Histopathology and clinical course of MOG-antibody-associated encephalomyelitis - Wikidata - awgldk - TriplyDB

Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

http://www.wikidata.org/entity/Q28259551

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Antibody biomarkers in CNS demyelinating diseases - a long and winding road Fulminant demyelinating encephalomyelitis: Insights from antibody studies and neuropathology Children with multiphasic disseminated encephalomyelitis and antibodies to the myelin oligodendrocyte glycoprotein (MOG): Extending the spectrum of MOG antibody positive diseases Autoantibodies to MOG in a distinct subgroup of adult multiple sclerosis MOG-IgG in NMO and related disorders: a multicenter study of 50 patients. Part 2: Epidemiology, clinical presentation, radiological and laboratory features, treatment responses, and long-term outcome Myelin Oligodendrocyte Glycoprotein: Deciphering a Target in Inflammatory Demyelinating Diseases.Influence of type I IFN signaling on anti-MOG antibody-mediated demyelination.Screening for MOG-IgG and 27 other anti-glial and anti-neuronal autoantibodies in 'pattern II multiple sclerosis' and brain biopsy findings in a MOG-IgG-positive case.Inflammatory demyelination without astrocyte loss in MOG antibody-positive NMOSD Usefulness of optical coherence tomography to distinguish optic neuritis associated with AQP4 or MOG in neuromyelitis optica spectrum disorders.MOG-IgG in NMO and related disorders: a multicenter study of 50 patients. Part 1: Frequency, syndrome specificity, influence of disease activity, long-term course, association with AQP4-IgG, and origin.MOG-IgG in NMO and related disorders: a multicenter study of 50 patients. Part 4: Afferent visual system damage after optic neuritis in MOG-IgG-seropositive versus AQP4-IgG-seropositive patients.MOG-IgG in NMO and related disorders: a multicenter study of 50 patients. Part 3: Brainstem involvement - frequency, presentation and outcome.Myelinosome formation represents an early stage of oligodendrocyte damage in multiple sclerosis and its animal model.Finding NMO: The Evolving Diagnostic Criteria of Neuromyelitis Optica.MOG antibody-positive, benign, unilateral, cerebral cortical encephalitis with epilepsy.MOG antibody-related disorders: common features and uncommon presentations.Can new chemical therapies improve the management of multiple sclerosis in children?Pattern II and pattern III MS are entities distinct from pattern I MS: evidence from cerebrospinal fluid analysis.ADEM-like presentation, anti-MOG antibodies, and MS pathology: TWO case reports.MOG antibody seropositivity in a patient with encephalitis: beyond the classical syndrome.Human antibodies against the myelin oligodendrocyte glycoprotein can cause complement-dependent demyelination.Neuromyelitis Optica: Deciphering a Complex Immune-Mediated Astrocytopathy.Clinical spectrum and IgG subclass analysis of anti-myelin oligodendrocyte glycoprotein antibody-associated syndromes: a multicenter study.Importance of Regular and Maintenance Therapy Adherence in Neuromyelitis Optica (NMO): Lessons from a Repeating Relapse Case.Mechanisms for lesion localization in neuromyelitis optica spectrum disorders.Differences in the Reponses to Apheresis Therapy of Patients With 3 Histopathologically Classified Immunopathological Patterns of Multiple Sclerosis.Myelin-oligodendrocyte-glycoprotein (MOG) autoantibodies as potential markers of severe optic neuritis and subclinical retinal axonal degeneration.Defining distinct features of anti-MOG antibody associated central nervous system demyelination.MOG encephalomyelitis: international recommendations on diagnosis and antibody testing.Progressive Demyelination in the Presence of Serum Myelin Oligodendrocyte Glycoprotein-IgG: A Case Report.MOG-IgG-Associated Optic Neuritis, Encephalitis, and Myelitis: Lessons Learned From Neuromyelitis Optica Spectrum Disorder.Diagnosis and Treatment of NMO Spectrum Disorder and MOG-Encephalomyelitis Optical coherence tomography in central nervous system demyelinating diseases related optic neuritis

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Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

http://www.wikidata.org/entity/Q28259551

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2015 nî lūn-bûn

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2015 թուականի Մարտին հրատարակուած գիտական յօդուած

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2015 թվականի մարտին հրատարակված գիտական հոդված

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2015年の論文

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Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

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Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

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Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

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Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

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Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

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Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

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Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

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Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

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Annals of clinical and translational neurology

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Histopathology and clinical course of MOG-antibody-associated encephalomyelitis

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Andreas Straube

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Birgit Ertl-Wagner

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Constanze Breithaupt

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Reinhard Hohlfeld

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Sarah Laurent

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P2860

Myelin-oligodendrocyte glycoprotein antibodies in adults with a neuromyelitis optica phenotype

The spectrum of MOG autoantibody-associated demyelinating diseases

Distinction between MOG antibody-positive and AQP4 antibody-positive NMO spectrum disorders

Heterogeneity of multiple sclerosis lesions: implications for the pathogenesis of demyelination

Overlapping demyelinating syndromes and anti–N-methyl-D-aspartate receptor encephalitis.

Presence of six different lesion types suggests diverse mechanisms of tissue injury in neuromyelitis optica

Antibodies to MOG have a demyelination phenotype and affect oligodendrocyte cytoskeleton.

Neuromyelitis optica spectrum disorders with aquaporin-4 and myelin-oligodendrocyte glycoprotein antibodies: a comparative study.

Complement activating antibodies to myelin oligodendrocyte glycoprotein in neuromyelitis optica and related disorders

Glycoproteins as targets of autoantibodies in CNS inflammation: MOG and more.

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