Quantitative post-mortem study of the hippocampus in chronic epilepsy: seizures do not inevitably cause neuronal loss
about
sameAs
Classic hippocampal sclerosis and hippocampal-onset epilepsy produced by a single “cryptic” episode of focal hippocampal excitation in awake ratsCellular injury and neuroinflammation in children with chronic intractable epilepsy.Hippocampal neuropathology of domoic acid-induced epilepsy in California sea lions (Zalophus californianus).Neuropathologic and clinical features of human medial temporal lobe epilepsy.Temporal lobe interictal epileptic discharges affect cerebral activity in "default mode" brain regionsReview: Hippocampal sclerosis in epilepsy: a neuropathology review.The effects of aqueous extract of Boswellia Serrata on hippocampal region CA1 and learning deficit in kindled rats.Progressive brain damage, synaptic reorganization and NMDA activation in a model of epileptogenic cortical dysplasia.Neurofibrillary tangle pathology and Braak staging in chronic epilepsy in relation to traumatic brain injury and hippocampal sclerosis: a post-mortem studyA new clinico-pathological classification system for mesial temporal sclerosis.Gene expression in temporal lobe epilepsy is consistent with increased release of glutamate by astrocytes.Beyond the CA1 subfield: Local hippocampal shape changes in MRI-negative temporal lobe epilepsy.Cognitive Dysfunction and Hippocampal Damage Induced by Hypoxic-Ischemic Brain Injury and Prolonged Febrile Convulsions in Immature RatsRemote effects of hippocampal sclerosis on effective connectivity during working memory encoding: a case of connectional diaschisis?Correlation of quantitative MRI and neuropathology in epilepsy surgical resection specimens--T2 correlates with neuronal tissue in gray matter.Somatostatin and neuropeptide Y neurons undergo different plasticity in parahippocampal regions in kainic acid-induced epilepsy.Sequel of spontaneous seizures after kainic acid-induced status epilepticus and associated neuropathological changes in the subiculum and entorhinal cortexEpilepsy: a review of selected clinical syndromes and advances in basic science.Echinoderm microtubule-associated protein -like protein 5 in anterior temporal neocortex of patients with intractable epilepsyVariability of sclerosis along the longitudinal hippocampal axis in epilepsy: a post mortem study.Time course and mechanism of hippocampal neuronal death in an in vitro model of status epilepticus: role of NMDA receptor activation and NMDA dependent calcium entry.Stimulation of Anterior Thalamic Nuclei Protects Against Seizures and Neuronal Apoptosis in Hippocampal CA3 Region of Kainic Acid-induced Epileptic Rats.Molecular neuropathology of temporal lobe epilepsy: complementary approaches in animal models and human disease tissue.In vitro status epilepticus but not spontaneous recurrent seizures cause cell death in cultured hippocampal neurons.Neurogenesis and epilepsy in the developing brainMore Severe Extratemporal Damages in Mesial Temporal Lobe Epilepsy With Hippocampal Sclerosis Than That With Other Lesions: A Multimodality MRI Study.Presurgical connectome and postsurgical seizure control in temporal lobe epilepsyBilateral reorganization of the dentate gyrus in hippocampal sclerosis: a postmortem study.Regional thalamic neuropathology in patients with hippocampal sclerosis and epilepsy: a postmortem studyVascular endothelial growth factor attenuates status epilepticus-induced behavioral impairments in rats.Sudden unexpected death in epilepsy in childhood.Surgical and postmortem pathology studies: contribution for the investigation of temporal lobe epilepsy.Epilepsies associated with hippocampal sclerosis.Modulation of medial temporal lobe activity in epilepsy patients with hippocampal sclerosis during verbal working memory.Anti-epileptic drugs in pediatric traumatic brain injury.Neurofilament heavy chain and heat shock protein 70 as markers of seizure-related brain injury.Short- and long-term surgical outcomes of temporal lobe epilepsy associated with hippocampal sclerosis: Relationships with neuropathology.Early tissue damage and microstructural reorganization predict disease severity in experimental epilepsy.Activation of mTOR signaling pathway is secondary to neuronal excitability in a mouse model of mesio-temporal lobe epilepsy.Do single seizures cause neuronal death in the human hippocampus?
P2860
Q29037057-1FA859C0-FB5F-4F35-AE73-56C4CE734FCEQ33611760-9C49BF88-427C-4FDD-95D3-B3278DDAD632Q33909109-F85A99F4-FDA0-4A31-8CB8-2DD75606541CQ33953722-52805C36-F4CB-4A50-9E6E-4B707D021CD9Q34167789-A95CF50F-4079-49D0-9574-5BAF2C2362EDQ34692930-BEE8D675-5054-438A-849D-397D37B9E1A1Q35052172-BB92B039-8FDD-44C9-96F3-0F48C725BC55Q35108304-3EFD9289-6EB2-4144-98E6-6B32E8682BC2Q35275393-0EAE3D3B-3A67-4ADB-9702-8BB5FC4E3EEDQ35630377-6797438E-9255-4B7E-80C4-69994B45E516Q35795750-F210633A-7B67-4C01-8547-88A9CC8B4FD6Q35923047-9DD317F3-A212-498F-B8B7-222FA9377C14Q35947639-3AAA4EB4-7E12-4E74-A25B-52332449F557Q35976184-2840A0BB-8E4B-4303-BEA1-3943A3EC29F3Q35980955-B3D53A76-2E7F-49B2-9368-6ACC1BC3C62DQ36103127-7902EABE-86F0-483F-9948-ABBDCC92DE16Q36133193-AF2DE5D7-A655-451D-93AB-A600F2DA16E5Q36377765-16FBD422-0918-452B-B3C6-E07DD8C4C8B6Q36391846-50CDCE4D-AA7F-4907-9905-E2BB22173875Q36406715-060E641E-82E9-45E6-B633-67439FB3D318Q36571496-136F8CBB-A3FD-475A-9D77-5B47EDB174FEQ36797830-43499EDC-7034-4CF0-A351-E4225DA9058EQ36850229-85D639A2-1A17-41F7-96DF-2EE69BB7C3A0Q37045489-3E00D5FE-3527-4A25-943C-5D4A44B14C15Q37181305-AB39AEDC-3136-4BCE-98AA-3C6E239670CCQ37207115-7208AE71-FB6D-4825-B2D1-1F5F22D88959Q37265424-4C489FE3-5441-43EF-8B29-BECA8A47318BQ37368344-BD6D8F8D-8DC3-4A5F-AAB9-DB79ABA046BFQ37715667-AA1435C1-29D0-4E9F-BE97-E963ECFC9FD0Q37783830-2625D78A-ACBF-45CF-AD21-410570E89C9EQ37875775-9A00604E-8E48-4FAD-A408-6A5377209775Q38072199-DDECAE40-F442-43E0-9AD3-3B28C9934DA0Q38211467-6487B855-C854-485F-99A4-8456AEEA6774Q38380468-B7CB6EA4-3002-416C-B666-AF7D6CFEE743Q38862827-CA934F41-BBE0-4745-820B-844EF30002FDQ39628369-074E387E-D84C-4B8A-8EE7-79DF30E34CC4Q40202396-29C911EB-CC57-4C70-9A70-751100E1A1B4Q41138308-7F5AC95E-84B5-40F1-8DA8-FB786720E07BQ41553148-DEF5A50A-3602-4EE4-BFD6-E4D8B9A4C52AQ41859837-7DED7A22-10F2-478D-AF87-FE38BE773A47
P2860
Quantitative post-mortem study of the hippocampus in chronic epilepsy: seizures do not inevitably cause neuronal loss
description
2005 nî lūn-bûn
@nan
2005 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Quantitative post-mortem study ...... inevitably cause neuronal loss
@ast
Quantitative post-mortem study ...... inevitably cause neuronal loss
@en
Quantitative post-mortem study ...... inevitably cause neuronal loss
@nl
type
label
Quantitative post-mortem study ...... inevitably cause neuronal loss
@ast
Quantitative post-mortem study ...... inevitably cause neuronal loss
@en
Quantitative post-mortem study ...... inevitably cause neuronal loss
@nl
prefLabel
Quantitative post-mortem study ...... inevitably cause neuronal loss
@ast
Quantitative post-mortem study ...... inevitably cause neuronal loss
@en
Quantitative post-mortem study ...... inevitably cause neuronal loss
@nl
P2093
P356
P1433
P1476
Quantitative post-mortem study ...... inevitably cause neuronal loss
@en
P2093
Jiemin Zhou
Lillian Martinian
Maria Thom
Sanjay Sisodiya
P304
P356
10.1093/BRAIN/AWH475
P407
P577
2005-06-01T00:00:00Z