Podocytic cytoskeletal disaggregation and basement-membrane detachment in puromycin aminonucleoside nephrosis
about
Loss of glomerular foot processes is associated with uncoupling of podocalyxin from the actin cytoskeletonPodocytesProtective effects of the mTOR inhibitor everolimus on cytoskeletal injury in human podocytes are mediated by RhoA signalingGlomerular permselectivity factors are not responsible for the increase in fractional clearance of albumin in rat glomerulonephritisPodoplanin, novel 43-kd membrane protein of glomerular epithelial cells, is down-regulated in puromycin nephrosisProteinuria precedes podocyte abnormalities inLamb2-/- mice, implicating the glomerular basement membrane as an albumin barrierPhosphorylation of podocalyxin (Ser415) Prevents RhoA and ezrin activation and disrupts its interaction with the actin cytoskeleton.Alterations in fatty acid utilization and an impaired antioxidant defense mechanism are early events in podocyte injury: a proteomic analysis.Extrarenal effects on the pathogenesis and relapse of idiopathic nephrotic syndrome in Buffalo/Mna ratsPathophysiologic implications of reduced podocyte number in a rat model of progressive glomerular injury.A vital role for Angptl3 in the PAN-induced podocyte loss by affecting detachment and apoptosis in vitro.Everolimus Stabilizes Podocyte Microtubules via Enhancing TUBB2B and DCDC2 Expression.The glomerular basement membrane as a model system to study the bioactivity of heparan sulfate glycosaminoglycans.A Podocyte-Based Automated Screening Assay Identifies Protective Small Molecules.Altered expression of glomerular heat shock protein 27 in experimental nephrotic syndrome.Podocytes … What's Under Yours? (Podocytes and Foot Processes and How They Change in Nephropathy).Permselective dysfunction of podocyte-podocyte contact upon angiotensin II unravels the molecular target for renoprotective intervention.Permeability factors in nephrotic syndrome and focal segmental glomerulosclerosis.A High-Content Screening Technology for Quantitatively Studying Podocyte Dynamics.Cytoskeletal changes in podocytes associated with foot process effacement in Masugi nephritis.LncRNA MALAT1 is dysregulated in diabetic nephropathy and involved in high glucose-induced podocyte injury via its interplay with β-catenin.
P2860
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P2860
Podocytic cytoskeletal disaggregation and basement-membrane detachment in puromycin aminonucleoside nephrosis
description
1993 nî lūn-bûn
@nan
1993 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1993 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
name
Podocytic cytoskeletal disaggr ...... ycin aminonucleoside nephrosis
@ast
Podocytic cytoskeletal disaggr ...... ycin aminonucleoside nephrosis
@en
Podocytic cytoskeletal disaggr ...... ycin aminonucleoside nephrosis
@nl
type
label
Podocytic cytoskeletal disaggr ...... ycin aminonucleoside nephrosis
@ast
Podocytic cytoskeletal disaggr ...... ycin aminonucleoside nephrosis
@en
Podocytic cytoskeletal disaggr ...... ycin aminonucleoside nephrosis
@nl
prefLabel
Podocytic cytoskeletal disaggr ...... ycin aminonucleoside nephrosis
@ast
Podocytic cytoskeletal disaggr ...... ycin aminonucleoside nephrosis
@en
Podocytic cytoskeletal disaggr ...... ycin aminonucleoside nephrosis
@nl
P2093
P2860
P1476
Podocytic cytoskeletal disaggr ...... ycin aminonucleoside nephrosis
@en
P2093
P2860
P304
P407
P577
1993-05-01T00:00:00Z