Downregulation of E-cadherin and Desmoglein 1 by autocrine hepatocyte growth factor during melanoma development - Wikidata - awgldk - TriplyDB

Downregulation of E-cadherin and Desmoglein 1 by autocrine hepatocyte growth factor during melanoma development

Downregulation of E-cadherin and Desmoglein 1 by autocrine hepatocyte growth factor during melanoma development

http://www.wikidata.org/entity/Q28214978

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The MET axis as a therapeutic target.The Complexity of the ERK/MAP-Kinase Pathway and the Treatment of Melanoma Skin Cancer Melanoma: from mutations to medicine Alternative signaling pathways as potential therapeutic targets for overcoming EGFR and c-Met inhibitor resistance in non-small cell lung cancer Comprehensive analysis of receptor tyrosine kinase activation in human melanomas reveals autocrine signaling through IGF-1R Epithelial-to-mesenchymal transition and the cancer stem cell phenotype: insights from cancer biology with therapeutic implications for colorectal cancer.Hypoxia negatively regulates antimetastatic PEDF in melanoma cells by a hypoxia inducible factor-independent, autophagy dependent mechanism.Regulatory and functional connection of microphthalmia-associated transcription factor and anti-metastatic pigment epithelium derived factor in melanoma Modulation of extracellular matrix/adhesion molecule expression by BRG1 is associated with increased melanoma invasiveness.Phase 2 study of RO4929097, a gamma-secretase inhibitor, in metastatic melanoma: SWOG 0933.Identification of genes potentially regulated by human polynucleotide phosphorylase (hPNPase old-35) using melanoma as a model.The role of receptor tyrosine kinase inhibition in treating gastrointestinal malignancy.Function and regulation of melanoma-stromal fibroblast interactions: when seeds meet soil.The role of the c-Met pathway in lung cancer and the potential for targeted therapy.Proliferation of Hepatic Oval Cells via Cyclooxygenase-2 and Extracellular Matrix Protein Signaling during Liver Regeneration Following 2-AAF/Partial Hepatectomy in Rats.The melanocyte differentiation program predisposes to metastasis after neoplastic transformation.Rapid growth of invasive metastatic melanoma in carcinogen-treated hepatocyte growth factor/scatter factor-transgenic mice carrying an oncogenic CDK4 mutation.Identification of novel cellular targets in biliary tract cancers using global gene expression technology.Epigenetic Silencing of SPINT2 Promotes Cancer Cell Motility via HGF-MET Pathway Activation in Melanoma UV-induced skin cancer: similarities--variations.Molecular targets in melanoma: strategies and challenges for diagnosis and therapy.Fibronectin induction abrogates the BRAF inhibitor response of BRAF V600E/PTEN-null melanoma cells.Interplay between cadherins and α2β1 integrin differentially regulates melanoma cell invasion.Melanoma invasion - current knowledge and future directions.Cutaneous melanoma in genetically modified animals.Endothelin-3 stimulates survival of goblet cells in organotypic cultures of fetal human colonic epithelium.Recent discoveries in the genetics of melanoma and their therapeutic implications.Growth factors and oncogenes as targets in melanoma: lost in translation?Lipopolysaccharide-induced phosphorylation of c-Met tyrosine residue 1003 regulates c-Met intracellular trafficking and lung epithelial barrier function.Hepsin colocalizes with desmosomes and induces progression of ovarian cancer in a mouse model.Melanoma and the tumor microenvironment Long-lasting molecular changes in human skin after repetitive in situ UV irradiation.Active Notch1 confers a transformed phenotype to primary human melanocytes.Melanoma proteoglycan modifies gene expression to stimulate tumor cell motility, growth, and epithelial-to-mesenchymal transition Varying effects of EGF, HGF and TGFβ on formation of invadopodia and invasiveness of melanoma cell lines of different origin Interleukin-32α induces migration of human melanoma cells through downregulation of E-cadherin.Progress of antibody-based inhibitors of the HGF-cMET axis in cancer therapy.The inflammatory tumor microenvironment, epithelial mesenchymal transition and lung carcinogenesis.Insights into melanoma: targeting the mTOR pathway for therapeutics.The role of intracellular protein O-glycosylation in cell adhesion and disease.

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P2860

Downregulation of E-cadherin and Desmoglein 1 by autocrine hepatocyte growth factor during melanoma development

http://www.wikidata.org/entity/Q28214978

description

2001 nî lūn-bûn

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2001 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2001 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

@zh-hant

2001年論文

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2001年論文

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2001年論文

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2001年论文

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name

Downregulation of E-cadherin a ...... or during melanoma development

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Downregulation of E-cadherin a ...... or during melanoma development

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Downregulation of E-cadherin a ...... or during melanoma development

@nl

type

label

Downregulation of E-cadherin a ...... or during melanoma development

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Downregulation of E-cadherin a ...... or during melanoma development

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Downregulation of E-cadherin a ...... or during melanoma development

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prefLabel

Downregulation of E-cadherin a ...... or during melanoma development

@ast

Downregulation of E-cadherin a ...... or during melanoma development

@en

Downregulation of E-cadherin a ...... or during melanoma development

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Downregulation of E-cadherin a ...... or during melanoma development

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G Li

http://www.w3.org/2001/XMLSchema#string

K Hashimoto

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K Satyamoorthy

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M Herlyn

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Y Hanakawa

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P2860

Haploinsufficiency of desmoplakin causes a striate subtype of palmoplantar keratoderma

Molecular cloning and expression of human hepatocyte growth factor

The zinc-finger protein slug causes desmosome dissociation, an initial and necessary step for growth factor-induced epithelial-mesenchymal transition

Identification of the hepatocyte growth factor receptor as the c-met proto-oncogene product

Molecular cloning and amino acid sequence of human plakoglobin, the common junctional plaque protein

Making a connection: direct binding between keratin intermediate filaments and desmosomal proteins

Breaking the connection: displacement of the desmosomal plaque protein desmoplakin from cell-cell interfaces disrupts anchorage of intermediate filament bundles and alters intercellular junction assembly

The amino-terminal domain of desmoplakin binds to plakoglobin and clusters desmosomal cadherin-plakoglobin complexes

Defining the interactions between intermediate filaments and desmosomes

Association of the HGF/SF receptor, c-met, with the cell-surface adhesion molecule, E-cadherin, and catenins in human tumor cells

P2888

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8125-35

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scholarly article

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2038759

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10.1038/SJ.ONC.1205034

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P433

56

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P478

20

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Helmut Schaider

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2001-12-06T00:00:00Z

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11576036

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1028697716

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11781826

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