Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
about
Mlx, a new Max-like bHLHZip family member: the center stage of a novel transcription factors regulatory pathway?Mad3 and Mad4: novel Max-interacting transcriptional repressors that suppress c-myc dependent transformation and are expressed during neural and epidermal differentiationStructure of pp32, an acidic nuclear protein which inhibits oncogene-induced formation of transformed fociMouse Brn-3 family of POU transcription factors: a new aminoterminal domain is crucial for the oncogenic activity of Brn-3aDual mechanisms of repression of E2F1 activity by the retinoblastoma gene productAntiproliferative properties of the USF family of helix-loop-helix transcription factorsNew Myc-interacting proteins: a second Myc network emerges.Mechanisms of apoptosis by c-Myc.The Max network gone mad.Hypoxia-induced alternative splicing in endothelial cells.Cloning of mid-G1 serum response genes and identification of a subset regulated by conditional myc expressionA genetic variant in primary miR-378 is associated with risk and prognosis of hepatocellular carcinoma in a Chinese population.Suppression of Myc, but not E1a, transformation activity by Max-associated proteins, Mad and Mxi1A dominant-negative mutant of Max that inhibits sequence-specific DNA binding by Myc proteins.Opposite regulation of gene transcription and cell proliferation by c-Myc and Max.Transactivation of gene expression by Myc is inhibited by mutation at the phosphorylation sites Thr-58 and Ser-62.Expression of the mad gene during cell differentiation in vivo and its inhibition of cell growth in vitroBiophysical characterization of the b-HLH-LZ of ΔMax, an alternatively spliced isoform of Max found in tumor cells: Towards the validation of a tumor suppressor role for the Max homodimers.Variant Max protein, derived by alternative splicing, associates with c-Myc in vivo and inhibits transactivationA minimal regulatory region maintains constitutive expression of the max gene.Identification of a thymidylate synthase ribonucleoprotein complex in human colon cancer cellsRepression of cyclin D1: a novel function of MYCZebra fish myc family and max genes: differential expression and oncogenic activity throughout vertebrate evolution.Cell cycle regulation of the c-Myc transcriptional activation domain.A bipartite nuclear localization signal in the retinoblastoma gene product and its importance for biological activityRetroviral insertions downstream of the heterogeneous nuclear ribonucleoprotein A1 gene in erythroleukemia cells: evidence that A1 is not essential for cell growth.EGFR mutation-induced alternative splicing of Max contributes to growth of glycolytic tumors in brain cancer.A high throughput embryonic stem cell screen identifies Oct-2 as a bifunctional regulator of neuronal differentiation.An overview of MYC and its interactome.Sin3 corepressor function in Myc-induced transcription and transformation.Functional Myc-Max heterodimer is required for activation-induced apoptosis in T cell hybridomas.Distinct DNA binding preferences for the c-Myc/Max and Max/Max dimers.Ras induces p21Cip1/Waf1 cyclin kinase inhibitor transcriptionally through Sp1-binding sites.Overproduction of v-Myc in the nucleus and its excess over Max are not required for avian fibroblast transformation.Sequence-specific transcriptional activation by Myc and repression by Max.Reconstitution of an E box-binding Myc:Max complex with recombinant full-length proteins expressed in Escherichia coli.L-myc and N-myc in hematopoietic malignancies.c-Myc induces cellular susceptibility to the cytotoxic action of TNF-alpha.Transcription activation by Myc and Max: flanking sequences target activation to a subset of CACGTG motifs in vivo.The c-Myc protein induces cell cycle progression and apoptosis through dimerization with Max.
P2860
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P2860
Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
description
1992 nî lūn-bûn
@nan
1992 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
@ast
Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
@en
Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
@nl
type
label
Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
@ast
Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
@en
Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
@nl
prefLabel
Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
@ast
Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
@en
Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
@nl
P2093
P1433
P1476
Alternative forms of Max as enhancers or suppressors of Myc-ras cotransformation
@en
P2093
P J Koskinen
T P Mäkelä
P356
10.1126/SCIENCE.256.5055.373
P407
P50
P577
1992-04-17T00:00:00Z