Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210 - Wikidata - awgldk - TriplyDB

Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

http://www.wikidata.org/entity/Q28291120

about

Flexible and versatile as a chameleon-sophisticated functions of microRNA-199a MicroRNAs as Biomarkers for Liver Disease and Hepatocellular Carcinoma Autophagy and microRNA in hepatitis B virus-related hepatocellular carcinoma MicroRNAs Expressed during Viral Infection: Biomarker Potential and Therapeutic Considerations MicroRNAs as Important Players in Host-hepatitis B Virus Interactions Hepatitis B Virus Infection, MicroRNAs and Liver Disease MicroRNAs may solve the mystery of chronic hepatitis B virus infection MicroRNAs, hepatitis C virus, and HCV/HIV-1 co-infection: new insights in pathogenesis and therapy Hepatitis B virus and microRNAs: Complex interactions affecting hepatitis B virus replication and hepatitis B virus-associated diseases Host cellular microRNA involvement in the control of hepatitis B virus gene expression and replication Roles of hepatocyte nuclear factors in hepatitis B virus infection Mammalian microRNA: an important modulator of host-pathogen interactions in human viral infections Genome-wide identification of direct HBx genomic targets Connecting rules from paired miRNA and mRNA expression data sets of HCV patients to detect both inverse and positive regulatory relationships.miR-106b-25/miR-17-92 clusters: polycistrons with oncogenic roles in hepatocellular carcinoma MicroRNAs as therapeutic strategy for hepatitis B virus-associated hepatocellular carcinoma: current status and future prospects Modulation of HBV replication by microRNA-15b through targeting hepatocyte nuclear factor 1α A re-examination of global suppression of RNA interference by HIV-1.Transcriptional suppression of miR-181c by hepatitis C virus enhances homeobox A1 expression.Ago-2-mediated slicer activity is essential for anti-flaviviral efficacy of RNAi.Control of hepatitis B virus replication by interferons and Toll-like receptor signaling pathways MiR-122 directly inhibits human papillomavirus E6 gene and enhances interferon signaling through blocking suppressor of cytokine signaling 1 in SiHa cells MicroRNAs associated with HBV infection and HBV-related HCC.Complex interactions between microRNAs and hepatitis B/C viruses.TGF-β and iron differently alter HBV replication in human hepatocytes through TGF-β/BMP signaling and cellular microRNA expression.Prospects for inhibiting the post-transcriptional regulation of gene expression in hepatitis B virus.Hepatitis B virus-specific miRNAs and Argonaute2 play a role in the viral life cycle.MiR-125b reduces porcine reproductive and respiratory syndrome virus replication by negatively regulating the NF-κB pathway Cellular microRNAs 498 and 320d regulate herpes simplex virus 1 induction of Kaposi's sarcoma-associated herpesvirus lytic replication by targeting RTA.Preparation of Small RNAs Using Rolling Circle Transcription and Site-Specific RNA Disconnection Liver microRNA hsa-miR-125a-5p in HBV chronic infection: correlation with HBV replication and disease progression MicroRNAs in Human Diseases: From Cancer to Cardiovascular Disease.Ectopic expression of microRNA-155 enhances innate antiviral immunity against HBV infection in human hepatoma cells MicroRNAs and human retroviruses Possible role of tocopherols in the modulation of host microRNA with potential antiviral activity in patients with hepatitis B virus-related persistent infection: a systematic review.MicroRNAs in Human Diseases: From Autoimmune Diseases to Skin, Psychiatric and Neurodegenerative Diseases Cellular microRNA miR-26a suppresses replication of porcine reproductive and respiratory syndrome virus by activating innate antiviral immunity.MicroRNAs in Human Diseases: From Lung, Liver and Kidney Diseases to Infectious Disease, Sickle Cell Disease and Endometrium Disease.Genomic Methylation Inhibits Expression of Hepatitis B Virus Envelope Protein in Transgenic Mice: A Non-Infectious Mouse Model to Study Silencing of HBV Surface Antigen Genes The expression of miR-125b-5p is increased in the serum of patients with chronic hepatitis B infection and inhibits the detection of hepatitis B virus surface antigen.

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Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

http://www.wikidata.org/entity/Q28291120

description

2010 nî lūn-bûn

@nan

2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

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name

Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

@ast

Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

@en

Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

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type

label

Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

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Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

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Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

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prefLabel

Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

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Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

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Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

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J.ANTIVIRAL.2010.08.008

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Antiviral Research

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Suppression of hepatitis B virus replication by microRNA-199a-3p and microRNA-210

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P2093

Guang-ling Zhang

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Hua Tang

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Min Liu

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Shu-qi Zheng

http://www.w3.org/2001/XMLSchema#string

Xin Li

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Yi-xuan Li

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169-75

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scholarly article

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2061333

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10.1016/J.ANTIVIRAL.2010.08.008

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2

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2010-11-01T00:00:00Z

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20728471

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