lesions. A study of 113 cases
@en
A quantitative analysis of oli ...... lesions: A study of 113 cases
@en
P2093
B Scheithauer
C Lucchinetti
C. Lucchinetti
H Lassmann
M Rodriguez
P2860
P304
P3181
P356
10.1093/BRAIN/122.12.2279
P407
P577
1999-12-01T00:00:00Z