Exercise-induced hyperaemia and leg oxygen uptake are not altered during effective inhibition of nitric oxide synthase with N(G)-nitro-L-arginine methyl ester in humans - Wikidata - awgldk - TriplyDB

Exercise-induced hyperaemia and leg oxygen uptake are not altered during effective inhibition of nitric oxide synthase with N(G)-nitro-L-arginine methyl ester in humans

Exercise-induced hyperaemia and leg oxygen uptake are not altered during effective inhibition of nitric oxide synthase with N(G)-nitro-L-arginine methyl ester in humans

http://www.wikidata.org/entity/Q28349449

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Hypoxic Vasodilation by Red Blood Cells: Evidence for an S-Nitrosothiol-Based Signal Contribution of non-endothelium-dependent substances to exercise hyperaemia: are they O(2) dependent?Skeletal muscle nitric oxide signaling and exercise: a focus on glucose metabolism Regulation of increased blood flow (hyperemia) to muscles during exercise: a hierarchy of competing physiological needs Red cell physiology and signaling relevant to the critical care setting Exogenous NO administration and alpha-adrenergic vasoconstriction in human limbs.Nitric oxide synthase inhibition during treadmill exercise reveals fiber-type specific vascular control in the rat hindlimb.Nitric oxide, but not vasodilating prostaglandins, contributes to the improvement of exercise hyperemia via ascorbic acid in healthy older adults.Progressive handgrip exercise: evidence of nitric oxide-dependent vasodilation and blood flow regulation in humans.Perfused skeletal muscle -- an experimental preparation for many questions!Effects of chronic nitric oxide synthase inhibition on responses to acute exercise in swine Effect of exercise training on endothelium-derived nitric oxide function in humans.Aging is associated with altered vasodilator kinetics in dynamically contracting muscle: role of nitric oxide.Effect of processing and storage on red blood cell function in vivo 'Fine-tuning' blood flow to the exercising muscle with advancing age: an update.Exercise hyperaemia: is anything obligatory but the hyperaemia?Effect of nitric oxide synthase inhibition on the exchange of glucose and fatty acids in human skeletal muscle.Nitric oxide and muscle blood flow in exercise.Roles of nitric oxide synthase and cyclooxygenase in leg vasodilation and oxygen consumption during prolonged low-intensity exercise in untrained humans.Tetrahydrobiopterin ameliorates the exaggerated exercise pressor response in patients with chronic kidney disease: a randomized controlled trial Exercise training reverses age-related decrements in endothelium-dependent dilation in skeletal muscle feed arteries NOS inhibition blunts and delays the compensatory dilation in hypoperfused contracting human muscles.Oxygen uptake kinetics: historical perspective and future directions.Vasodilator interactions in skeletal muscle blood flow regulation.Arginine-based inhibitors of nitric oxide synthase: therapeutic potential and challenges.Nitric oxide transport in blood: a third gas in the respiratory cycle.Regulation of the skeletal muscle blood flow in humans.Regulation of skeletal muscle blood flow during exercise in ageing humans.Cardiovascular Adaptations to Exercise Training.Effects of combined inhibition of ATP-sensitive potassium channels, nitric oxide, and prostaglandins on hyperemia during moderate exercise.Acute tetrahydrobiopterin supplementation attenuates sympathetic vasoconstrictor responsiveness in resting and contracting skeletal muscle of healthy rats.Skeletal muscle contraction-induced vasodilation in the microcirculation.Low blood flow at onset of moderate-intensity exercise does not limit muscle oxygen uptake.Involvement of nitric oxide synthase in skeletal muscle adaptation to chronic overload.Cytochrome P450 2C9 plays an important role in the regulation of exercise-induced skeletal muscle blood flow and oxygen uptake in humans.Inhibition of nitric oxide synthase by L-NAME speeds phase II pulmonary .VO2 kinetics in the transition to moderate-intensity exercise in man.Blunted sympathetic vasoconstriction in contracting skeletal muscle of healthy humans: is nitric oxide obligatory?Local inhibition of nitric oxide and prostaglandins independently reduces forearm exercise hyperaemia in humans.Combined NO and PG inhibition augments alpha-adrenergic vasoconstriction in contracting human skeletal muscle.Impact of combined NO and PG blockade on rapid vasodilation in a forearm mild-to-moderate exercise transition in humans.

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P2860

Exercise-induced hyperaemia and leg oxygen uptake are not altered during effective inhibition of nitric oxide synthase with N(G)-nitro-L-arginine methyl ester in humans

http://www.wikidata.org/entity/Q28349449

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2001 nî lūn-bûn

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2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2001 թվականի փետրվարին հրատարակված գիտական հոդված

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2001年の論文

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J.1469-7793.2001.0257J.X

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The Journal of Physiology

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Exercise-induced hyperaemia an ...... rginine methyl ester in humans

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P2860

Isolation of nitric oxide synthetase, a calmodulin-requiring enzyme

Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor

Inhibition of nitric oxide synthesis by NG-nitro-L-arginine methyl ester (L-NAME): requirement for bioactivation to the free acid, NG-nitro-L-arginine.

Nitric oxide production and NO synthase gene expression contribute to vascular regulation during exercise.

Nitric oxide regulates mitochondrial respiration and cell functions by inhibiting cytochrome oxidase.

Adenine nucleotide degradation in slow-twitch red muscle.

Comparison of the inhibitory potencies of N(G)-methyl-, N(G)-nitro- and N(G)-amino-L-arginine on EDRF function in the rat: evidence for continuous basal EDRF release.

Nitric oxide release is present from incubated skeletal muscle preparations.

Contribution of endothelium-derived nitric oxide to exercise-induced vasodilation.

Canine hindlimb blood flow and O2 uptake after inhibition of EDRF/NO synthesis.

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257-64

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10.1111/J.1469-7793.2001.0257J.X

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Pt 1

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531

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2001-02-15T00:00:00Z

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11179408

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