Allele-specific induction of IL-1β expression by C/EBPβ and PU.1 contributes to increased tuberculosis susceptibility - Wikidata - awgldk - TriplyDB

Allele-specific induction of IL-1β expression by C/EBPβ and PU.1 contributes to increased tuberculosis susceptibility

Allele-specific induction of IL-1β expression by C/EBPβ and PU.1 contributes to increased tuberculosis susceptibility

http://www.wikidata.org/entity/Q28396007

about

New insights into TB physiology suggest untapped therapeutic opportunities Construction and analysis of the transcription factor-microRNA co-regulatory network response to Mycobacterium tuberculosis: a view from the blood.PU.1 serves a critical role in the innate defense against Aspergillus fumigatus via dendritic cell-associated C-type lectin receptor-1 and toll-like receptors-2 and 4 in THP-1-derived macrophages PU.1 is involved in the immune response to Aspergillus fumigatus through upregulating Dectin-1 expression.Serum IL-1β and IL-18 correlate with ESR and CRP in multidrug-resistant tuberculosis patients.Genetic Variation in Toll-Interacting Protein Is Associated With Leprosy Susceptibility and Cutaneous Expression of Interleukin 1 Receptor Antagonist.Interactions between Type 1 Interferons and the Th17 Response in Tuberculosis: Lessons Learned from Autoimmune Diseases.Nitric oxide prevents a pathogen-permissive granulocytic inflammation during tuberculosis.PPAR-α Activation Mediates Innate Host Defense through Induction of TFEB and Lipid Catabolism.Myeloid-derived NF-κB negative regulation of PU.1 and c/EBP-β-driven pro-inflammatory cytokine production restrains LPS-induced shock.Sex differences in the C57BL/6 model of Mycobacterium tuberculosis infection.Plasma Cytokine Predictors of Tuberculosis Recurrence in Antiretroviral-Treated Human Immunodeficiency Virus-infected Individuals from Durban, South Africa.Type I Interferons in the Pathogenesis of Tuberculosis: Molecular Drivers and Immunological Consequences.A proline deletion in IFNAR1 impairs IFN-signaling and underlies increased resistance to tuberculosis in humans.Multiple DNA-binding modes for the ETS family transcription factor PU.1.Is Receptor-Interacting Protein Kinase 3 a Viable Therapeutic Target for Mycobacterium tuberculosis Infection?Lysosomal Cathepsin Release Is Required for NLRP3-Inflammasome Activation by Mycobacterium tuberculosis in Infected Macrophages.Tolerating the Unwelcome Guest; How the Host Withstands Persistent

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Allele-specific induction of IL-1β expression by C/EBPβ and PU.1 contributes to increased tuberculosis susceptibility

http://www.wikidata.org/entity/Q28396007

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2014 nî lūn-bûn

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2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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Boping Zhou

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Christopher M Sassetti

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Gucheng Zeng

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Guoliang Zhang

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Hui Yang

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Jun Yue

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Mingfeng Liao

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Mingxia Zhang

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Senlin Zhan

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Shaoyuan Li

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P2860

Pathogenesis, immunology, and diagnosis of latent Mycobacterium tuberculosis infection

Lethal influenza virus infection in macaques is associated with early dysregulation of inflammatory related genes

Chronic pulmonary function impairment caused by initial and recurrent pulmonary tuberculosis following treatment

A gamma interferon independent mechanism of CD4 T cell mediated control of M. tuberculosis infection in vivo

Interleukin-1 polymorphisms associated with increased risk of gastric cancer

Systemic disease during Streptococcus pneumoniae acute lung infection requires 12-lipoxygenase-dependent inflammation

Genetic variants in IL1A and IL1B contribute to the susceptibility to 2009 pandemic H1N1 influenza A virus.

The lta4h locus modulates susceptibility to mycobacterial infection in zebrafish and humans

The influence of host and bacterial genotype on the development of disseminated disease with Mycobacterium tuberculosis

Human TLR1 deficiency is associated with impaired mycobacterial signaling and protection from leprosy reversal reaction.

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