Identification of key processes that control tumor necrosis factor availability in a tuberculosis granuloma
about
Tuneable resolution as a systems biology approach for multi-scale, multi-compartment computational modelsA multi-scale approach to designing therapeutics for tuberculosisMultiscale computational modeling reveals a critical role for TNF-α receptor 1 dynamics in tuberculosis granuloma formation.CXCR7 controls competition for recruitment of β-arrestin 2 in cells expressing both CXCR4 and CXCR7.Tuberculosis: global approaches to a global disease.MAPK involvement in cytokine production in response to Corynebacterium pseudotuberculosis infection.Multi-scale modeling predicts a balance of tumor necrosis factor-α and interleukin-10 controls the granuloma environment during Mycobacterium tuberculosis infection.Differential risk of tuberculosis reactivation among anti-TNF therapies is due to drug binding kinetics and permeability.Systems biology approaches for understanding cellular mechanisms of immunity in lymph nodes during infectionNF-κB Signaling Dynamics Play a Key Role in Infection Control in Tuberculosis.Lung necrosis and neutrophils reflect common pathways of susceptibility to Mycobacterium tuberculosis in genetically diverse, immune-competent mice.A hybrid multi-compartment model of granuloma formation and T cell priming in tuberculosis.Microenvironments in tuberculous granulomas are delineated by distinct populations of macrophage subsets and expression of nitric oxide synthase and arginase isoforms.A multifaceted approach to modeling the immune response in tuberculosis.Systems approach to tuberculosis vaccine development.Functional complexity of the Leishmania granuloma and the potential of in silico modeling.In silico models of M. tuberculosis infection provide a route to new therapiesIdentifying mechanisms driving formation of granuloma-associated fibrosis during Mycobacterium tuberculosis infection.A population model capturing dynamics of tuberculosis granulomas predicts host infection outcomes.SOCS1 gene expression is increased in severe pulmonary tuberculosis.Deletion of TGF-β1 Increases Bacterial Clearance by Cytotoxic T Cells in a Tuberculosis Granuloma Model.A Multi-Compartment Hybrid Computational Model Predicts Key Roles for Dendritic Cells in Tuberculosis Infection.Low Levels of T Cell Exhaustion in Tuberculous Lung Granulomas
P2860
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P2860
Identification of key processes that control tumor necrosis factor availability in a tuberculosis granuloma
description
2010 nî lūn-bûn
@nan
2010 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Identification of key processe ...... ty in a tuberculosis granuloma
@ast
Identification of key processe ...... ty in a tuberculosis granuloma
@en
Identification of key processe ...... ty in a tuberculosis granuloma
@nl
type
label
Identification of key processe ...... ty in a tuberculosis granuloma
@ast
Identification of key processe ...... ty in a tuberculosis granuloma
@en
Identification of key processe ...... ty in a tuberculosis granuloma
@nl
prefLabel
Identification of key processe ...... ty in a tuberculosis granuloma
@ast
Identification of key processe ...... ty in a tuberculosis granuloma
@en
Identification of key processe ...... ty in a tuberculosis granuloma
@nl
P2093
P2860
P1476
Identification of key processe ...... ty in a tuberculosis granuloma
@en
P2093
Denise E Kirschner
Jennifer J Linderman
Matthew A Schaller
Mohammad Fallahi-Sichani
Steven L Kunkel
P2860
P304
P356
10.1371/JOURNAL.PCBI.1000778
P407
P577
2010-05-06T00:00:00Z