Mycobacterium tuberculosis lipoarabinomannan-mediated IRAK-M induction negatively regulates Toll-like receptor-dependent interleukin-12 p40 production in macrophages
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Hemocyanins Stimulate Innate Immunity by Inducing Different Temporal Patterns of Proinflammatory Cytokine Expression in Macrophages.Mycolic acid modification by the mmaA4 gene of M. tuberculosis modulates IL-12 productionGranulocyte-macrophage colony stimulating factor-mediated innate responses in tuberculosisExogenous Nef is an inhibitor of Mycobacterium tuberculosis-induced tumor necrosis factor-alpha production and macrophage apoptosis.Integrated microRNA-mRNA-analysis of human monocyte derived macrophages upon Mycobacterium avium subsp. hominissuis infection.Mycobacterial PIMs inhibit host inflammatory responses through CD14-dependent and CD14-independent mechanismsPathogenic Mycobacterium bovis strains differ in their ability to modulate the proinflammatory activation phenotype of macrophages.Engagement of the mannose receptor by tumoral mucins activates an immune suppressive phenotype in human tumor-associated macrophages.Manipulation of the endocytic pathway and phagocyte functions by Mycobacterium tuberculosis lipoarabinomannan.Novel developments in the epidemic of human immunodeficiency virus and tuberculosis coinfectionSynergistic regulation of Pseudomonas aeruginosa-induced cytokine production in human monocytes by mannose receptor and TLR2.Sepsis-induced suppression of lung innate immunity is mediated by IRAK-M.Innate immune response of alveolar macrophage to house dust mite allergen is mediated through TLR2/-4 co-activationMacrophage immunoregulatory pathways in tuberculosis.Expression of inhibitory regulators of innate immunity in patients with active tuberculosis.Soluble mannosylated myelin peptide inhibits the encephalitogenicity of autoreactive T cells during experimental autoimmune encephalomyelitisLipoarabinomannan and related glycoconjugates: structure, biogenesis and role in Mycobacterium tuberculosis physiology and host-pathogen interactionInterleukin-12 and tuberculosis: an old story revisitedB cell intrinsic TLR signals amplify but are not required for humoral immunity.Pulmonary surfactant protein A and surfactant lipids upregulate IRAK-M, a negative regulator of TLR-mediated inflammation in human macrophages.Mannose-capped lipoarabinomannan from Mycobacterium tuberculosis preferentially inhibits sphingosine-1-phosphate-induced migration of Th1 cells.Intracellular signalling cascades regulating innate immune responses to Mycobacteria: branching out from Toll-like receptors.Endocytosis of Mycobacterium tuberculosis heat shock protein 60 is required to induce interleukin-10 production in macrophages.Mannosylated lipoarabinomannans from Mycobacterium avium subsp. paratuberculosis alters the inflammatory response by bovine macrophages and suppresses killing of Mycobacterium avium subsp. avium organisms.Blimp-1/PRDM1 mediates transcriptional suppression of the NLR gene NLRP12/Monarch-1In situ IL-12/23p40 production during mycobacterial infection is sustained by CD11bhigh dendritic cells localized in tissue sites distinct from those harboring bacilli.Adjusting to a new home: Mycobacterium tuberculosis gene expression in response to an intracellular lifestyle.Mannan core branching of lipo(arabino)mannan is required for mycobacterial virulence in the context of innate immunityIRAK-M regulation and function in host defense and immune homeostasis.The Mycobacterium tuberculosis protein pair PE9 (Rv1088)-PE10 (Rv1089) forms heterodimers and induces macrophage apoptosis through Toll-like receptor 4.Cytokines and Chemokines in Mycobacterium tuberculosis Infection.TLR4 induces tumor growth and inhibits paclitaxel activity in MyD88-positive human ovarian carcinoma in vitro.Wear particles promote endotoxin tolerance in macrophages by inducing interleukin-1 receptor-associated kinase-M expression.Detection of Lipid and Amphiphilic Biomarkers for Disease Diagnostics.Characterization of LRRFIP1.IRAK-M alters the polarity of macrophages to facilitate the survival of Mycobacterium tuberculosis.A novel mouse model of conditional IRAK-M deficiency in myeloid cells: application in lung Pseudomonas aeruginosa infection.Loss of the innate immunity negative regulator IRAK-M leads to enhanced host immune defense against tumor growth.Mycobacteria bypass mucosal NF-kB signalling to induce an epithelial anti-inflammatory IL-22 and IL-10 response.Pulmonary M. tuberculosis infection delays Th1 immunity via immunoadaptor DAP12-regulated IRAK-M and IL-10 expression in antigen-presenting cells.
P2860
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P2860
Mycobacterium tuberculosis lipoarabinomannan-mediated IRAK-M induction negatively regulates Toll-like receptor-dependent interleukin-12 p40 production in macrophages
description
2005 nî lūn-bûn
@nan
2005 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Mycobacterium tuberculosis lip ...... p40 production in macrophages
@ast
Mycobacterium tuberculosis lip ...... p40 production in macrophages
@en
Mycobacterium tuberculosis lip ...... p40 production in macrophages
@nl
type
label
Mycobacterium tuberculosis lip ...... p40 production in macrophages
@ast
Mycobacterium tuberculosis lip ...... p40 production in macrophages
@en
Mycobacterium tuberculosis lip ...... p40 production in macrophages
@nl
prefLabel
Mycobacterium tuberculosis lip ...... p40 production in macrophages
@ast
Mycobacterium tuberculosis lip ...... p40 production in macrophages
@en
Mycobacterium tuberculosis lip ...... p40 production in macrophages
@nl
P2093
P2860
P921
P356
P1476
Mycobacterium tuberculosis lip ...... p40 production in macrophages
@en
P2093
Asima Bhattacharyya
Joyoti Basu
Manikuntala Kundu
Sanchita Basu
Shresh Pathak
Sushil Kumar Pathak
P2860
P304
P356
10.1074/JBC.M506471200
P407
P577
2005-12-30T00:00:00Z