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Pulmonary exposure to carbon black nanoparticles and vascular effects

Pulmonary exposure to carbon black nanoparticles and vascular effects

http://www.wikidata.org/entity/Q28384793

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Inflammation and Vascular Effects after Repeated Intratracheal Instillations of Carbon Black and Lipopolysaccharide Particulate matter and atherosclerosis: a bibliometric analysis of original research articles published in 1973-2014 Carbon black and titanium dioxide nanoparticles induce distinct molecular mechanisms of toxicity Diesel exhaust particulate induces pulmonary and systemic inflammation in rats without impairing endothelial function ex vivo or in vivo Intratracheally instilled titanium dioxide nanoparticles translocate to heart and liver and activate complement cascade in the heart of C57BL/6 mice Changes in cardiopulmonary function induced by nanoparticles Effect of Agglomeration on the Toxicity of Nano-sized Carbon Black in Sprague-Dawley Rats Inhalation of carbon black nanoparticles aggravates pulmonary inflammation in mice Modest effect on plaque progression and vasodilatory function in atherosclerosis-prone mice exposed to nanosized TiO(2)Reduced pulmonary function and increased pro-inflammatory cytokines in nanoscale carbon black-exposed workers.Induction of inflammasome-dependent pyroptosis by carbon black nanoparticles.Effects of maternal exposure to ultrafine carbon black on brain perivascular macrophages and surrounding astrocytes in offspring mice.Carbon black nanoparticles promote endothelial activation and lipid accumulation in macrophages independently of intracellular ROS production.Disrupted Nitric Oxide Metabolism from Type II Diabetes and Acute Exposure to Particulate Air Pollution.From particles to patients: oxidative stress and the cardiovascular effects of air pollution.Acute respiratory symptoms following massive carbon black exposure.Engineered nanomaterial risk. Lessons learnt from completed nanotoxicology studies: potential solutions to current and future challenges.Nanoparticles and the cardiovascular system: a critical review.Perturbation of physiological systems by nanoparticles.Nanosafety research--are we on the right track?Nanomaterial translocation--the biokinetics, tissue accumulation, toxicity and fate of materials in secondary organs--a review.The biological effects upon the cardiovascular system consequent to exposure to particulates of less than 500 nm in size.Atherosclerosis and vasomotor dysfunction in arteries of animals after exposure to combustion-derived particulate matter or nanomaterials.Foam cell formation by particulate matter (PM) exposure: a review.Bio-effect of nanoparticles in the cardiovascular system.Integrated proteomic and metabolomic analysis to assess the effects of pure and benzo[a]pyrene-loaded carbon black particles on energy metabolism and motility in the human endothelial cell line EA.hy926.Repair activity of oxidatively damaged DNA and telomere length in human lung epithelial cells after exposure to multi-walled carbon nanotubes.Nonlinear effects of nanoparticles: biological variability from hormetic doses, small particle sizes, and dynamic adaptive interactions.Pulmonary exposure to particles from diesel exhaust, urban dust or single-walled carbon nanotubes and oxidatively damaged DNA and vascular function in apoE(-/-) mice.Rat lung response to ozone and fine particulate matter (PM2.5) exposures.Zinc oxide nanoparticles-induced intercellular adhesion molecule 1 expression requires Rac1/Cdc42, mixed lineage kinase 3, and c-Jun N-terminal kinase activation in endothelial cells.ZnS nanoarchitectures induced dysfunction of vascular endothelial cells in vitro and in vivo.Pulmonary and atherogenic effects of multi-walled carbon nanotubes (MWCNT) in apolipoprotein-E-deficient mice.Carbon black suppresses the osteogenesis of mesenchymal stem cells: the role of mitochondria.Multiwall carbon nano-onions induce DNA damage and apoptosis in human umbilical vein endothelial cells.Ultrafine carbon black attenuates the antihypertensive effect of captopril in spontaneously hypertensive rats.Resveratrol prevents nanoparticles-induced inflammation and oxidative stress via downregulation of PKC-α and NADPH oxidase in lung epithelial A549 cells.Nanosicherheitsforschung - sind wir auf dem richtigen Weg?In VivoNanotoxicity Assays in Animal Models Human Biomonitoring of Engineered Nanoparticles: An Appraisal of Critical Issues and Potential Biomarkers

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P2860

Pulmonary exposure to carbon black nanoparticles and vascular effects

http://www.wikidata.org/entity/Q28384793

description

2010 nî lūn-bûn

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2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Pulmonary exposure to carbon black nanoparticles and vascular effects

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Pulmonary exposure to carbon black nanoparticles and vascular effects

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Pulmonary exposure to carbon black nanoparticles and vascular effects

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Pulmonary exposure to carbon black nanoparticles and vascular effects

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Pulmonary exposure to carbon black nanoparticles and vascular effects

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Pulmonary exposure to carbon black nanoparticles and vascular effects

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Pulmonary exposure to carbon black nanoparticles and vascular effects

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Pulmonary exposure to carbon black nanoparticles and vascular effects

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Particle and Fibre Toxicology

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Pulmonary exposure to carbon black nanoparticles and vascular effects

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Håkan Wallin

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Janne K Folkmann

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Lise K Vesterdal

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Peter Møller

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P2860

Pulmonary nanoparticle exposure disrupts systemic microvascular nitric oxide signaling

Nanoparticle inhalation augments particle-dependent systemic microvascular dysfunction

Cardiovascular effects of pulmonary exposure to single-wall carbon nanotubes

Ambient air pollution and atherosclerosis in Los Angeles

Systemic and cardiovascular effects of airway injury and inflammation: ultrafine particle exposure in humans.

The induction of vascular endothelial growth factor by ultrafine carbon black contributes to the increase of alveolar-capillary permeability.

Potential role of ultrafine particles in associations between airborne particle mass and cardiovascular health.

Atherosclerosis — An Inflammatory Disease

Endothelial nitric oxide synthase in vascular disease: from marvel to menace

A major role for VCAM-1, but not ICAM-1, in early atherosclerosis

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10.1186/1743-8977-7-33

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1

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Nicklas Raun Jacobsen

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