SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
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SOD1 Transcriptional and Posttranscriptional Regulation and Its Potential Implications in ALSFlavin-containing monooxygenase mRNA levels are up-regulated in als brain areas in SOD1-mutant miceRegulation of CuZnSOD and its redox signaling potential: implications for amyotrophic lateral sclerosisCis-acting single nucleotide polymorphisms alter MicroRNA-mediated regulation of human brain-expressed transcriptsMicroarray analysis of peripheral blood lymphocytes from ALS patients and the SAFE detection of the KEGG ALS pathway.Proteasome activation is a mechanism for pyrazolone small molecules displaying therapeutic potential in amyotrophic lateral sclerosisAltered intracellular localization of SOD1 in leukocytes from patients with sporadic amyotrophic lateral sclerosis.Genotype-property patient-phenotype relations suggest that proteome exhaustion can cause amyotrophic lateral sclerosis.The anabolic/androgenic steroid nandrolone exacerbates gene expression modifications induced by mutant SOD1 in muscles of mice models of amyotrophic lateral sclerosis.From Transcriptome to Noncoding RNAs: Implications in ALS Mechanism.An indicator cell assay for blood-based diagnostics.Epigallocatechin-3-gallate Protects against Hydrogen Peroxide-Induced Inhibition of Osteogenic Differentiation of Human Bone Marrow-Derived Mesenchymal Stem CellsTorsinA rescues ER-associated stress and locomotive defects in C. elegans models of ALS.Regulation of FMO and PON detoxication systems in ALS human tissues.Effect of Oxidative Damage on the Stability and Dimerization of Superoxide Dismutase 1.H63D HFE polymorphisms are associated with increased disease duration and decreased muscle superoxide dismutase-1 expression in amyotrophic lateral sclerosis patients.Detecting intracellular cysteine redox states by in-cell NMR spectroscopy.Superoxide dismutase 1 is positively selected to minimize protein aggregation in great apes.Study of the anti-inflammatory effects of low-dose radiation: The contribution of biphasic regulation of the antioxidative system in endothelial cells.Long non-coding and coding RNAs characterization in Peripheral Blood Mononuclear Cells and Spinal Cord from Amyotrophic Lateral Sclerosis patients.Lymphoblastoid cell lines as a model to understand amyotrophic lateral sclerosis disease mechanisms.SOD1 in Amyotrophic Lateral Sclerosis: "Ambivalent" Behavior Connected to the Disease.Theme 10 Molecular Cell BiologyTHEME 8 HUMAN CELL BIOLOGY AND PATHOLOGYSOD1 stimulates lamellipodial protrusions in Neuro 2A cell lines
P2860
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P2860
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
description
2010 nî lūn-bûn
@nan
2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
@ast
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
@en
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
@nl
type
label
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
@ast
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
@en
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
@nl
prefLabel
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
@ast
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
@en
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
@nl
P2093
P50
P1476
SOD1 mRNA expression in sporadic amyotrophic lateral sclerosis
@en
P2093
Annalisa Davin
Elena Alvisi
Emanuela Cova
John Richard Cashman
Kenneth Abel
Stefania Guareschi
Stella Gagliardi
P304
P356
10.1016/J.NBD.2010.04.008
P407
P577
2010-08-01T00:00:00Z