Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers - Wikidata - awgldk - TriplyDB

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

http://www.wikidata.org/entity/Q28382770

about

Prenatal exposure to arsenic and cadmium impacts infectious disease-related genes within the glucocorticoid receptor signal transduction pathway Chronic occupational exposure to arsenic induces carcinogenic gene signaling networks and neoplastic transformation in human lung epithelial cells Toxicogenomic profiling of chemically exposed humans in risk assessment Epidemiologic evidence for association between adverse environmental exposures in early life and epigenetic variation: a potential link to disease susceptibility?Arsenic and human health: epidemiologic progress and public health implications Identifying important life stages for monitoring and assessing risks from exposures to environmental contaminants: results of a World Health Organization review Early-life Exposure to Endocrine Disrupting Chemicals and Later-life Health Outcomes: An Epigenetic Bridge?Predicting later-life outcomes of early-life exposures Environmental chemicals and type 2 diabetes: an updated systematic review of the epidemiologic evidence Gut microbiome perturbations induced by bacterial infection affect arsenic biotransformation Early-life arsenic exposure: methylation capacity and beyond Mechanisms of divalent metal toxicity in affective disorders Effects of low-dose drinking water arsenic on mouse fetal and postnatal growth and development Role of reactive oxygen species in arsenic-induced transformation of human lung bronchial epithelial (BEAS-2B) cells Prenatal Arsenic Exposure and Birth Outcomes among a Population Residing near a Mining-Related Superfund Site In utero and early childhood exposure to arsenic decreases lung function in children In utero arsenic exposure and fetal immune repertoire in a US pregnancy cohort Arsenic and cardiovascular disease Progress and prospects of reactive oxygen species in metal carcinogenesis In utero and early life arsenic exposure in relation to long-term health and disease Arsenic Toxicity in Male Reproduction and Development Perturbation of defense pathways by low-dose arsenic exposure in zebrafish embryos In utero arsenic exposure and infant infection in a United States cohort: a prospective study Incorporating epigenetic data into the risk assessment process for the toxic metals arsenic, cadmium, chromium, lead, and mercury: strategies and challenges Prenatal environmental exposures, epigenetics, and disease Low level exposure to monomethyl arsonous acid-induced the over-production of inflammation-related cytokines and the activation of cell signals associated with tumor progression in a urothelial cell model Interleukin-8 (IL-8) over-production and autocrine cell activation are key factors in monomethylarsonous acid [MMA(III)]-induced malignant transformation of urothelial cells Early life environment and developmental immunotoxicity in inflammatory dysfunction and disease Disruption of Skin Stem Cell Homeostasis following Transplacental Arsenicosis; Alleviation by Combined Intake of Selenium and Curcumin Prenatal arsenic exposure alters gene expression in the adult liver to a proinflammatory state contributing to accelerated atherosclerosis Towards prenatal biomonitoring in North Carolina: assessing arsenic, cadmium, mercury, and lead levels in pregnant women Maternal influences on the transmission of leukocyte gene expression profiles in population samples from Brisbane, Australia The Importance of the Biological Impact of Exposure to the Concept of the Exposome The Comparative Toxicogenomics Database facilitates identification and understanding of chemical-gene-disease associations: arsenic as a case study.Prenatal arsenic exposure and the epigenome: altered microRNAs associated with innate and adaptive immune signaling in newborn cord blood.Prenatal arsenic exposure and shifts in the newborn proteome: interindividual differences in tumor necrosis factor (TNF)-responsive signaling The NRF2-mediated oxidative stress response pathway is associated with tumor cell resistance to arsenic trioxide across the NCI-60 panel.Long-term health consequences of prenatal arsenic exposure: links to the genome and the epigenome.Neonatal Metabolomic Profiles Related to Prenatal Arsenic Exposure Cord blood T cell subpopulations and associations with maternal cadmium and arsenic exposures.

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P2860

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

http://www.wikidata.org/entity/Q28382770

description

2007 nî lūn-bûn

@nan

2007 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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name

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

@ast

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

@en

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

@nl

type

label

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

@ast

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

@en

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

@nl

prefLabel

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

@ast

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

@en

Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

@nl

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JOURNAL.PGEN.0030207

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Activation of inflammation/NF-kappaB signaling in infants born to arsenic-exposed mothers

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Bradley J Hogan

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Chandni Valiathan

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Chulabhorn Mahidol

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Krittinee Kandjanapa

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Leona D Samson

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Manlin Luo

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Mathuros Ruchirawat

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Panida Navasumrit

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Sanchita Bhattacharya

http://www.w3.org/2001/XMLSchema#string

Sumitra Soontararuks

http://www.w3.org/2001/XMLSchema#string

P2860

Arsenic can mediate skin neoplasia by chronic stimulation of keratinocyte-derived growth factors.

Arsenic induces overexpression of growth factors in human keratinocytes

Isolation and properties of cDNA clones encoding SRF, a transcription factor that binds to the c-fos serum response element

Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profiles.

Integrating phenotypic and expression profiles to map arsenic-response networks

GoMiner: a resource for biological interpretation of genomic and proteomic data

EXPANDER--an integrative program suite for microarray data analysis

Gene expression of inflammatory molecules in circulating lymphocytes from arsenic-exposed human subjects.

Arsenic enhancement of skin neoplasia by chronic stimulation of growth factors

Summaries of Affymetrix GeneChip probe level data

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e207

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4797723

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10.1371/JOURNAL.PGEN.0030207

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11

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3

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J Peter Svensson

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2007-11-01T00:00:00Z

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5808862

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18039032

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maternal health

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2082467

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