about
Autoimmunity to two forms of glutamate decarboxylase in insulin-dependent diabetes mellitusShared HLA class II-associated genetic susceptibility and resistance, related to the HLA-DQB1 gene, in IgA deficiency and common variable immunodeficiencyHuman major histocompatibility complex contains a new cluster of genes between the HLA-D and complement C4 lociParasitic worms and inflammatory diseasesHLA DPA1, DPB1 alleles and haplotypes contribute to the risk associated with type 1 diabetes: analysis of the type 1 diabetes genetics consortium families.Genetic analysis of type 1 diabetes using whole genome approachesDefining the role of the MHC in autoimmunity: a review and pooled analysisAssociation of hypoxia inducible factor-1 alpha gene polymorphism with both type 1 and type 2 diabetes in a Caucasian (Hungarian) sample.The familial nature of rheumatoid arthritis.Atopy, airway responsiveness, and genes.Environmental factors in the etiology of type 1 diabetes, celiac disease, and narcolepsy.Major histocompatibility complex class III genes and susceptibility to immunoglobulin A deficiency and common variable immunodeficiency.Investigation of the mode of inheritance of insulin-dependent diabetes mellitus in Japanese subjectsMultireactive pattern of serum autoantibodies in asymptomatic individuals with immunoglobulin A deficiencyHigh incidence of glucose intolerance in Vogt-Koyanagi-Harada diseaseGenetic and physiological association of diabetes susceptibility with raised Na+/H+ exchange activity.FokI polymorphism, vitamin D receptor, and interleukin-1 receptor haplotypes are associated with type 1 diabetes in the Dalmatian population.NeuroD1 gene and interleukin-18 gene polymorphisms in type 1 diabetes in Dalmatian population of Southern Croatia.The genetics of type I (insulin dependent) diabetes mellitus.CD4+ beta islet cell-reactive T cell clones that suppress autoimmune diabetes in nonobese diabetic mice.Prevention of insulin-dependent diabetes mellitus in nonobese diabetic mice by immunogenic but not by tolerated peptides.In vivo evidence for the contribution of human histocompatibility leukocyte antigen (HLA)-DQ molecules to the development of diabetes.An Abd transgene prevents diabetes in nonobese diabetic mice by inducing regulatory T cells.Induction of insulitis by glutamic acid decarboxylase peptide-specific and HLA-DQ8-restricted CD4(+) T cells from human DQ transgenic mice.Serologic laboratory findings in malignancy.WIN 54954 treatment of mice infected with a diabetogenic strain of group B coxsackievirusImmunological studies on type 1 diabetes in identical twinsInterleukin-1 promotes hyperglycemia and insulitis in mice normally resistant to streptozotocin-induced diabetes.Peptide presentation by HLA-DQ molecules is associated with the development of immune tolerance.
P2860
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P2860
description
1990 nî lūn-bûn
@nan
1990 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Genetic control of autoimmunity in type 1 diabetes
@ast
Genetic control of autoimmunity in type 1 diabetes
@en
Genetic control of autoimmunity in type 1 diabetes
@nl
type
label
Genetic control of autoimmunity in type 1 diabetes
@ast
Genetic control of autoimmunity in type 1 diabetes
@en
Genetic control of autoimmunity in type 1 diabetes
@nl
prefLabel
Genetic control of autoimmunity in type 1 diabetes
@ast
Genetic control of autoimmunity in type 1 diabetes
@en
Genetic control of autoimmunity in type 1 diabetes
@nl
P1433
P1476
Genetic control of autoimmunity in type 1 diabetes
@en
P2093
P407
P577
1990-04-01T00:00:00Z