Resistance to MPTP-neurotoxicity in α-synuclein knockout mice is complemented by human α-synuclein and associated with increased β-synuclein and Akt activation
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Αlpha-Synuclein as a Mediator in the Interplay between Aging and Parkinson's DiseaseEpigenetic landscape of Parkinson's disease: emerging role in disease mechanisms and therapeutic modalitiesAnimal models of Parkinson's disease: vertebrate geneticsLoss of α-Synuclein Does Not Affect Mitochondrial Bioenergetics in Rodent Neurons.Gene-environment interaction models to unmask susceptibility mechanisms in Parkinson's disease.Nanomolar concentration of alpha-synuclein enhances dopaminergic neuronal survival via Akt pathway.Targeted Overexpression of α-Synuclein by rAAV2/1 Vectors Induces Progressive Nigrostriatal Degeneration and Increases Vulnerability to MPTP in MouseNMR Metabolomics Analysis of Parkinson's DiseaseMitochondrial dysfunction in genetic animal models of Parkinson's diseaseInteraction between Neuromelanin and Alpha-Synuclein in Parkinson's Disease.Parkinson's disease: don't mess with calcium.Classic and new animal models of Parkinson's disease.Prokineticin-2 upregulation during neuronal injury mediates a compensatory protective response against dopaminergic neuronal degeneration.Region-specific deficits in dopamine, but not norepinephrine, signaling in a novel A30P α-synuclein BAC transgenic mouse.The function of α-synuclein.Destabilization of survival factor MEF2D mRNA by neurotoxin in models of Parkinson's disease.The c-Abl inhibitor, nilotinib, protects dopaminergic neurons in a preclinical animal model of Parkinson's disease.Akt as a victim, villain and potential hero in Parkinson's disease pathophysiology and treatment.Endogenous alpha-synuclein influences the number of dopaminergic neurons in mouse substantia nigraMicroRNAs and Target Genes As Biomarkers for the Diagnosis of Early Onset of Parkinson Disease.α-Synuclein accumulation and GBA deficiency due to L444P GBA mutation contributes to MPTP-induced parkinsonism.Alpha-synuclein alters differently gene expression of Sirts, PARPs and other stress response proteins: implications for neurodegenerative disorders.7,8-dihydroxyflavone Ameliorates Motor Deficits Via Suppressing α-synuclein Expression and Oxidative Stress in the MPTP-induced Mouse Model of Parkinson's Disease.Current perspective of mitochondrial biology in Parkinson's disease.
P2860
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P2860
Resistance to MPTP-neurotoxicity in α-synuclein knockout mice is complemented by human α-synuclein and associated with increased β-synuclein and Akt activation
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Resistance to MPTP-neurotoxici ...... β-synuclein and Akt activation
@ast
Resistance to MPTP-neurotoxici ...... β-synuclein and Akt activation
@en
Resistance to MPTP-neurotoxici ...... β-synuclein and Akt activation
@nl
type
label
Resistance to MPTP-neurotoxici ...... β-synuclein and Akt activation
@ast
Resistance to MPTP-neurotoxici ...... β-synuclein and Akt activation
@en
Resistance to MPTP-neurotoxici ...... β-synuclein and Akt activation
@nl
prefLabel
Resistance to MPTP-neurotoxici ...... β-synuclein and Akt activation
@ast
Resistance to MPTP-neurotoxici ...... β-synuclein and Akt activation
@en
Resistance to MPTP-neurotoxici ...... β-synuclein and Akt activation
@nl
P2093
P2860
P50
P921
P3181
P1433
P1476
Resistance to MPTP-neurotoxici ...... β-synuclein and Akt activation
@en
P2093
Allen S Mandir
Bobby Thomas
Shaida A Andrabi
Wanda Stirling
P2860
P304
P3181
P356
10.1371/JOURNAL.PONE.0016706
P407
P577
2011-01-31T00:00:00Z