Lipopolysaccharide binding protein and serum amyloid A secretion by human intestinal epithelial cells during the acute phase response
about
Dual role of lipopolysaccharide (LPS)-binding protein in neutralization of LPS and enhancement of LPS-induced activation of mononuclear cellsSerum amyloid A: an acute-phase protein involved in tumour pathogenesisCytokine modulation of liver annexin 1 expression during experimental endotoxemiaComparison of Cellular Uptake and Inflammatory Response via Toll-Like Receptor 4 to Lipopolysaccharide and Titanium Dioxide NanoparticlesCRISPLD2 is expressed at low levels during septic shock and is associated with procalcitoninStructure and Expression of Different Serum Amyloid A (SAA) Variants and their Concentration-Dependent Functions During Host InsultsLipopolysaccharide-binding protein for monitoring of postoperative sepsis: complemental to C-reactive protein or redundant?LPS-binding protein enables intestinal epithelial restitution despite LPS exposure.Micro-RNA-155 deficiency prevents alcohol-induced serum endotoxin increase and small bowel inflammation in mice.Markers of systemic exposures to products of intestinal bacteria in a dietary intervention study.Serum Amyloid A Induces Inflammation, Proliferation and Cell Death in Activated Hepatic Stellate Cells.Proliferation of intestinal crypt cells by gastrin-induced ornithine decarboxylaseReduction Impairs the Antibacterial Activity but Benefits the LPS Neutralization Ability of Human Enteric Defensin 5Serum lipopolysaccharide binding protein levels predict severity of lung injury and mortality in patients with severe sepsis.Obesity-associated cancer risk: the role of intestinal microbiota in the etiology of the host proinflammatory state.Serum amyloid A induces interleukin-6 in dermal fibroblasts via Toll-like receptor 2, interleukin-1 receptor-associated kinase 4 and nuclear factor-κB.Elevated levels of lipopolysaccharide-binding protein and soluble CD14 in plasma in neonatal early-onset sepsis.LPS-binding protein circulates in association with apoB-containing lipoproteins and enhances endotoxin-LDL/VLDL interaction.High-fat, high-fructose, high-cholesterol feeding causes severe NASH and cecal microbiota dysbiosis in juvenile Ossabaw swine.
P2860
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P2860
Lipopolysaccharide binding protein and serum amyloid A secretion by human intestinal epithelial cells during the acute phase response
description
1999 nî lūn-bûn
@nan
1999 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Lipopolysaccharide binding pro ...... uring the acute phase response
@ast
Lipopolysaccharide binding pro ...... uring the acute phase response
@en
Lipopolysaccharide binding pro ...... uring the acute phase response
@nl
type
label
Lipopolysaccharide binding pro ...... uring the acute phase response
@ast
Lipopolysaccharide binding pro ...... uring the acute phase response
@en
Lipopolysaccharide binding pro ...... uring the acute phase response
@nl
prefLabel
Lipopolysaccharide binding pro ...... uring the acute phase response
@ast
Lipopolysaccharide binding pro ...... uring the acute phase response
@en
Lipopolysaccharide binding pro ...... uring the acute phase response
@nl
P2093
P1476
Lipopolysaccharide binding pro ...... uring the acute phase response
@en
P2093
Buurman WA
Dentener MA
Vreugdenhil AC
P304
P407
P577
1999-09-01T00:00:00Z