The expression of the chemorepellent Semaphorin 3A is selectively induced in terminal Schwann cells of a subset of neuromuscular synapses that display limited anatomical plasticity and enhanced vulnerability in motor neuron disease
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Major Histocompatibility Complex I Expression by Motor Neurons and Its Implication in Amyotrophic Lateral SclerosisEphrin-A3 promotes and maintains slow muscle fiber identity during postnatal development and reinnervation.The complex molecular biology of amyotrophic lateral sclerosis (ALS)ALS as a distal axonopathy: molecular mechanisms affecting neuromuscular junction stability in the presymptomatic stages of the diseaseAltered Satellite Cell Responsiveness and Denervation Implicated in Progression of Rotator-Cuff InjuryBeyond Parkinson disease: amyotrophic lateral sclerosis and the axon guidance pathwayThe role of oxidative stress in degeneration of the neuromuscular junction in amyotrophic lateral sclerosisDeregulated expression of cytoskeleton related genes in the spinal cord and sciatic nerve of presymptomatic SOD1(G93A) Amyotrophic Lateral Sclerosis mouse model.Semaphorin 3A signaling through neuropilin-1 is an early trigger for distal axonopathy in the SOD1G93A mouse model of amyotrophic lateral sclerosisSemaphorin 3A inhibits growth of adult sympathetic and parasympathetic neurones via distinct cyclic nucleotide signalling pathwaysProgress in therapy development for amyotrophic lateral sclerosisDecorin, erythroblastic leukaemia viral oncogene homologue B4 and signal transducer and activator of transcription 3 regulation of semaphorin 3A in central nervous system scar tissueDelayed disease onset and extended survival in the SOD1G93A rat model of amyotrophic lateral sclerosis after suppression of mutant SOD1 in the motor cortexEarly gene expression changes in spinal cord from SOD1(G93A) Amyotrophic Lateral Sclerosis animal model.Semaphorins in axon regeneration: developmental guidance molecules gone wrong?Accelerating axonal growth promotes motor recovery after peripheral nerve injury in miceAnti-Semaphorin 3A neutralization monoclonal antibody prevents sepsis development in lipopolysaccharide-treated mice.Ciliary neurotrophic factor is not required for terminal sprouting and compensatory reinnervation of neuromuscular synapses: re-evaluation of CNTF null mice.Dysregulated expression of death, stress and mitochondrion related genes in the sciatic nerve of presymptomatic SOD1(G93A) mouse model of Amyotrophic Lateral Sclerosis.Molecular signatures of amyotrophic lateral sclerosis disease progression in hind and forelimb muscles of an SOD1(G93A) mouse modelExpression of a Mutant SEMA3A Protein with Diminished Signalling Capacity Does Not Alter ALS-Related Motor Decline, or Confer Changes in NMJ Plasticity after BotoxA-Induced Paralysis of Male Gastrocnemic MuscleOpening Pandora's jar: a primer on the putative roles of CRMP2 in a panoply of neurodegenerative, sensory and motor neuron, and central disorders.Axon Guidance Molecules and Neural Circuit Remodeling After Spinal Cord Injury.Molecular/genetic manipulation of extrinsic axon guidance factors for CNS repair and regeneration.Schwann cells expressing dismutase active mutant SOD1 unexpectedly slow disease progression in ALS mice.Semaphorin function in neural plasticity and disease.Age- and Activity-Related Differences in the Abundance of Myosin Essential and Regulatory Light Chains in Human Muscle.The role of immune semaphorins in multiple sclerosis.Neuron-glia interactions: the roles of Schwann cells in neuromuscular synapse formation and functionClass 3 semaphorins as a therapeutic target.The emerging role of class-3 semaphorins and their neuropilin receptors in oncology.Transcriptional analysis reveals distinct subtypes in amyotrophic lateral sclerosis: implications for personalized therapy.Perisynaptic Schwann Cells at the Neuromuscular Synapse: Adaptable, Multitasking Glial Cells.The Role of Skeletal Muscle in Amyotrophic Lateral Sclerosis.The role of semaphorin3A in myogenic regeneration and the formation of functional neuromuscular junctions on new fibres.The Axon Guidance Protein Semaphorin 3A Is Increased in the Motor Cortex of Patients With Amyotrophic Lateral Sclerosis.New perspectives on amyotrophic lateral sclerosis: the role of glial cells at the neuromuscular junction.Terminal Schwann Cells Lead Synapse Remodelling following Injury(1,2).Label-Free LC-MS/MS Proteomic Analysis of Cerebrospinal Fluid Identifies Protein/Pathway Alterations and Candidate Biomarkers for Amyotrophic Lateral SclerosisIdentification of 17 Highly Expressed Genes within Mouse Lumbar Spinal Cord Anterior Horn Region from an In-Situ Hybridization Atlas of 3430 Genes: Implications for Motor Neuron Disease.
P2860
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P2860
The expression of the chemorepellent Semaphorin 3A is selectively induced in terminal Schwann cells of a subset of neuromuscular synapses that display limited anatomical plasticity and enhanced vulnerability in motor neuron disease
description
2006 nî lūn-bûn
@nan
2006 թուականին հրատարակուած գիտական յօդուած
@hyw
2006 թվականին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
The expression of the chemorep ...... bility in motor neuron disease
@ast
The expression of the chemorep ...... bility in motor neuron disease
@en
The expression of the chemorep ...... bility in motor neuron disease
@nl
type
label
The expression of the chemorep ...... bility in motor neuron disease
@ast
The expression of the chemorep ...... bility in motor neuron disease
@en
The expression of the chemorep ...... bility in motor neuron disease
@nl
prefLabel
The expression of the chemorep ...... bility in motor neuron disease
@ast
The expression of the chemorep ...... bility in motor neuron disease
@en
The expression of the chemorep ...... bility in motor neuron disease
@nl
P2093
P3181
P1476
The expression of the chemorep ...... bility in motor neuron disease
@en
P2093
Floor J Stam
Fred De Winter
Freek L van Muiswinkel
Joost Verhaagen
Liselijn A B Wisman
Peter R Bär
P304
P3181
P356
10.1016/J.MCN.2006.03.002
P407
P577
2006-05-03T00:00:00Z