Promoter hypermethylation mediated downregulation of FBP1 in human hepatocellular carcinoma and colon cancer - Wikidata - awgldk - TriplyDB

Promoter hypermethylation mediated downregulation of FBP1 in human hepatocellular carcinoma and colon cancer

Promoter hypermethylation mediated downregulation of FBP1 in human hepatocellular carcinoma and colon cancer

http://www.wikidata.org/entity/Q28477572

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The expression of glyceraldehyde-3-phosphate dehydrogenase associated cell cycle (GACC) genes correlates with cancer stage and poor survival in patients with solid tumors Xmrk, kras and myc transgenic zebrafish liver cancer models share molecular signatures with subsets of human hepatocellular carcinoma Interplay between epigenetics and metabolism in oncogenesis: mechanisms and therapeutic approaches Proteomic differences between hepatocellular carcinoma and nontumorous liver tissue investigated by a combined gel-based and label-free quantitative proteomics study.Suppression of gluconeogenic gene expression by LSD1-mediated histone demethylation.Genes dysregulated to different extent or oppositely in estrogen receptor-positive and estrogen receptor-negative breast cancers.TRIM24 links glucose metabolism with transformation of human mammary epithelial cells NPM1 activates metabolic changes by inhibiting FBP1 while promoting the tumorigenicity of pancreatic cancer cells.The combination of three molecular markers can be a valuable predictive tool for the prognosis of hepatocellular carcinoma patients.Association between FBP1 and hypoxia-related gene expression in clear cell renal cell carcinoma Loss of FBP1 by Snail-mediated repression provides metabolic advantages in basal-like breast cancer.The tumor microenvironment: An irreplaceable element of tumor budding and epithelial-mesenchymal transition-mediated cancer metastasis Remodelling of the hepatic epigenetic landscape of glucose-intolerant rainbow trout (Oncorhynchus mykiss) by nutritional status and dietary carbohydrates Decreased fructose-1,6-bisphosphatase-2 expression promotes glycolysis and growth in gastric cancer cells.Inhibiting histone deacetylases suppresses glucose metabolism and hepatocellular carcinoma growth by restoring FBP1 expression.Epigenetics and cancer metabolism.Interactions between epigenetics and metabolism in cancers.Metabolism-epigenome crosstalk in physiology and diseases.Aberrant DNA methylation in hepatocellular carcinoma tumor suppression (Review).Metabostemness: a new cancer hallmark.Meta-analysis of gene expression profiles identifies differential biomarkers for hepatocellular carcinoma and cholangiocarcinoma.Loss of fructose-1,6-bisphosphatase induces glycolysis and promotes apoptosis resistance of cancer stem-like cells: an important role in hexavalent chromium-induced carcinogenesis.Targeted knockdown of polo-like kinase 1 alters metabolic regulation in melanoma.Down-regulation of FBP1 by ZEB1-mediated repression confers to growth and invasion in lung cancer cells.miR-615-5p is restrictedly expressed in cirrhotic and cancerous liver tissues and its overexpression alleviates the tumorigenic effects in hepatocellular carcinoma.mTORC2 controls cancer cell survival by modulating gluconeogenesis.TIGAR, TIGAR, burning bright.Quantitative proteomic analysis in metastatic renal cell carcinoma reveals a unique set of proteins with potential prognostic significance.Switch of glycolysis to gluconeogenesis by dexamethasone for treatment of hepatocarcinoma.Invalidation of mitophagy by FBP1-mediated repression promotes apoptosis in breast cancer.Increased miR-21a provides metabolic advantages through suppression of FBP1 expression in non-small cell lung cancer cells.YAP suppresses gluconeogenic gene expression through PGC1α.Loss of FBP1 facilitates aggressive features of hepatocellular carcinoma cells through the Warburg effect.Tissue-based quantitative proteome analysis of human hepatocellular carcinoma using tandem mass tags.Targeting FBPase is an emerging novel approach for cancer therapy.CCAAT-enhancer binding protein-α (C/EBPα) and hepatocyte nuclear factor 4α (HNF4α) regulate expression of the human fructose-1,6-bisphosphatase 1 (FBP1) gene in human hepatocellular carcinoma HepG2 cells.Down Expression of FBP1 Is a Negative Prognostic Factor for Non-Small-Cell Lung Cancer.Non-canonical functions of enzymes facilitate cross-talk between cell metabolic and regulatory pathways.Energy stress-induced lncRNA HAND2-AS1 represses HIF1α-mediated energy metabolism and inhibits osteosarcoma progression.Significance of Methylation of FBP1 Gene in Non-Small Cell Lung Cancer.

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Promoter hypermethylation mediated downregulation of FBP1 in human hepatocellular carcinoma and colon cancer

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Promoter hypermethylation medi ...... lar carcinoma and colon cancer

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Promoter hypermethylation medi ...... lar carcinoma and colon cancer

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Promoter hypermethylation medi ...... lar carcinoma and colon cancer

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Promoter hypermethylation medi ...... lar carcinoma and colon cancer

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Promoter hypermethylation medi ...... lar carcinoma and colon cancer

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Promoter hypermethylation medi ...... lar carcinoma and colon cancer

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Promoter hypermethylation medi ...... lar carcinoma and colon cancer

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Jianbin Zhang

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Jianming Zheng

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Mengqi Zhu

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Zhiping Qian

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P2860

Epigenetic gene silencing in cancer – a mechanism for early oncogenic pathway addiction?

Warburg effect revisited: an epigenetic link between glycolysis and gastric carcinogenesis

The fundamental role of epigenetic events in cancer

Fructose-1,6-bisphosphatase mediates cellular responses to DNA damage and aging in Saccharomyces cerevisiae.

Experimental therapeutics: targeting the redox Achilles heel of cancer

The epigenomics of cancer

CpG island hypermethylation and tumor suppressor genes: a booming present, a brighter future.

Reactive oxygen species: a breath of life or death?

Redox regulation of cell survival

Aberrant methylation of human L- and M-fructose 1,6-bisphosphatase genes in cancer.

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