c-Jun activation is associated with proliferation and angiogenesis in invasive breast cancer
about
Critical role of c-Jun overexpression in liver metastasis of human breast cancer xenograft modelA role of fukutin, a gene responsible for Fukuyama type congenital muscular dystrophy, in cancer cells: a possible role to suppress cell proliferationIn situ proximity ligation detection of c-Jun/AP-1 dimers reveals increased levels of c-Jun/Fra1 complexes in aggressive breast cancer cell lines in vitro and in vivoc-JUN prevents methylation of p16(INK4a) (and Cdk6): the villain turned bodyguardc-Jun induces mammary epithelial cellular invasion and breast cancer stem cell expansion.Cap-Independent Translational Control of CarcinogenesisTargeting c-Jun and JunB proteins as potential anticancer cell therapyp21-activated kinase 3 (PAK3) is an AP-1 regulated gene contributing to actin organisation and migration of transformed fibroblastsSrc Inhibition Can Synergize with Gemcitabine and Reverse Resistance in Triple Negative Breast Cancer Cells via the AKT/c-Jun PathwayActivator protein 1 (AP-1) contributes to EpCAM-dependent breast cancer invasion.Somatic excision demonstrates that c-Jun induces cellular migration and invasion through induction of stem cell factor.Prolactin cooperates with loss of p53 to promote claudin-low mammary carcinomasc-Jun regulates phosphoinositide-dependent kinase 1 transcription: implication for Akt and protein kinase C activities and melanoma tumorigenesisExpression levels of transcription factors c-Fos and c-Jun and transmembrane protein HAb18G/CD147 in urothelial carcinoma of the bladder.C-jun inhibits mammary apoptosis in vivo.Emerging roles of ATF2 and the dynamic AP1 network in cancer.Beneficial bacteria stimulate host immune cells to counteract dietary and genetic predisposition to mammary cancer in mice.Impaired CK1 delta activity attenuates SV40-induced cellular transformation in vitro and mouse mammary carcinogenesis in vivoSafety and tolerability of an intratumorally injected DNAzyme, Dz13, in patients with nodular basal-cell carcinoma: a phase 1 first-in-human trial (DISCOVER)Genetic variants in the H2AFX promoter region are associated with risk of sporadic breast cancer in non-Hispanic white women aged <or=55 yearsTransgenic expression of polyomavirus middle T antigen in the mouse prostate gives rise to carcinoma.Interaction between c-jun and androgen receptor determines the outcome of taxane therapy in castration resistant prostate cancer.c-Jun promotes cell migration and drives expression of the motility factor ENPP2 in soft tissue sarcomasM-Ras induces Ral and JNK activation to regulate MEK/ERK-independent gene expression in MCF-7 breast cancer cells.Oleanolic acid induces migration in Mv1Lu and MDA-MB-231 epithelial cells involving EGF receptor and MAP kinases activation.ETV6-NTRK3 fusion oncogene initiates breast cancer from committed mammary progenitors via activation of AP1 complex.The oncogenic transcription factor c-Jun regulates glutaminase expression and sensitizes cells to glutaminase-targeted therapy.Understanding signaling cascades in melanomaTumour sampling method can significantly influence gene expression profiles derived from neoadjuvant window studies.COP1 and GSK3β cooperate to promote c-Jun degradation and inhibit breast cancer cell tumorigenesis.Mitochondrial dysfunction and reactive oxygen species imbalance promote breast cancer cell motility through a CXCL14-mediated mechanismCyclophosphamide alters the gene expression profile in patients treated with high doses prior to stem cell transplantation.Essential Roles of E3 Ubiquitin Ligases in p53 Regulation.Key genes and pathways predicted in papillary thyroid carcinoma based on bioinformatics analysis.Identification of critical genes and gene interaction networks that mediate osteosarcoma metastasis to the lungs.Biliverdin reductase: a target for cancer therapy?c-Jun Contributes to Transcriptional Control of GNA12 Expression in Prostate Cancer Cells.JunD Is Required for Proliferation of Prostate Cancer Cells and Plays a Role in Transforming Growth Factor-β (TGF-β)-induced Inhibition of Cell Proliferation.KRIT1 loss of function causes a ROS-dependent upregulation of c-JunNANOG upregulates c-Jun oncogene expression through binding the c-Jun promoter.
P2860
Q21260411-A072C1D5-E5B0-4BFA-B86B-A4AC5E6DDBB5Q24314890-DF493225-AB69-4FA8-9952-6B6B26F7CAA2Q24315958-A9A3EC7B-8360-4354-9C88-8829E529E180Q24617313-99CEFFAC-E258-48AC-954A-FD154855FC61Q24633165-7DA4F1B2-97D1-4F1B-BD3F-6AA8B938714CQ26744030-DC6B7479-1ED0-4A76-BA20-F473F1BAF1A8Q28238777-2FF57A3A-4C1B-4F0C-8294-4B59C686FF6AQ28293789-CC863A66-F694-4B3A-9342-E6E2E8EFB9A8Q28468471-463A5E29-F169-4300-B6EA-84B9B6CD78EDQ30427356-12DA625A-49E7-4FBF-8D16-DD582FAE1BC9Q30479005-DC939B06-54E3-41EA-9237-909C1756BDC8Q33555787-EF5C2842-6EE6-4EEE-8B7E-7F4F2F656444Q33569860-30679076-8D6B-407F-A007-24E6F5E2CE5DQ33672321-B677FB36-7749-4ED9-8020-CE64FDCD87B7Q33711758-08687ADD-1683-4D26-AAEB-7E279B43039FQ33866973-E67DE8B2-D275-46FC-980E-C20255B5AC0AQ34039445-D02839D4-590E-44F6-ADF0-89056091C1DEQ34123850-EEEA8788-0E57-4B9D-B5A5-3D89BB20FD2CQ34343851-31781DE6-6223-4B7B-921B-E251330432ADQ34537469-D2541B41-B8F4-44BD-934B-665EB05A1A3DQ34982720-E6FA23F7-9096-4429-97E0-D598561F59F1Q35048550-39686B00-925F-478F-B01A-CC41A75B34A3Q35756057-C32D3AE9-81E5-4809-8885-7233010111C7Q35830796-E550E50E-B2C4-4DD7-B1E6-6CABDC7FC22FQ36288371-98179948-6F28-48FC-9A5C-1935F526ECC9Q36328085-723ADA3E-4BA3-40FF-899F-895FE3F81974Q36815149-68194D15-0624-4B3A-87B3-409ECC13ABFBQ37034487-58B859A4-E3CF-45FD-A891-D56C1B31E159Q37072929-8C963C53-DD01-4545-B984-E712A8CFB99FQ37162398-3A9AC96A-D82E-4CFC-8607-261ED6E2C06BQ37383628-973AB030-2B8A-4095-B0A1-294AB4105A69Q37505854-9BDE74E4-8D47-4795-89AC-90844E2111E0Q37691041-DC0443B3-C11D-4DFF-82E4-B6F655EE98A7Q38441699-98115972-E7BE-4F27-AC41-DDCD05196204Q38453976-ED253E9A-9F84-439B-9FAB-0AE89521F3B0Q38531952-AB5DFF88-21C8-4DD7-B044-0E3D9A22D994Q38708261-047785C5-7AFE-4777-B84A-2F64113835AAQ38760746-95CFDF52-011C-4EFD-A576-BF86644E753EQ38873324-8B140C05-3DFE-47A4-B4DF-05DAB148F9AEQ38957311-26F24AFF-2115-41B1-AD09-858387F9DAA3
P2860
c-Jun activation is associated with proliferation and angiogenesis in invasive breast cancer
description
2006 nî lūn-bûn
@nan
2006 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
c-Jun activation is associated ...... esis in invasive breast cancer
@ast
c-Jun activation is associated ...... esis in invasive breast cancer
@en
c-Jun activation is associated ...... esis in invasive breast cancer
@nl
type
label
c-Jun activation is associated ...... esis in invasive breast cancer
@ast
c-Jun activation is associated ...... esis in invasive breast cancer
@en
c-Jun activation is associated ...... esis in invasive breast cancer
@nl
prefLabel
c-Jun activation is associated ...... esis in invasive breast cancer
@ast
c-Jun activation is associated ...... esis in invasive breast cancer
@en
c-Jun activation is associated ...... esis in invasive breast cancer
@nl
P2093
P3181
P1433
P1476
c-Jun activation is associated ...... esis in invasive breast cancer
@en
P2093
Astrid E Greijer
Elsken van der Wall
Marije M Vleugel
Paul J van Diest
Reinhard Bos
P304
P3181
P356
10.1016/J.HUMPATH.2006.01.022
P407
P577
2006-06-01T00:00:00Z