Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner - Wikidata - awgldk - TriplyDB

Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

http://www.wikidata.org/entity/Q28476620

about

CD47 update: a multifaceted actor in the tumour microenvironment of potential therapeutic interest Protective role of S-nitrosoglutathione (GSNO) against cognitive impairment in rat model of chronic cerebral hypoperfusion.Synaptic therapy in Alzheimer's disease: a CREB-centric approach.Role of amyloid peptides in vascular dysfunction and platelet dysregulation in Alzheimer's disease The soluble guanylate cyclase stimulator BAY 41-2272 inhibits vascular smooth muscle growth through the cAMP-dependent protein kinase and cGMP-dependent protein kinase pathways.The matricellular protein thrombospondin-1 globally regulates cardiovascular function and responses to stress via CD47.Reduction in inflammation and the expression of amyloid precursor protein and other proteins related to Alzheimer's disease following gastric bypass surgery.Soluble beta amyloid evokes alteration in brain norepinephrine levels: role of nitric oxide and interleukin-1.CD47 signaling pathways controlling cellular differentiation and responses to stress.Activated cyclin-dependent kinase 5 promotes microglial phagocytosis of fibrillar β-amyloid by up-regulating lipoprotein lipase expression Regulation of soluble guanylate cyclase by matricellular thrombospondins: implications for blood flow.Thrombospondins: old players, new games Modulating nitric oxide signaling in the CNS for Alzheimer's disease therapy.Regulation of cellular redox signaling by matricellular proteins in vascular biology, immunology, and cancer Oxyradical stress increases the biosynthesis of 2-arachidonoylglycerol: involvement of NADPH oxidase.Expression and Processing of Amyloid Precursor Protein in Vascular Endothelium.Reduced nitric oxide bioavailability mediates cerebroarterial dysfunction independent of cerebral amyloid angiopathy in a mouse model of Alzheimer's disease.

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Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

http://www.wikidata.org/entity/Q28476620

description

2010 nî lūn-bûn

@nan

2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

@ast

Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

@en

Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

@nl

type

label

Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

@ast

Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

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Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

@nl

prefLabel

Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

@ast

Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

@en

Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

@nl

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JOURNAL.PONE.0015686

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Amyloid-β inhibits No-cGMP signaling in a CD36- and CD47-dependent manner

@en

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David D Roberts

http://www.w3.org/2001/XMLSchema#string

Hubert B Shih

http://www.w3.org/2001/XMLSchema#string

Jeff S Isenberg

http://www.w3.org/2001/XMLSchema#string

Thomas W Miller

http://www.w3.org/2001/XMLSchema#string

Yichen Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Current Insights into Molecular Mechanisms of Alzheimer Disease and Their Implications for Therapeutic Approaches

Thrombospondin-1 inhibits nitric oxide signaling via CD36 by inhibiting myristic acid uptake

Adhesion of human T cells to antigen-presenting cells through SIRPbeta2-CD47 interaction costimulates T-cell proliferation

Thrombospondin-1 inhibits endothelial cell responses to nitric oxide in a cGMP-dependent manner

CD47 is necessary for inhibition of nitric oxide-stimulated vascular cell responses by thrombospondin-1

Inhibition of Alzheimer's beta-amyloid induced vasoactivity and proinflammatory response in microglia by a cGMP-dependent mechanism

Alzheimer's disease is a synaptic failure

Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide

A beta-peptides enhance vasoconstriction in cerebral circulation.

CD36, a scavenger receptor involved in immunity, metabolism, angiogenesis, and behavior.

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e15686

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scholarly article

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10.1371/JOURNAL.PONE.0015686

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12

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5

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2010-12-22T00:00:00Z

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252110447

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21203512

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3008726

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