Direct impairment of vascular function by diesel exhaust particulate through reduced bioavailability of endothelium-derived nitric oxide induced by superoxide free radicals - Wikidata - awgldk - TriplyDB

Direct impairment of vascular function by diesel exhaust particulate through reduced bioavailability of endothelium-derived nitric oxide induced by superoxide free radicals

Direct impairment of vascular function by diesel exhaust particulate through reduced bioavailability of endothelium-derived nitric oxide induced by superoxide free radicals

http://www.wikidata.org/entity/Q28388631

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Nanoparticle inhalation impairs coronary microvascular reactivity via a local reactive oxygen species-dependent mechanism Antioxidants in Cardiovascular Therapy: Panacea or False Hope?Nanomaterials vs Ambient Ultrafine Particles: an Opportunity to Exchange Toxicology Knowledge Circulating factors induce coronary endothelial cell activation following exposure to inhaled diesel exhaust and nitrogen dioxide in humans: evidence from a novel translational in vitro model Diesel exhaust particulate induces pulmonary and systemic inflammation in rats without impairing endothelial function ex vivo or in vivo Mechanisms of diesel-induced endothelial nitric oxide synthase dysfunction in coronary arterioles Urinary bisphenol a concentration and angiography-defined coronary artery stenosis Comparative cardiopulmonary toxicity of exhausts from soy-based biofuels and diesel in healthy and hypertensive rats Update in environmental and occupational medicine 2009 Acute decreases in proteasome pathway activity after inhalation of fresh diesel exhaust or secondary organic aerosol Inhaled nickel nanoparticles alter vascular reactivity in C57BL/6 mice Toxicity and cellular uptake of gold nanorods in vascular endothelium and smooth muscles of isolated rat blood vessel: importance of surface modification Short-term effects of ambient particulates and gaseous pollutants on the incidence of transient ischaemic attack and minor stroke: a case-crossover study Nanoparticles, human health hazard and regulation Diesel exhaust particulate increases the size and complexity of lesions in atherosclerotic mice Altered nitric oxide bioavailability contributes to diesel exhaust inhalation-induced cardiovascular dysfunction in man Cardiovascular health and particulate vehicular emissions: a critical evaluation of the evidence Air pollution, vascular disease and thrombosis: linking clinical data and pathogenic mechanisms.CD36-dependent 7-ketocholesterol accumulation in macrophages mediates progression of atherosclerosis in response to chronic air pollution exposure.A novel genetic score approach using instruments to investigate interactions between pathways and environment: application to air pollution.Controlled exposure to particulate matter from urban street air is associated with decreased vasodilation and heart rate variability in overweight and older adults Combustion-derived nanoparticulate induces the adverse vascular effects of diesel exhaust inhalation.Reducing personal exposure to particulate air pollution improves cardiovascular health in patients with coronary heart disease.Disrupted Nitric Oxide Metabolism from Type II Diabetes and Acute Exposure to Particulate Air Pollution.Whole and particle-free diesel exhausts differentially affect cardiac electrophysiology, blood pressure, and autonomic balance in heart failure-prone rats Hazard identification of particulate matter on vasomotor dysfunction and progression of atherosclerosis.Cardiovascular effects of particulate air pollution exposure: time course and underlying mechanisms.From particles to patients: oxidative stress and the cardiovascular effects of air pollution.Nanoparticles and the cardiovascular system: a critical review.Indoor aerosols: from personal exposure to risk assessment.The role of oxidative stress in the cardiovascular actions of particulate air pollution.Nanodiagnostics, nanopharmacology and nanotoxicology of platelet-vessel wall interactions.Atherosclerosis and vasomotor dysfunction in arteries of animals after exposure to combustion-derived particulate matter or nanomaterials.Oxidative Stress and Cardiovascular Risk: Obesity, Diabetes, Smoking, and Pollution: Part 3 of a 3-Part Series.Air Pollution and Other Environmental Modulators of Cardiac Function.Characterization of the components of urban particulate matter mediating impairment of nitric oxide-dependent relaxation in intrapulmonary arteries.Alteration of peripheral blood monocyte gene expression in humans following diesel exhaust inhalation.Toxicology of ZnO and TiO2 nanoparticles on hepatocytes: impact on metabolism and bioenergetics.Exposure to nitrogen dioxide is not associated with vascular dysfunction in man Systemic effects of controlled exposure to diesel exhaust: a meta-analysis from randomized controlled trials

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P2860

Direct impairment of vascular function by diesel exhaust particulate through reduced bioavailability of endothelium-derived nitric oxide induced by superoxide free radicals

http://www.wikidata.org/entity/Q28388631

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2009 nî lūn-bûn

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P2860

Nanoparticle inhalation augments particle-dependent systemic microvascular dysfunction

Particulate matter exposure impairs systematic microvascular endothelium-dependent dilation

Systemic microvascular dysfunction and inflammation after pulmonary particulate matter exposure

Concentrated ambient air particles induce vasoconstriction of small pulmonary arteries in rats

Association of fine particulate matter from different sources with daily mortality in six U.S. cities

Ambient air pollution and atherosclerosis in Los Angeles

Pulmonary and systemic distribution of inhaled ultrafine silver particles in rats

Pollutant particles produce vasoconstriction and enhance MAPK signaling via angiotensin type I receptor

Combustion-derived nanoparticles: a review of their toxicology following inhalation exposure

An association between air pollution and mortality in six U.S. cities

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