The effect of transient global ischemia on the interaction of Src and Fyn with the N-methyl-D-aspartate receptor and postsynaptic densities: possible involvement of Src homology 2 domains
about
Involvement of BDNF receptor TrkB in spatial memory formationTransient cerebral ischemia increases tyrosine phosphorylation of the synaptic RAS-GTPase activating protein, SynGAPDecay accelerating factor (CD55) protects neuronal cells from chemical hypoxia-induced injuryChronic zinc exposure decreases the surface expression of NR2A-containing NMDA receptors in cultured hippocampal neurons.Zinc induces a Src family kinase-mediated up-regulation of NMDA receptor activity and excitotoxicityInhibition of NR2B phosphorylation restores alterations in NMDA receptor expression and improves functional recovery following traumatic brain injury in micePhosphorylation of Tyrosine 1070 at the GluN2B Subunit Is Regulated by Synaptic Activity and Critical for Surface Expression of N-Methyl-D-aspartate (NMDA) ReceptorsFyn in Neurodevelopment and Ischemic Brain Injury.Protracted Tyrosine Phosphorylation of the Glutamate Receptor Subunit NR2 in the Rat Hippocampus Following Transient Cerebral Ischemia is Prevented by Intra-Ischemic Hypothermia.A use-dependent tyrosine dephosphorylation of NMDA receptors is independent of ion flux.Enhanced NMDA receptor tyrosine phosphorylation and increased brain injury following neonatal hypoxia-ischemia in mice with neuronal Fyn overexpression.Neuroprotection of GluR5-containing kainate receptor activation against ischemic brain injury through decreasing tyrosine phosphorylation of N-methyl-D-aspartate receptors mediated by Src kinase.Lymphocyte cell kinase activation mediates neuroprotection during ischemic preconditioning.Tyrosine phosphorylation of the N-methyl-D-aspartate receptor by exogenous and postsynaptic density-associated Src-family kinases.Differential effects of hypoxia-ischemia on subunit expression and tyrosine phosphorylation of the NMDA receptor in 7- and 21-day-old rats.Inhibition of Src activity decreases tyrosine phosphorylation of occludin in brain capillaries and attenuates increase in permeability of the blood-brain barrier after transient focal cerebral ischemia.Reappearance of hippocampal CA1 neurons after ischemia is associated with recovery of learning and memory.mGluR1 antagonist decreases tyrosine phosphorylation of NMDA receptor and attenuates infarct size after transient focal cerebral ischemia.S-nitrosylation of c-Src via NMDAR-nNOS module promotes c-Src activation and NR2A phosphorylation in cerebral ischemia/reperfusion.
P2860
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P2860
The effect of transient global ischemia on the interaction of Src and Fyn with the N-methyl-D-aspartate receptor and postsynaptic densities: possible involvement of Src homology 2 domains
description
1999 nî lūn-bûn
@nan
1999 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
The effect of transient global ...... ment of Src homology 2 domains
@ast
The effect of transient global ...... ment of Src homology 2 domains
@en
The effect of transient global ...... ment of Src homology 2 domains
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type
label
The effect of transient global ...... ment of Src homology 2 domains
@ast
The effect of transient global ...... ment of Src homology 2 domains
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The effect of transient global ...... ment of Src homology 2 domains
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prefLabel
The effect of transient global ...... ment of Src homology 2 domains
@ast
The effect of transient global ...... ment of Src homology 2 domains
@en
The effect of transient global ...... ment of Src homology 2 domains
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P2093
P2860
P1476
The effect of transient global ...... ment of Src homology 2 domains
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P2093
P2860
P356
10.1097/00004647-199908000-00007
P407
P577
1999-08-01T00:00:00Z
P5875
P6179
1051488343