Regulation of cardiac gene expression during myocardial growth and hypertrophy: molecular studies of an adaptive physiologic response
about
Expression cloning of cardiotrophin 1, a cytokine that induces cardiac myocyte hypertrophyThe neuron-restrictive silencer element-neuron-restrictive silencer factor system regulates basal and endothelin 1-inducible atrial natriuretic peptide gene expression in ventricular myocytesFocal adhesion kinase and p130Cas mediate both sarcomeric organization and activation of genes associated with cardiac myocyte hypertrophyCardiac hypertrophy with preserved contractile function after selective deletion of GLUT4 from the heartNRSF regulates the fetal cardiac gene program and maintains normal cardiac structure and functionMolecular and cellular mechanisms of cardiotoxicity.Calreticulin induces dilated cardiomyopathyFluorescence-based force/tension sensors: a novel tool to visualize mechanical forces in structural proteins in live cellsSerine-910 phosphorylation of focal adhesion kinase is critical for sarcomere reorganization in cardiomyocyte hypertrophyPositional specification of ventricular myosin light chain 2 expression in the primitive murine heart tubec-Jun N-terminal kinase-interacting protein 1 inhibits gene expression in response to hypertrophic agonists in neonatal rat ventricular myocytesStatins as antioxidant therapy for preventing cardiac myocyte hypertrophyActivation of NF-kappa B is required for hypertrophic growth of primary rat neonatal ventricular cardiomyocytesInhibition of norepinephrine-induced cardiac hypertrophy in s100beta transgenic mice.Essential role of beta-adrenergic receptor kinase 1 in cardiac development and functionAccumulation of muscle ankyrin repeat protein transcript reveals local activation of primary myotube endcompartments during muscle morphogenesisPPARα activation inhibits endothelin-1-induced cardiomyocyte hypertrophy by prevention of NFATc4 binding to GATA-4Developmental expression and cardiac transcriptional regulation of Myh7b, a third myosin heavy chain in the vertebrate heartCardiomyopathy in Irx4-deficient mice is preceded by abnormal ventricular gene expressionGTPase Activating Protein (Sh3 Domain) Binding Protein 1 Regulates the Processing of MicroRNA-1 during Cardiac Hypertrophy.Intermolecular failure of L-type Ca2+ channel and ryanodine receptor signaling in hypertrophy.Deep mRNA sequencing for in vivo functional analysis of cardiac transcriptional regulators: application to Galphaq.Phosphorylation-dependent degradation of p300 by doxorubicin-activated p38 mitogen-activated protein kinase in cardiac cells.microRNAs in heart disease: putative novel therapeutic targets?Opposing effects of Jun kinase and p38 mitogen-activated protein kinases on cardiomyocyte hypertrophyCardiac-specific overexpression of RhoA results in sinus and atrioventricular nodal dysfunction and contractile failure.Mechanical strain activates BNP gene transcription through a p38/NF-kappaB-dependent mechanism.Therapeutic effects of vitamin D analogs on cardiac hypertrophy in spontaneously hypertensive rats.Retinoid-dependent pathways suppress myocardial cell hypertrophy.Lack of ataxia telangiectasia mutated kinase induces structural and functional changes in the heart: role in β-adrenergic receptor-stimulated apoptosisPostmortem cardiac tissue maintains gene expression profile even after late harvesting.Insulin-like growth factor-1 enhances ventricular hypertrophy and function during the onset of experimental cardiac failureDifferential regulation of two types of intracellular calcium release channels during end-stage heart failureDeep RNA sequencing reveals novel cardiac transcriptomic signatures for physiological and pathological hypertrophy17 Beta-estradiol differentially affects left ventricular and cardiomyocyte hypertrophy following myocardial infarction and pressure overloadDisruption of adenylyl cyclase type V does not rescue the phenotype of cardiac-specific overexpression of Galphaq protein-induced cardiomyopathyTrichostatin A accentuates doxorubicin-induced hypertrophy in cardiac myocytes.The cardiac ventricular 5-HT4 receptor is functional in late foetal development and is reactivated in heart failure.Inhibition of a signaling pathway in cardiac muscle cells by active mitogen-activated protein kinase kinaseCardiomyocytes from phorbol myristate acetate-activated mesenchymal stem cells restore electromechanical function in infarcted rat hearts.
P2860
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P2860
Regulation of cardiac gene expression during myocardial growth and hypertrophy: molecular studies of an adaptive physiologic response
description
1991 nî lūn-bûn
@nan
1991 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1991 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
name
Regulation of cardiac gene exp ...... adaptive physiologic response
@ast
Regulation of cardiac gene exp ...... adaptive physiologic response
@en
Regulation of cardiac gene exp ...... adaptive physiologic response
@nl
type
label
Regulation of cardiac gene exp ...... adaptive physiologic response
@ast
Regulation of cardiac gene exp ...... adaptive physiologic response
@en
Regulation of cardiac gene exp ...... adaptive physiologic response
@nl
prefLabel
Regulation of cardiac gene exp ...... adaptive physiologic response
@ast
Regulation of cardiac gene exp ...... adaptive physiologic response
@en
Regulation of cardiac gene exp ...... adaptive physiologic response
@nl
P2093
P3181
P1433
P1476
Regulation of cardiac gene exp ...... adaptive physiologic response
@en
P2093
P304
P3181
P407
P577
1991-12-01T00:00:00Z