Cardiolipin remodeling by TAZ/tafazzin is selectively required for the initiation of mitophagy - Wikidata - awgldk - TriplyDB

Cardiolipin remodeling by TAZ/tafazzin is selectively required for the initiation of mitophagy

Cardiolipin remodeling by TAZ/tafazzin is selectively required for the initiation of mitophagy

http://www.wikidata.org/entity/Q28383403

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Barth Syndrome: From Mitochondrial Dysfunctions Associated with Aberrant Production of Reactive Oxygen Species to Pluripotent Stem Cell Studies Sending Out an SOS: Mitochondria as a Signaling Hub Developmental Sex Differences in the Metabolism of Cardiolipin in Mouse Cerebral Cortex Mitochondria Cardiolipin Regulates Mitophagy through the Protein Kinase C Pathway Cardiolipin content, linoleic acid composition, and tafazzin expression in response to skeletal muscle overload and unload stimuli.Cardiolipin in Central Nervous System Physiology and Pathology.Expanding perspectives on the significance of mitophagy in cancer.Mitochondrial Bioenergetics and Dysfunction in Failing Heart.Overexpression of mitochondrial oxodicarboxylate carrier (ODC1) preserves oxidative phosphorylation in a yeast model of Barth syndrome.The role of nonbilayer phospholipids in mitochondrial structure and function.Mechanisms and Physiological Roles of Mitophagy in Yeast.Shedding light on mitophagy in neurons: what is the evidence for PINK1/Parkin mitophagy in vivo?Loss of tafazzin results in decreased myoblast differentiation in C2C12 cells: A myoblast model of Barth syndrome and cardiolipin deficiency.Decreasing cytosolic translation is beneficial to yeast and human Tafazzin-deficient cells.

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P2860

Cardiolipin remodeling by TAZ/tafazzin is selectively required for the initiation of mitophagy

http://www.wikidata.org/entity/Q28383403

description

2015 nî lūn-bûn

@nan

2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի ապրիլին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

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2015年论文

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name

Cardiolipin remodeling by TAZ/ ...... or the initiation of mitophagy

@ast

Cardiolipin remodeling by TAZ/ ...... or the initiation of mitophagy

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Cardiolipin remodeling by TAZ/ ...... or the initiation of mitophagy

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type

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Cardiolipin remodeling by TAZ/ ...... or the initiation of mitophagy

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Cardiolipin remodeling by TAZ/ ...... or the initiation of mitophagy

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Cardiolipin remodeling by TAZ/ ...... or the initiation of mitophagy

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Cardiolipin remodeling by TAZ/ ...... or the initiation of mitophagy

@ast

Cardiolipin remodeling by TAZ/ ...... or the initiation of mitophagy

@en

Cardiolipin remodeling by TAZ/ ...... or the initiation of mitophagy

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Cardiolipin remodeling by TAZ/ ...... or the initiation of mitophagy

@en

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Ji-Ming Ye

http://www.w3.org/2001/XMLSchema#string

Jun Zhang

http://www.w3.org/2001/XMLSchema#string

Paul Hsu

http://www.w3.org/2001/XMLSchema#string

Xiaolei Liu

http://www.w3.org/2001/XMLSchema#string

Yuguang Shi

http://www.w3.org/2001/XMLSchema#string

P2860

PINK1 is selectively stabilized on impaired mitochondria to activate Parkin

Cardiolipin externalization to the outer mitochondrial membrane acts as an elimination signal for mitophagy in neuronal cells

Defective remodeling of cardiolipin and phosphatidylglycerol in Barth syndrome

Mitochondrial respiratory chain supercomplexes are destabilized in Barth Syndrome patients

Synapses of amphids defective (SAD-A) kinase promotes glucose-stimulated insulin secretion through activation of p21-activated kinase (PAK1) in pancreatic β-Cells

Cardiac and skeletal muscle defects in a mouse model of human Barth syndrome

Characterization of a transgenic short hairpin RNA-induced murine model of Tafazzin deficiency

Identification of the human mitochondrial linoleoyl-coenzyme A monolysocardiolipin acyltransferase (MLCL AT-1)

Fission and selective fusion govern mitochondrial segregation and elimination by autophagy

Crystal structure and biochemical analyses reveal Beclin 1 as a novel membrane binding protein

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