Interactions among genetic variants in apoptosis pathway genes, reflux symptoms, body mass index, and smoking indicate two distinct etiologic patterns of esophageal adenocarcinoma - Wikidata - awgldk - TriplyDB

Interactions among genetic variants in apoptosis pathway genes, reflux symptoms, body mass index, and smoking indicate two distinct etiologic patterns of esophageal adenocarcinoma

Interactions among genetic variants in apoptosis pathway genes, reflux symptoms, body mass index, and smoking indicate two distinct etiologic patterns of esophageal adenocarcinoma

http://www.wikidata.org/entity/Q28396031

about

Recent developments in esophageal adenocarcinoma FASL rs763110 polymorphism contributes to cancer risk: an updated meta-analysis involving 43,295 subjects Interactions between genetic polymorphisms in the apoptotic pathway and environmental factors on esophageal adenocarcinoma risk Association between polymorphisms in cancer-related genes and early onset of esophageal adenocarcinoma.Whole-miRNome profiling identifies prognostic serum miRNAs in esophageal adenocarcinoma: the influence of Helicobacter pylori infection status.Polymorphisms in SELE gene and risk of coal workers' pneumoconiosis in Chinese: a case-control study.Genome-wide DNA methylation profiling of cell-free serum DNA in esophageal adenocarcinoma and Barrett esophagus.The Role of Gastroesophageal Reflux and Other Factors during Progression to Esophageal Adenocarcinoma Effects of High-Order Interactions among IGFBP-3 Genetic Polymorphisms, Body Mass Index and Soy Isoflavone Intake on Breast Cancer Susceptibility.Body mass index in relation to oesophageal and oesophagogastric junction adenocarcinomas: a pooled analysis from the International BEACON Consortium A newly identified susceptibility locus near FOXP1 modifies the association of gastroesophageal reflux with Barrett's esophagus.Bladder cancer epidemiology and genetic susceptibility.Angiogenesis-related agents in esophageal cancer.A clinical perspective on the role of chronic inflammation in gastrointestinal cancer.Association of MDM2 T309G and p53 Arg72Pro polymorphisms and gastroesophageal reflux disease with survival in esophageal adenocarcinoma.Impact of risk factors on functional status in maintenance hemodialysis patients.Interactions Between Genetic Variants and Environmental Factors Affect Risk of Esophageal Adenocarcinoma and Barrett's Esophagus.Association of candidate genetic variations with gastric cardia adenocarcinoma in Chinese population: a multiple interaction analysis.

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P2860

Interactions among genetic variants in apoptosis pathway genes, reflux symptoms, body mass index, and smoking indicate two distinct etiologic patterns of esophageal adenocarcinoma

http://www.wikidata.org/entity/Q28396031

description

2010 nî lūn-bûn

@nan

2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Interactions among genetic var ...... s of esophageal adenocarcinoma

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Interactions among genetic var ...... s of esophageal adenocarcinoma

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Interactions among genetic var ...... s of esophageal adenocarcinoma

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type

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Interactions among genetic var ...... s of esophageal adenocarcinoma

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Interactions among genetic var ...... s of esophageal adenocarcinoma

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Interactions among genetic var ...... s of esophageal adenocarcinoma

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prefLabel

Interactions among genetic var ...... s of esophageal adenocarcinoma

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Interactions among genetic var ...... s of esophageal adenocarcinoma

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Interactions among genetic var ...... s of esophageal adenocarcinoma

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JCO.2009.26.2790

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Journal of Clinical Oncology

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Interactions among genetic var ...... s of esophageal adenocarcinoma

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David C Christiani

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Feng Chen

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John C Wain

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Kofi Asomaning

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Li Su

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Matthew H Kulke

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Norman S Nishioka

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Rebecca S Heist

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Rihong Zhai

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Xihong Lin

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P2860

The Hallmarks of Cancer

Apoptosis gene polymorphisms, age, smoking and the risk of non-small cell lung cancer

Survival trends in patients with gastric and esophageal adenocarcinomas: a population-based study

Power of multifactor dimensionality reduction for detecting gene-gene interactions in the presence of genotyping error, missing data, phenocopy, and genetic heterogeneity.

Detecting pathway-based gene-gene and gene-environment interactions in pancreatic cancer.

The effects of insulin-like growth factors on tumorigenesis and neoplastic growth.

Multifactor-dimensionality reduction reveals high-order interactions among estrogen-metabolism genes in sporadic breast cancer.

Bladder cancer predisposition: a multigenic approach to DNA-repair and cell-cycle-control genes.

Quantitative trait Loci analysis using the false discovery rate.

Variants in the GH-IGF axis confer susceptibility to lung cancer.

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2445-51

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3511907

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10.1200/JCO.2009.26.2790

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14

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28

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Chau-Chyun Sheu

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2010-05-10T00:00:00Z

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20385987

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body mass index

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2881724

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