A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis - Wikidata - awgldk - TriplyDB

A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

http://www.wikidata.org/entity/Q28388782

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MyD88 mediates in vivo effector functions of alveolar macrophages in acute lung inflammatory responses to carbon nanotube exposure The complex cascade of cellular events governing inflammasome activation and IL-1β processing in response to inhaled particles Immunotoxicological impact of engineered nanomaterial exposure: mechanisms of immune cell modulation Immunomodulation of nanoparticles in nanomedicine applications Evaluating the mechanistic evidence and key data gaps in assessing the potential carcinogenicity of carbon nanotubes and nanofibers in humans Mechanisms of lung fibrosis induced by carbon nanotubes: towards an Adverse Outcome Pathway (AOP)From immunotoxicity to nanotherapy: the effects of nanomaterials on the immune system Pulmonary instillation of MWCNT increases lung permeability, decreases gp130 expression in the lungs, and initiates cardiovascular IL-6 transsignaling C₆₀ exposure augments cardiac ischemia/reperfusion injury and coronary artery contraction in Sprague Dawley rats The alarmin IL-1α is a master cytokine in acute lung inflammation induced by silica micro- and nanoparticles Impact of pulmonary exposure to gold core silver nanoparticles of different size and capping agents on cardiovascular injury IL-33 modulates chronic airway resistance changes induced by multi-walled carbon nanotubes Atomic layer deposition coating of carbon nanotubes with aluminum oxide alters pro-fibrogenic cytokine expression by human mononuclear phagocytes in vitro and reduces lung fibrosis in mice in vivo Innate Immune Responses to Nanoparticle Exposure in the Lung Inhalation of rod-like carbon nanotubes causes unconventional allergic airway inflammation Intravenously delivered graphene nanosheets and multiwalled carbon nanotubes induce site-specific Th2 inflammatory responses via the IL-33/ST2 axis Cardiac Ischemia Reperfusion Injury Following Instillation of 20 nm Citrate-capped Nanosilver Single-walled carbon nanotube exposure induces membrane rearrangement and suppression of receptor-mediated signalling pathways in model mast cells.Influence of physicochemical properties of silver nanoparticles on mast cell activation and degranulation.Rosette Nanotubes Alter IgE-Mediated Degranulation in the Rat Basophilic Leukemia (RBL)-2H3 Cell Line.Elimination of p19(ARF)-expressing cells enhances pulmonary function in mice.Contribution of engineered nanomaterials physicochemical properties to mast cell degranulation Scale of health: indices of safety and efficacy in the evolving environment of large biological datasets.Implications of scavenger receptors in the safe development of nanotherapeutics.Vaccine technologies: From whole organisms to rationally designed protein assemblies.Crucial role of the biological barrier at the primary targeted organs in controlling the translocation and toxicity of multi-walled carbon nanotubes in the nematode Caenorhabditis elegans.Multifunctional Polymer-Coated Carbon Nanotubes for Safe Drug Delivery.Stat-6 signaling pathway and not Interleukin-1 mediates multi-walled carbon nanotube-induced lung fibrosis in mice: insights from an adverse outcome pathway framework.Mechanistic insight into the impact of nanomaterials on asthma and allergic airway disease.Macrophage sensing of single-walled carbon nanotubes via Toll-like receptors.Macrophage polarization and activation at the interface of multi-walled carbon nanotube-induced pulmonary inflammation and fibrosis.Type 2 Immune Mechanisms in Carbon Nanotube-Induced Lung Fibrosis.

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P2860

A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

http://www.wikidata.org/entity/Q28388782

description

2012 nî lūn-bûn

@nan

2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

name

A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

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A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

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A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

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type

label

A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

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A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

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A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

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prefLabel

A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

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A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

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A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

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A carbon nanotube toxicity paradigm driven by mast cells and the IL-₃₃/ST₂ axis

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Apparao M Rao

http://www.w3.org/2001/XMLSchema#string

Jared M Brown

http://www.w3.org/2001/XMLSchema#string

Pranita Katwa

http://www.w3.org/2001/XMLSchema#string

Rakhee N Urankar

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Robert B Fick

http://www.w3.org/2001/XMLSchema#string

Susana C Hilderbrand

http://www.w3.org/2001/XMLSchema#string

Xiaojia Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Pulmonary fibrotic response to aspiration of multi-walled carbon nanotubes

Dispersal state of multiwalled carbon nanotubes elicits profibrogenic cellular responses that correlate with fibrogenesis biomarkers and fibrosis in the murine lung

Multiwalled carbon nanotubes induce a fibrogenic response by stimulating reactive oxygen species production, activating NF-kB signaling, and promoting fibroblast-to-myofibroblast transformation

IL-33 is processed into mature bioactive forms by neutrophil elastase and cathepsin G

Endogenous IL-33 is highly expressed in mouse epithelial barrier tissues, lymphoid organs, brain, embryos, and inflamed tissues: in situ analysis using a novel Il-33-LacZ gene trap reporter strain

Serum soluble ST2: a potential novel mediator in left ventricular and infarct remodeling after acute myocardial infarction

IL-33 mediates inflammatory responses in human lung tissue cells

Multi-walled carbon nanotube instillation impairs pulmonary function in C57BL/6 mice

Expansion of cardiac ischemia/reperfusion injury after instillation of three forms of multi-walled carbon nanotubes

Mast cell-deficient W-sash c-kit mutant Kit W-sh/W-sh mice as a model for investigating mast cell biology in vivo

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2904-12

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scholarly article

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3911006

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10.1002/SMLL.201200873

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18

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8

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Ramakrishna Podila

Christopher J Wingard

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2012-09-24T00:00:00Z

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229014905

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22777948

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carbon nanotube

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3445726

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