CLEC5A-Mediated Enhancement of the Inflammatory Response in Myeloid Cells Contributes to Influenza Virus Pathogenicity In Vivo - Wikidata - awgldk - TriplyDB

CLEC5A-Mediated Enhancement of the Inflammatory Response in Myeloid Cells Contributes to Influenza Virus Pathogenicity In Vivo

CLEC5A-Mediated Enhancement of the Inflammatory Response in Myeloid Cells Contributes to Influenza Virus Pathogenicity In Vivo

http://www.wikidata.org/entity/Q28354040

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Transcriptome analysis of bronchoalveolar lavage fluid from children with severe Mycoplasma pneumoniae pneumonia reveals novel gene expression and immunodeficiency Myeloid C-Type Lectin Receptors in Viral Recognition and Antiviral Immunity.Enteric Virome Sensing-Its Role in Intestinal Homeostasis and Immunity.C-Type Lectin Receptors in Antiviral Immunity and Viral Escape.

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P2860

CLEC5A-Mediated Enhancement of the Inflammatory Response in Myeloid Cells Contributes to Influenza Virus Pathogenicity In Vivo

http://www.wikidata.org/entity/Q28354040

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2017 nî lūn-bûn

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2017 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2017年の論文

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Jian Teddy Zheng

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Ooiean Teng

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Sin Fun Sia

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Suzanne Cole

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Szu-Ting Chen

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Tsui-Ling Hsu

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Tzu-Yun Hsu

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Wenwei Tu

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P2860

Myeloid DAP12-associating lectin (MDL)-1 is a cell surface receptor involved in the activation of myeloid cells

CLEC5A is critical for dengue-virus-induced lethal disease

IFITM3 restricts the morbidity and mortality associated with influenza

Recognition of single-stranded RNA viruses by Toll-like receptor 7

RIG-I-mediated antiviral responses to single-stranded RNA bearing 5'-phosphates

The role of viral, host, and secondary bacterial factors in influenza pathogenesis

Induction of proinflammatory cytokines in human macrophages by influenza A (H5N1) viruses: a mechanism for the unusual severity of human disease?

The receptor TREML4 amplifies TLR7-mediated signaling during antiviral responses and autoimmunity

Fatal outcome of human influenza A (H5N1) is associated with high viral load and hypercytokinemia

Influenza virus activates inflammasomes via its intracellular M2 ion channel

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10.1128/JVI.01813-16

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Sophie A Valkenburg

Roberto Bruzzone

Shie-Liang Hsieh

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