Troponin I degradation and covalent complex formation accompanies myocardial ischemia/reperfusion injury
about
Gene regulation, alternative splicing, and posttranslational modification of troponin subunits in cardiac development and adaptation: a focused reviewMutations in genes encoding fast-twitch contractile proteins cause distal arthrogryposis syndromesMuscle-specific RING finger 1 is a bona fide ubiquitin ligase that degrades cardiac troponin IRaised cardiac troponinsA proteolytic NH2-terminal truncation of cardiac troponin I that is up-regulated in simulated microgravityStructural based insights into the role of troponin in cardiac muscle pathophysiologyTop-down quantitative proteomics identified phosphorylation of cardiac troponin I as a candidate biomarker for chronic heart failureCalcium-regulated conformational change in the C-terminal end segment of troponin I and its binding to tropomyosinThe C terminus of cardiac troponin I stabilizes the Ca2+-activated state of tropomyosin on actin filamentsWhy does troponin I have so many phosphorylation sites? Fact and fancyAnalysis of myotilin turnover provides mechanistic insight into the role of myotilinopathy-causing mutationsIschemic dysfunction in transgenic mice expressing troponin I lacking protein kinase C phosphorylation sitesMaintained contractile reserve in a transgenic mouse model of myocardial stunningMultiple Reaction Monitoring to Identify Site-Specific Troponin I Phosphorylated Residues in the Failing Human HeartTNNI1, TNNI2 and TNNI3: Evolution, regulation, and protein structure-function relationships.Alterations to myofibrillar protein function in nonischemic regions of the heart early after myocardial infarction.Diastolic dysfunction and heart failure with a preserved ejection fraction: Relevance in critical illness and anaesthesiaDo induced tachycardias within the scope of electrophysiological studies lead to elevated plasma troponin I levels?Proteomic changes in hearts of kyphoscoliosis (ky) mutant mice in the absence of structural pathology: implication for the analysis of early human heart disease.SELDI-TOF MS analysis of the Cardiac Troponin I forms present in plasma from patients with myocardial infarction.Combined troponin I Ser-150 and Ser-23/24 phosphorylation sustains thin filament Ca(2+) sensitivity and accelerates deactivation in an acidic environment.Dietary red palm oil supplementation reduces myocardial infarct size in an isolated perfused rat heart model.Genome informatics: current status and future prospects.Disruption of Caenorhabditis elegans muscle structure and function caused by mutation of troponin IRecent insights into muscle fatigue at the cross-bridge level.Myofilament calcium sensitization delays decompensated hypertrophy differently between the sexes following myocardial infarctionAn inhibitor of the δPKC interaction with the d subunit of F1Fo ATP synthase reduces cardiac troponin I release from ischemic rat hearts: utility of a novel ammonium sulfate precipitation techniquePatients ≥ 75 years with acute coronary syndrome but without critical epicardial coronary disease: prevalence, characteristics, and outcome.Dietary red palm oil supplementation decreases infarct size in cholesterol fed ratsTear me down: role of calpain in the development of cardiac ventricular hypertrophy.Serum troponins as biomarkers of drug-induced cardiac toxicity.Modifications of myofilament protein phosphorylation and function in response to cardiac arrest induced in a swine modelGrowth hormone secretagogues protect mouse cardiomyocytes from in vitro ischemia/reperfusion injury through regulation of intracellular calcium.Ablation of junctin or triadin is associated with increased cardiac injury following ischaemia/reperfusionIn vivo phosphorylation site mapping in mouse cardiac troponin I by high resolution top-down electron capture dissociation mass spectrometry: Ser22/23 are the only sites basally phosphorylated.Troponin as a marker of myocardiac damage in drug-induced cardiotoxicity.Myocardial regulatory proteins and heart failure.Functional consequences of sarcomeric protein abnormalities in failing myocardium.Heart failure, myocardial stunning, and troponin: a key regulator of the cardiac myofilament.Overview: the maturing of proteomics in cardiovascular research.
P2860
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P2860
Troponin I degradation and covalent complex formation accompanies myocardial ischemia/reperfusion injury
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Troponin I degradation and cov ...... al ischemia/reperfusion injury
@ast
Troponin I degradation and cov ...... al ischemia/reperfusion injury
@en
Troponin I degradation and cov ...... al ischemia/reperfusion injury
@nl
type
label
Troponin I degradation and cov ...... al ischemia/reperfusion injury
@ast
Troponin I degradation and cov ...... al ischemia/reperfusion injury
@en
Troponin I degradation and cov ...... al ischemia/reperfusion injury
@nl
prefLabel
Troponin I degradation and cov ...... al ischemia/reperfusion injury
@ast
Troponin I degradation and cov ...... al ischemia/reperfusion injury
@en
Troponin I degradation and cov ...... al ischemia/reperfusion injury
@nl
P2093
P356
P1433
P1476
Troponin I degradation and cov ...... al ischemia/reperfusion injury
@en
P2093
D K Arrell
J E Van Eyk
J L McDonough
P356
10.1161/01.RES.84.1.9
P407
P577
1999-01-01T00:00:00Z