HMG CoA reductase inhibitors affect the fibrinolytic system of human vascular cells in vitro: a comparative study using different statins - Wikidata - awgldk - TriplyDB

HMG CoA reductase inhibitors affect the fibrinolytic system of human vascular cells in vitro: a comparative study using different statins

HMG CoA reductase inhibitors affect the fibrinolytic system of human vascular cells in vitro: a comparative study using different statins

http://www.wikidata.org/entity/Q28344912

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Comparative gene expression profiling in three primary human cell lines after treatment with a novel inhibitor of Rho kinase or atorvastatin Do the pleiotropic effects of statins in the vasculature predict a role in inflammatory diseases?Simvastatin re-couples dysfunctional endothelial nitric oxide synthase in experimental subarachnoid hemorrhage Statin therapy is not associated with improved vascular access outcomes.Plasminogen activator inhibitor-1: physiologic role, regulation, and the influence of common pharmacologic agents.Statins as first-line therapy for acute coronary syndrome?Statins in the first-line therapy of acute coronary syndrome - similar to aspirin?Effect of orally administered simvastatin on prevention of postoperative adhesion in rats.Statins in sepsis.Topical simvastatin enhances tissue regeneration in full-thickness skin wounds in rat models Risk of arteriovenous fistula failure associated with hypnotic use in hemodialysis patients: a nested case-control study.Atorvastatin affects several angiogenic mediators in human endothelial cells.Human cardiac fibroblasts express B-type natriuretic peptide: fluvastatin ameliorates its up-regulation by interleukin-1alpha, tumour necrosis factor-alpha and transforming growth factor-beta.Protective effects of statins involving both eNOS and tPA in focal cerebral ischemia Statins suppress glucose-induced plasminogen activator inhibitor-1 expression by regulating RhoA and nuclear factor-κB activities in cardiac microvascular endothelial cells.Concomitant administration of different doses of simvastatin with ivabradine influence on PAI-1 and heart rate in normo- and hypercholesterolaemic rats.Effect of statins on experimental postoperative adhesion: a systematic review and meta-analysis The inflammatory cytokine oncostatin M induces PAI-1 in human vascular smooth muscle cells in vitro via PI 3-kinase and ERK1/2-dependent pathways

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P2860

HMG CoA reductase inhibitors affect the fibrinolytic system of human vascular cells in vitro: a comparative study using different statins

http://www.wikidata.org/entity/Q28344912

description

2002 nî lūn-bûn

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2002 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2002 թվականի հունվարին հրատարակված գիտական հոդված

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2002年の論文

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British Journal of Pharmacology

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P2093

Christoph Kaun

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Franz Wiesbauer

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Gerald Maurer

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Gerlinde Zorn

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Kurt Huber

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P2860

Effects of the 3-hydroxy-3-methylglutaryl-CoA reductase inhibitors, atorvastatin and simvastatin, on the expression of endothelin-1 and endothelial nitric oxide synthase in vascular endothelial cells

Randomised trial of cholesterol lowering in 4444 patients with coronary heart disease: the Scandinavian Simvastatin Survival Study (4S)

The effect of pravastatin on coronary events after myocardial infarction in patients with average cholesterol levels. Cholesterol and Recurrent Events Trial investigators

Prevention of coronary heart disease with pravastatin in men with hypercholesterolemia. West of Scotland Coronary Prevention Study Group

Differences in the effects of HMG-CoA reductase inhibitors on proliferation and viability of smooth muscle cells in culture.

The evolving role of statins in the management of atherosclerosis.

Comparison of one-year efficacy and safety of atorvastatin versus lovastatin in primary hypercholesterolemia. Atorvastatin Study Group I.

Atorvastatin: an effective lipid-modifying agent in familial hypercholesterolemia.

Increased type 1 plasminogen activator inhibitor gene expression in atherosclerotic human arteries

Role of plasminogen activator inhibitor type 1 in the pathogenesis of coronary artery diseases.

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10.1038/SJ.BJP.0704454

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