Endocannabinoids control spasticity in a multiple sclerosis model
about
Whole plant cannabis extracts in the treatment of spasticity in multiple sclerosis: a systematic reviewThe endocannabinoid system as an emerging target of pharmacotherapyEvidence for the efficacy and effectiveness of THC-CBD oromucosal spray in symptom management of patients with spasticity due to multiple sclerosisPalmitoylethanolamide, a Natural Retinoprotectant: Its Putative Relevance for the Treatment of Glaucoma and Diabetic RetinopathyNeural Plasticity in Multiple Sclerosis: The Functional and Molecular BackgroundEffects of homologues and analogues of palmitoylethanolamide upon the inactivation of the endocannabinoid anandamideThe synthetic cannabinoid WIN55,212-2 attenuates hyperalgesia and allodynia in a rat model of neuropathic painEndocannabinoid system: emerging role from neurodevelopment to neurodegeneration.TASK1 modulates inflammation and neurodegeneration in autoimmune inflammation of the central nervous system.Neuropharmacology of the endocannabinoid signaling system-molecular mechanisms, biological actions and synaptic plasticity.The pharmacology of palmitoylethanolamide and first data on the therapeutic efficacy of some of its new formulations.New approaches in the management of spasticity in multiple sclerosis patients: role of cannabinoids.Neuroprotective therapies after perinatal hypoxic-ischemic brain injury.Modulation of cannabinoid receptor activation as a neuroprotective strategy for EAE and stroke.Enhancement of endocannabinoid signaling by fatty acid amide hydrolase inhibition: a neuroprotective therapeutic modalityThe endocannabinoid system as a target for the treatment of neurodegenerative disease.Control of spasticity in a multiple sclerosis model is mediated by CB1, not CB2, cannabinoid receptors.Cannabinoid control of neuroinflammation related to multiple sclerosis.Experimental autoimmune encephalomyelitis disrupts endocannabinoid-mediated neuroprotectionImmunoregulation of a viral model of multiple sclerosis using the synthetic cannabinoid R+WIN55,212Cannabinoids and neuroinflammation.New potent and selective inhibitors of anandamide reuptake with antispastic activity in a mouse model of multiple sclerosisManipulation of the endocannabinoid system by a general anaestheticAnandamide inhibits Theiler's virus induced VCAM-1 in brain endothelial cells and reduces leukocyte transmigration in a model of blood brain barrier by activation of CB(1) receptorsT-cell-mediated disruption of the neuronal microtubule network: correlation with early reversible axonal dysfunction in acute experimental autoimmune encephalomyelitis.Anandamide, Acting via CB2 Receptors, Alleviates LPS-Induced Neuroinflammation in Rat Primary Microglial Cultures.The endocannabinoid signaling system: a potential target for next-generation therapeutics for alcoholism.The effect of the palmitoylethanolamide analogue, palmitoylallylamide (L-29) on pain behaviour in rodent models of neuropathyEvaluation of the Effects of Sativex (THC BDS: CBD BDS) on Inhibition of Spasticity in a Chronic Relapsing Experimental Allergic Autoimmune Encephalomyelitis: A Model of Multiple Sclerosis.The therapeutic potential of drugs that target cannabinoid receptors or modulate the tissue levels or actions of endocannabinoids.Mast cell-glia axis in neuroinflammation and therapeutic potential of the anandamide congener palmitoylethanolamide.Direct suppression of autoreactive lymphocytes in the central nervous system via the CB2 receptorAn optimized GC-MS method detects nanomolar amounts of anandamide in mouse brain.Brain 2-Arachidonoylglycerol Levels Are Dramatically and Rapidly Increased Under Acute Ischemia-Injury Which Is Prevented by Microwave Irradiation.Oral Palmitoylethanolamide Treatment Is Associated with Reduced Cutaneous Adverse Effects of Interferon-β1a and Circulating Proinflammatory Cytokines in Relapsing-Remitting Multiple Sclerosis.P2X7 receptors control 2-arachidonoylglycerol production by microglial cells.The monoacylglycerol lipase inhibitor JZL184 attenuates LPS-induced increases in cytokine expression in the rat frontal cortex and plasma: differential mechanisms of actionCB2 cannabinoid receptors as an emerging target for demyelinating diseases: from neuroimmune interactions to cell replacement strategies.Modulation of the balance between cannabinoid CB(1) and CB(2) receptor activation during cerebral ischemic/reperfusion injury.TRPV1 gates tissue access and sustains pathogenicity in autoimmune encephalitis.
P2860
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P2860
Endocannabinoids control spasticity in a multiple sclerosis model
description
2001 nî lūn-bûn
@nan
2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Endocannabinoids control spasticity in a multiple sclerosis model
@ast
Endocannabinoids control spasticity in a multiple sclerosis model
@en
Endocannabinoids control spasticity in a multiple sclerosis model
@nl
type
label
Endocannabinoids control spasticity in a multiple sclerosis model
@ast
Endocannabinoids control spasticity in a multiple sclerosis model
@en
Endocannabinoids control spasticity in a multiple sclerosis model
@nl
prefLabel
Endocannabinoids control spasticity in a multiple sclerosis model
@ast
Endocannabinoids control spasticity in a multiple sclerosis model
@en
Endocannabinoids control spasticity in a multiple sclerosis model
@nl
P2093
P3181
P356
P1433
P1476
Endocannabinoids control spasticity in a multiple sclerosis model
@en
P2093
A Khanolkar
J L Croxford
R G Pertwee
V Di Marzo
P3181
P356
10.1096/FJ.00-0399FJE
P407
P577
2001-02-01T00:00:00Z